1. Pulmonary vascular changes induced by congenital obstruction of pulmonary venous return 
Masato Endo, MD, Shigeo Yamaki, MD, Mikio Ohmi, MD, Koichi Tabayashi, MD 
The Annals of Thoracic Surgery 
Volume 69, Issue 1, Pages (January 2000)
DOI: /S (99)

2. Fig 1
(A) Medial thickening and intimal fibrosis of the small pulmonary artery in case 6 and (B) marked medial thickening with intimal fibrosis of small pulmonary vein in case 5. (Elastica Masson stain, × 100.)
The Annals of Thoracic Surgery  , DOI: ( /S (99) )

3. Fig 2
Lymphangiectasia in lung parenchyma in case 6. (Lym = lymph vessel; Alv = alveolus.) (Elastica Masson stain, × 100.)
The Annals of Thoracic Surgery  , DOI: ( /S (99) )

4. Fig 3
The correlations between medial thicknesses (Dr = 100 μm) of pulmonary arteries and preoperative systolic pulmonary arterial pressures in 8 PVO cases and in 20 VSD cases. The slope of the regression line in PVO cases is greater than that of VSD cases (0.24 versus 0.07, p < 0.025). (PAP = pulmonary arterial pressure; PVO = pulmonary venous obstruction; VSD = ventricular septal defect.)
The Annals of Thoracic Surgery  , DOI: ( /S (99) )

5. Fig 4
The medial thickness (Dr = 100 μm) of pulmonary veins in PVO cases and in VSD cases. (PVO = pulmonary venous obstruction; VSD = ventricular septal defect, ∗p = )
The Annals of Thoracic Surgery  , DOI: ( /S (99) )

6. Fig 5
Progression of intimal changes, expressed as the percentage of the numbers of the vessels with intimal changes, (A) of pulmonary arteries and (B) pulmonary veins in PVO and VSD cases. (PVO = pulmonary venous obstruction; VSD = ventricular septal defect.)
The Annals of Thoracic Surgery  , DOI: ( /S (99) )