Spinal cord stimulation treatment for angina pectoris: more than a placebo? Philippe A Gersbach, MD, Mustafa G Hasdemir, MD, Eric Eeckhout, MD, PhD, Ludwig.

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Presentation transcript:

Spinal cord stimulation treatment for angina pectoris: more than a placebo? Philippe A Gersbach, MD, Mustafa G Hasdemir, MD, Eric Eeckhout, MD, PhD, Ludwig K von Segesser, MD The Annals of Thoracic Surgery Volume 72, Issue 3, Pages S1100-S1104 (September 2001) DOI: 10.1016/S0003-4975(01)02950-2

Fig 1 Main effects of 2 and 5 V cervical spinal cord stimulation on hemodynamics. Substantial increases are shown in cardiac output and minimal variations in heart rate- and intensity-dependent rise in blood pressure. Typical short initial decreases (↓) in heart rate and blood pressure are evident. (ART1 = systemic blood pressure [systolic, mean diastolic]; ART2 = left ventricular pressure; HF = heart rate; HZV = cardiac output [scale 1 to 10 L, time on horizontal axis]; PAP = pulmonary artery pressure; ZVD = central venous pressure [artifacts due to cardiac output measurements].) The Annals of Thoracic Surgery 2001 72, S1100-S1104DOI: (10.1016/S0003-4975(01)02950-2)

Fig 2 Effects of high (10 V) and middle (5 V) intensity cervical spinal cord stimulation during short interval stimulation. Exhaustion mechanisms are absent. Changes include short initial decreases (↓) in heart rate and blood pressure followed by intensity-dependent increases, and a fast return to base line values after spinal cord stimulation. Major hemodynamic changes are seen at 10 V. ∗Compression of ocular globes. (ART1 = systemic blood pressure [systolic, mean diastolic]; ART2 = left ventricular pressure; HF = heart rate.) The Annals of Thoracic Surgery 2001 72, S1100-S1104DOI: (10.1016/S0003-4975(01)02950-2)