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Shock! Eric Alison Lexi Kevin. Article e&db=cmedm&AN=24509521&site=ehost-

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Presentation on theme: "Shock! Eric Alison Lexi Kevin. Article e&db=cmedm&AN=24509521&site=ehost-"— Presentation transcript:

1 Shock! Eric Alison Lexi Kevin

2 Article e&db=cmedm&AN=24509521&site=ehost- live

3 Distributive shock EXPANDED INTRAVASCULAR SPACE Anaphylactic shock tx- Epi-pen, Benadryl, Steroids (SoluMedrol) neurogenic tx- Immobilization, IV fluids, steroids, surgery Septic tx- Antibiotics, IV fluids

4 Hypovolemic Shock Decreased Blood volume -leads to decreased blood pressure. MAP of 65-75 is generally the accepted minimum needed to perfuse vital organs. -Internal or External blood loss (hemorrhage) -Severe loss of extracellular fluids (vomiting, diarrhea, and excessive diuresis) tx- IV fluids, blood transfusion, stop the cause of fluid/blood loss

5 Obstructive Shock CIRCULATORY BLOCKAGE WHICH DISRUPTS CARDIAC OUTPUT *Pulmonary Embolism tx- anticoagulation therapy * Cardiac tamponade tx- pericardiocentesis * Tension Pneumothorax tx- Chest tube

6 Cardiogenic Shock Cardiogenic shock is a systolic blood pressure less than 90 mmHg for at least 30 minutes secondary to myocardial dysfunction MI with loss of > 40% of left ventricle myocardium Right ventricle MI End-stage cardiomyopathy Papillary muscle dysfunction Cardiac rupture Congenital heart defects tx- Stop the cause, surgery, cardiac catheterization, medications, heart transplant

7 Treatment Call 911 US/CAN, UK 999, Europe 112, Australia 000, Brazil 192, Argentina 107, Chile 131 -Check pt. airway, breathing, and circulation (start CPR) -Elevate legs 12 inches and keep warm -Administer O2 and IV fluids

8 Current Practice Biomedical Press published results from a study Feb 6, 2014 involving twenty-five patients. “The patients suffered from shock secondary to myocardial infarction and were successfully treated with percutaneous coronary intervention. Before the inclusion, 16/25 patients presented a cardiac arrest in the presence of medical staff. Norepinephrine was titrated to increase MAP from 65 mmHg to 85 mmHg during 1 h..In conclusion, a short-term increase in MAP with norepinephrine in resuscitated cardiogenic shock complicated by post reperfusion disease is associated with better cardiac performance and improved microcirculatory variables.” (Perez)

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