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A general framework Exogenous variables Stressor, pathogen, culture Internal Process Health Outcome Chronic Stress Chronic disease Environmental exposure.

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Presentation on theme: "A general framework Exogenous variables Stressor, pathogen, culture Internal Process Health Outcome Chronic Stress Chronic disease Environmental exposure."— Presentation transcript:

1 A general framework Exogenous variables Stressor, pathogen, culture Internal Process Health Outcome Chronic Stress Chronic disease Environmental exposure Social inequity Cultural norms “Person” variables Temperament Personality Acute Stress Affect Environmental exposure

2 A general framework Endogenous variables Stressor, pathogen, culture Internal Process Health Outcome ψ Affective state Self-perception Perceived vulnerability Self-efficacy, etc. Physical Arousal “Allostatic load” HPT activation Inflammation “Metabolic syndrome” Immunocompetence

3 A general framework Outcome variables Stressor, pathogen, culture Internal Process Health Outcome Behavioral Health behaviors Alcohol drug abuse Risk, etc. Biomedical CHD BMI Infection, etc.

4 Course topics  Overview of Health behavior concepts  Applications of personality theory to health  Basic attitude theory, self-regulation, self-efficacy  General Social-Cognitive / Affective Models  Judgments of vulnerability, risk estimation:  Self-awareness, "automaticity" and Cognitive Escape.  Socio-economic Status, race / ethnicity, and health.  Psychoimmunology: affect, coping, interventions  Policy, Economic and Political Influences on Health  Spirituality, happiness, mindfulness & well-being

5 Health behavior & behavioral medicine concepts  CHD as core example of health behavior process  Behavioral variables Smoking Dietary  Affect / stress Anger Chronic arousal, Depression  Physical process Stress or Arousal (“allostatic load”) Hypothalamic-Pituitary-Adrenal axis (HPA axis) Immunomodulation, inflammation.   Exercise Sleep, etc.

6 Personality theory & health / health behavior  Stable, individual differences:  Direct effects “Type A” personality & chronic arousal “Negative affectivity” and immune (or behavioral) effects “Neuroticism” (versus optimism?) and common factor in disease vulnerability  Indirect effects Sensation seeking and risk taking Conscientiousness and precautionary behavior Impulsivity  risk taking, perceived vulnerability Temperament and relative balance of inhibition v. activation (“Bis – Bas”)

7 Basic attitude theory, self-regulation  Simple utility models of behavior  Outcome expectancies  Beliefs x values  intentions  More complex attitude theories  Perceived vulnerability to health threats  Risk estimation  Health Belief models  “Action Identification”, Autonomous Regulation and similar social-cognitive models  Regulatory models  Self-efficacy expectancies  Cybernetic / feedback models

8 Social-Cognitive / Affective Models  “Dual Process” models  Affect v. cognitions  Impulse and Self-Control  Cognitive capacity and self-regulation of impulse  Classic self-regulation & social cognitive models  Goals, values, behavioral dispositions and behavioral self-regulation  Self-efficacy (again)  Health protection motivation Perceived vulnerability Outcome expectancies Efficacy expectancies

9 Judgments of vulnerability  Perceived threat  Cognitive heuristics and risk estimation  Perceived control and vulnerability judgments  Motivated risk perception  Affect (“need states”) and judgments of health risks  Realistic & unrealistic optimism  Unrealistic optimism and mental health  Dispositional optimism and immune function / health status  Optimism (realistic or unrealistic) and risk behavior

10 Self-awareness, "automaticity" and health  Controlled versus automatic processing  The limitations of conscious controls over behavior  “Mindlessness” and automaticity  Anchoring effects  Automatically activation: Cognitive processes Behavioral “scripts”  “Mindfulness” interventions (“making the unconscious conscious…”)  Cognitive Escape and strategic mindlessness

11 Social group processes  Core dimensions of society & health:  Socio-economic Status The robust effect of the SES gradient Increasing SES stratification and health  Minority group stress Stress, helplessness, anger and immune functioning Sexual orientation, stress, disfranchisement & health  Race / ethnicity  Mechanisms  Physical barriers Health care access “Food deserts”  Subordination and immune function

12 Psychoimmunology  Some basic immune system features  Stress, affect, coping and immune status  Reviews of effects Marital stress Bereavement Experimental stress induction  Immune functioning and, e.g., CHD  Psychological variables and immune status  Self-perception “Self-discrepancy” Self-efficacy  Thought suppression  Ψ Interventions

13 Policy, Economic and Political Influences  Industry & politics and health  The Oreo ® and obesity  Tobacco / drug / alcohol policy  Can health policy shape behavior?  Models of environmental influence  The “built environment” and health  “Thin French women”: culture and health

