Cytogenetic and molecular events in adenoma and well-differentiated thyroid follicular- cell neoplasia  Paola Caria, Roberta Vanni  Cancer Genetics and.

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Cytogenetic and molecular events in adenoma and well-differentiated thyroid follicular- cell neoplasia  Paola Caria, Roberta Vanni  Cancer Genetics and Cytogenetics  Volume 203, Issue 1, Pages 21-29 (November 2010) DOI: 10.1016/j.cancergencyto.2010.08.025 Copyright © 2010 Elsevier Inc. Terms and Conditions

Fig. 1 Tumors originating from thyroid hormone–producing cells. Cancer Genetics and Cytogenetics 2010 203, 21-29DOI: (10.1016/j.cancergencyto.2010.08.025) Copyright © 2010 Elsevier Inc. Terms and Conditions

Fig. 2 Common cytogenetic numerical changes in adenoma. A representative G-banded karyotype with 54 chromosomes is shown. Cancer Genetics and Cytogenetics 2010 203, 21-29DOI: (10.1016/j.cancergencyto.2010.08.025) Copyright © 2010 Elsevier Inc. Terms and Conditions

Fig. 3 Common cytogenetic structural changes in benign thyroid proliferation. A partial karyotype showing a t(5;19)(q13;q13) is shown. Cancer Genetics and Cytogenetics 2010 203, 21-29DOI: (10.1016/j.cancergencyto.2010.08.025) Copyright © 2010 Elsevier Inc. Terms and Conditions

Fig. 4 Schematic representation of chromosome abnormalities leading to fusion genes in papillary thyroid carcinoma (a–c), adenoma, and follicular carcinoma (d). Cancer Genetics and Cytogenetics 2010 203, 21-29DOI: (10.1016/j.cancergencyto.2010.08.025) Copyright © 2010 Elsevier Inc. Terms and Conditions

Fig. 5 Schematic representation of bacterial artificial chromosome clones flanking RET (a, left) and PPARγ (b, left) used to reveal gene disruption via the fluorescence in situ hybridization break-apart probe strategy (a and b, right). Arrows point to the break-apart signals. Cancer Genetics and Cytogenetics 2010 203, 21-29DOI: (10.1016/j.cancergencyto.2010.08.025) Copyright © 2010 Elsevier Inc. Terms and Conditions

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