Neuroretinitis in patients with multiple sclerosis Kyle E Williams, MD, Lenworth N Johnson, MD  Ophthalmology  Volume 111, Issue 2, Pages 335-340 (February 2004) DOI: 10.1016/S0161-6420(03)00663-8

Figure 1 Patient 1. Right fundus showing hemimacular “star” exudates. Ophthalmology 2004 111, 335-340DOI: (10.1016/S0161-6420(03)00663-8)

Figure 2 A, T2 weighted brain magnetic resonance imaging (MRI) scan of patient 2 showing the occipital region increased signal intensity (arrow) of tumefactive multiple sclerosis. B, T2 weighted brain MRI scan 1 week later, and after intravenous corticosteroid treatment, showing regression of the hyperintensity. Ophthalmology 2004 111, 335-340DOI: (10.1016/S0161-6420(03)00663-8)

Figure 3 A, Occipital brain lesion biopsy results of patient 2 showing gray matter hypercellularity and perivascular lymphocytic infiltrate (inset) compatible with acute multiple sclerosis. B, White matter lymphocytic infiltrate and hypercellularity from the occipital lesion brain biopsy in patient 2. Ophthalmology 2004 111, 335-340DOI: (10.1016/S0161-6420(03)00663-8)

Figure 4 Left fundus of patient 3 showing optic disc edema with exuberant peripapillary and macular exudates. Ophthalmology 2004 111, 335-340DOI: (10.1016/S0161-6420(03)00663-8)

Figure 5 Left fundus of patient 3 showing optic disc edema with peripapillary exudates and macular “star” exudates. Ophthalmology 2004 111, 335-340DOI: (10.1016/S0161-6420(03)00663-8)

Figure 6 Patient 3. Left fundus fluorescein angiogram showing diffuse disc leakage in early (A) and late (B) frames. Ophthalmology 2004 111, 335-340DOI: (10.1016/S0161-6420(03)00663-8)