1. Volume 24, Issue 2, Pages 81-89 (June 2017)
Lung injury induced by Bisphenol A: A food contaminant, is ameliorated by selenium supplementation 
Azza S. Abedelhaffez, Ebtihal A. Abd El-Aziz, Mohamed A. Abdel Aziz, Asmaa M. Ahmed 
Pathophysiology 
Volume 24, Issue 2, Pages (June 2017)
DOI: /j.pathophys
Copyright © 2017 Elsevier B.V. Terms and Conditions

2. Fig. 1
(A& B) Mean lung homogenate levels of MDA and SOD of the studied groups.
Data are displayed as mean±SEM of eight rats.
*P<0.05 as compared to control group.
# P<0.05 as compared to BPA group.
Pathophysiology  , 81-89DOI: ( /j.pathophys )
Copyright © 2017 Elsevier B.V. Terms and Conditions

3. Fig. 2
Representative RT-PCR detection of IL-18 mRNA fragments in lung tissue homogenates of the studied groups. β-actin was used in parallel as an internal control.
Pathophysiology  , 81-89DOI: ( /j.pathophys )
Copyright © 2017 Elsevier B.V. Terms and Conditions

4. Fig. 3
Representative Western blotting analysis of FSTL1 in lung tissue homogenates of the studied groups. β-actin was used in parallel as an internal control.
Pathophysiology  , 81-89DOI: ( /j.pathophys )
Copyright © 2017 Elsevier B.V. Terms and Conditions

5. Fig. 4
Representative Western blotting analysis of ADAMTS5 in lung tissue homogenates of the studied groups. β-actin was used in parallel as an internal control.
Pathophysiology  , 81-89DOI: ( /j.pathophys )
Copyright © 2017 Elsevier B.V. Terms and Conditions

6. Fig. 5
Histopathological changes in lung tissue from rats of the studied groups: A),B) control and Se groups revealed normal alveoli and interstitial tissue. C) BPA group revealed thickening of the alveolar walls by heavy chronic inflammatory cellular infiltrate (lymphocytes) and fibrosis. D) BPA+ Se group revealed significant decrease of the lung damage with reduction in the chronic inflammatory cellular infiltrate and fibrosis.
Pathophysiology  , 81-89DOI: ( /j.pathophys )
Copyright © 2017 Elsevier B.V. Terms and Conditions

7. Fig. 6
Immunohistochemical expression of FSTL1 & ADAMTS5 of the studied groups (400x): A) & B) Weak FSTL1 expression in control and Se groups. C) Strong FSTL1 cytoplasmic expression in alveolar epithelium and fibrotic areas in alveolar wall of BPA group. D) Moderate FSTL1 cytoplasmic expression in alveolar epithelium of BPA +Se group. E) & F) Moderate ADAMTS5 expression in bronchial smooth muscle of control and Se groups. G) Weak ADAMTS5 expression in bronchial smooth muscle of BPA group. H) Strong ADAMTS5 expression in bronchial smooth muscle of BPA +Se group. I) & J) Moderate ADAMTS5 cytoplasmic expression in alveolar epithelium of control and Se groups. K) Weak ADAMTS5 cytoplasmic expression in alveolar epithelium of BPA group. L) Strong ADAMTS5 cytoplasmic expression in alveolar epithelium of BPA +Se group.
Pathophysiology  , 81-89DOI: ( /j.pathophys )
Copyright © 2017 Elsevier B.V. Terms and Conditions

8. Fig. 7
(A &B) Mean FSTL1 and ADAMTS5 expressions of the studied groups.
Data are displayed as mean±SEM of eight rats.
*P<0.05 as compared to control group.
# P<0.05 as compared to BPA group.
Pathophysiology  , 81-89DOI: ( /j.pathophys )
Copyright © 2017 Elsevier B.V. Terms and Conditions