Opener A “Smart Drug”
Figure 17.1 One Gene, One Enzyme (Part 1)
Figure 17.1 One Gene, One Enzyme (Part 2)
Figure 17.2 Hemoglobin Polymorphism
Figure 17.3 Genetic Diseases of Membrane Proteins (Part 1)
Figure 17.3 Genetic Diseases of Membrane Proteins (Part 2)
Figure 17.3 Genetic Diseases of Membrane Proteins (Part 3)
Figure 17.3 Genetic Diseases of Membrane Proteins (Part 4)
Figure 17.4 Prion Proteins
Figure 17.5 A Fragile-X Chromosome at Metaphase
Figure 17.6 Strategies for Isolating Human Genes
Figure 17.7 RFLP Mapping (Part 1)
Figure 17.7 RFLP Mapping (Part 2)
Figure Methylcytosine in DNA Is a “Hot Spot” for Mutagenesis (Part 1)
Figure Methylcytosine in DNA Is a “Hot Spot” for Mutagenesis (Part 2)
Figure 17.9 The CGG Repeat in the Fragile-X Gene Expands with Each Generation
Figure Genetic Screening of Newborns for Phenylketonuria (Part 1)
Figure Genetic Screening of Newborns for Phenylketonuria (Part 2)
Figure DNA Testing by Allele-Specific Cleavage
Figure DNA Testing by Allele-Specific Oligonucleotide Hybridization (Part 1)
Figure DNA Testing by Allele-Specific Oligonucleotide Hybridization (Part 2)
Figure A Cancer Cell with Its Normal Neighbors
Figure Dividing Cells Are Especially Susceptible to Genetic Damage
Figure Oncogene Products Stimulate Cell Division
Figure The “Two-Hit” Hypothesis for Cancer
Figure Tumor Suppressor Gene Products Inhibit Cell Division and Cancer
Figure Multiple Mutations Transform a Normal Colon Epithelial Cell into a Cancer Cell (1)
Figure Multiple Mutations Transform a Normal Colon Epithelial Cell into a Cancer Cell (2)
Figure Multiple Mutations Transform a Normal Colon Epithelial Cell into a Cancer Cell (3)
Figure Strategies for Killing Cancer Cells
Figure Gene Therapy: The Ex Vivo Approach (Part 1)
Figure Gene Therapy: The Ex Vivo Approach (Part 2)
Figure Two Approaches to Sequencing DNA
Figure The Human Genome
Figure Is This the Future of Medicine?
Figure Proteomics (Part 1)
Figure Proteomics (Part 2)
Table 17.1 Human Cancers Known To Be Caused by Viruses