14 The big picture: Spirituality, happiness, mindfulness & well-being  Spirituality  Are religious influences on health “real”?  Subjective spirituality and health  Happiness  Positive coping  “Happiness training” and health  Mindfulness  Stress reduction  Coping & health  Well-being  Personal autonomy and “eudaimonic well-being”  Quality of life

15 General Approaches to Health Research: Evolving conceptions of mind  body Core construct: Stressor, pathogen Internal Process Health Outcome 1.General process: mediating models What explains or accounts for a stress  outcome effect Basic theory development & testing 2.Individual differences: moderating models Establish “boundary conditions” of effect or theory Specify sub-population characteristics of an effect Descriptive or theory-based: important to specify in advance for efficacy trials.

16 Mediating (and additive) models Environ- mental change, Adaptation syndrome Arousal / “allostatic load”, inflammation CHD Developmental changesDevelopmental changes SES, culture, etc.SES, culture, etc. Individual stressIndividual stress Ψ changeΨ change Health behavior: Diet, smoking… Ψ ; distress, helplessness Physiological: Corticosteroids, pro-inflammatory cytokines, HPA activation Lipids, insulin section & “metabolic syndrome” Ψ ; distress, helplessness Physiological: Corticosteroids, pro-inflammatory cytokines, HPA activation Lipids, insulin section & “metabolic syndrome”

17 Mediating (and additive) models Environ- mental change, Adaptation syndrome Arousal, inflammation Arousal, inflammation CHD Psych. Process Health behavior: diet, exercise, smoking… Self-efficacy Helplessness / depression Social isolation Self-efficacy Helplessness / depression Social isolation

18 Basic mediating models in health behavior Stress Illness Immune function Exposure to pathogens Arousal (coritco- steroids) Negative health behavior

19 Health models with structural exogenous variables Stress Illness “Allostatic load” / Immune function Negative health behavior Exposure to pathogens Socio- economic status Structural & cultural barriers to health care

20 Basic moderating model Stress Health status Immune function Health behavior Interaction of stimulus by Ψ resources “ Optimism”, “hardiness”, social support

21 Direct effects of Ψ on health  Psychoimmunology; Adar’s work on affect, learning and immune function  Classical conditioning models:  immune status, tolerance, withdrawal, placebo effects  Etiology: arousal or affective effects on health  stress responses, arousal, and cardio-vascular health  bereavement and health: see House on social ties and mortality  affect (depression), self-concept, optimism, “sense of coherence” and immune function  Specific stressors and obesity Key issue: articulation of complex relations among CNS, ANS, Immune, and other systems.

22 Indirect effects of Ψ on health  “Health behaviors”: risks, protective behaviors [primary prevention] Individual social / cognitive models of... Self-regulation (self-awareness, self-monitoring, self- efficacy) Health information processing Self-perception and decision making: optimism (realistic or unrealistic) readiness to (“stage of”) change risk estimation (normative and non-normative) change motivation, intrinsic – extrinsic motive Affective state, Alcohol & drug use Social support

23 Indirect effects of Ψ on health; Health behaviors Group-level variables controlling exposure, definition, and availability of (un)healthy behavior social norms and/or socially structured rewards and punishments gender, age, cultural group effects; “X”, smoking, etc. models of (un)healthy behavior; processes of modeling influences relations of individuals/groups with health “system”; providers, govt., schools, etc. Cultural level variables Economic / corporate incentives for (un)healthy behaviors Main effects of socio-economic disparity Main effects of ethnicity(“objective” status?)

24 Illness related behaviors 2 nd / 3 rd prevention  Key steps:  Recognition of health problems  Definition of “disturbance” or problem  Treatment or help seeking  Recognition of a health threat  surveillance & early detection  basic health information approaches  Perceived vulnerability & susceptibility: Core precursor of virtually all health models Weinstein: core Ψ variables (controllability) Khaneman: Relative irrationality of risk perception  interpretation of symptoms or signs; implicit health models Health belief model  Info about health threat  “Cues to action” Health cognitions

25 Illness related behaviors 2 nd / 3 rd prevention  Definition of “disturbance” or problem  interpretation of symptoms or signs; implicit health models Health belief model  Info about health threat  “Cues to action” Health cognitions  outcome expectancies for health / illness behavior  “adaptation level” and drifting criteria for diagnosis  Causal attribution models

26 Illness related behaviors 2 nd / 3 rd prevention  Treatment or help seeking  Health belief / health barrier models: approach of treatment source  Individual and group differences in efficacy for behavior change  Coping models: instrumental v. affective coping  Socio-cultural variables in treatment response  adherence to treatments


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