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1 Mentoring the Mentor Stuart White, DC, DACBN, CCN Whole Health Associates 1406 Vermont Houston, Texas 77006 713/522-6336

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Presentation on theme: "1 Mentoring the Mentor Stuart White, DC, DACBN, CCN Whole Health Associates 1406 Vermont Houston, Texas 77006 713/522-6336"— Presentation transcript:

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2 1 Mentoring the Mentor Stuart White, DC, DACBN, CCN Whole Health Associates 1406 Vermont Houston, Texas /

3 2 Mentor goals: Ò To declare what is possible and establish a commitment to that possibility Ò Address personal and professional barriers limiting the ability to serve Ò Evolution of vision/mission/ethics that drive success Ò Create immediate action steps to apply learning and growth Ò Construct the round table of applied trophologists

4 3 Mentoring the mentor: Ò Who are the mentors? – Practitioners Ò Who are we mentoring? – Patients and GAP Ò What’s the purpose? – Optimized life Ò How does it work? – Whatever you learn you teach someone else (anyone else) Ò Who’s is included? – Self selection, you pick yourself

5 4 Mentoring the mentor: Ò Each participant attends monthly teleconferences (1 hour in duration, 4 th Thursday of month) creating a round table discussion/exploration of the dynamics and details of a nutrition-based holistic practice Ò Each participant chooses how to convey the notes and information to their world and community – no information squandering

6 5 Review - Distinguish yourself It is more apparent why people are choosing alternative health care professionals who specialize in a functional approach No matter you specialty or technique you must distinguish yourself as an expert – people are just seeking to understand and they need you to do so Typically in the healthcare industry people are receiving shallow answers that leave them puzzled with the mystery of “Why is this happening to me?” and “ What can I do about it?” Trends research over 10 years ago identified a number of factors essential to being successful in the nutritional field – one of those was establishing yourself as an expert

7 6 Review - Explanation as hope The practitioner’s ability to explain health issues and therapeutic outcomes creates an inflation of understanding in the patient which feels like hope Today in the professional world there is so much avoidance of ‘giving false hope’ that often we end up offering little hope at all I propose another model that bolsters hope and expectation and subsequently practices accountability as to whether the therapeutic endeavors are achieved or not As long as the hope that has been instilled is revisited and acknowledged as being accomplished or not the betrayal of false hope can be avoided So as an example, if a practitioner was describing the potential for nutritional intervention through supplements and diet modification to improve the lipid profile, then s/he would need to revisit to success or failure of the experiment within a reasonable period of time Our community is starving for legitimate hope, as a starting place, as empowerment to begin, as an idea to act upon There is genius in hope

8 Mentor Considerations HPA Axis and Immune Cytokine Signaling

9 Seven Pillars Unified Mechanisms of Health Promoting Physiology

10 9 7 Pillars of Healing 7 Unified Mechanisms of Health  Endocrine/Hormonal  Glycemic Management  pH Bioterrain  Immuno-Inflammatory  Circulatory Status  Digestive Potency  Cellular Vitality

11 Normal Miracle Endocrine Hormonal Glycemic Management pH Bioterrain Minerals Immune Inflammatory Circulatory Status Digestive Potency Cellular Vitality

12 Eternal truth - Fulfillment is not a goal to achieve, But always a by- product of sacrifice

13 12 1 -The Endocrine Axis  Most powerful system to activate the rest of body  7 glandular levels  PMG’s first, lifestyle modification second, herbs third, HRT last

14 13 StressorsHormonal/endocrine adaptationGlandular fatigue & imbalance Depletion of organ reserve and nutrient/mineral substrates Reduced homeostatic mechanisms Enhanced physiology/personality #1 Core Physiologic Principle Stress hyper/hypo reactivity Altered psychoneuroimmunologic mechanisms Restored adaptive mechanisms Symptoms – physical/personality modulation Increased glandular strength/resilience Disease diagnosis – chronic progression Medical Intervention – Drugs & Surgery Increased organ reserve – repletion of substrates Death Nutrient repletion – target fortification

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17 16 Symplex F/M(3,3) Hypothalmex/us(1,1) Black Currant Seed(1,1)

18 17 The Stress Model  The HPTA is at the heart of the body’s ability to respond to the environment  Cortisol elevation is the result of Corticotrophin Releasing Hormone (CRH) arising from the parvocellular neurons of the paraventricular nucleus (PVN) - this is the ‘master’ stress hormone released in response to the perception of stress  Stressful stimuli are generalized as:  Physical – pain, trauma, infection, hypotension, exercise, hypoglycemia  Psychological – bereavement, fear, personal loss, anger (the perception that God is not in control – something is wrong)  CRH is released into the portal circulation of the Median Eminence and is carried by venous blood to the corticotroph cells of the anterior pituitary where it binds to the cell surface receptors stimulating the release of Adrenocorticotropic Hormone (ACTH)  ACTH reaches the adrenal cortex stimulating the synthesis of Cortisol (glucocorticoid) and also androgenic hormones like androstenidione and DHEA (both may convert to testosterone and DHT in peripheral tissues )

19 18 The Stress Model  Cortisol maintains blood glucose during stressful ‘fight or flight’ challenges so that as more metabolic fuel is consumed a critical amount is maintained for brain function and to support the activated survival organs such as the heart, lungs, and skeletal muscle with renewable supply of fuel  Cortisol also participates with Aldosterone (mineralocorticoid) in driving sodium reabsorption from the renal tubules conserving electrolytes and water within the vasculature to provide blood and perfusion pressures to vital organs  Cortisol concentrations rise until it effects negative feedback on the CRH neurons and the pituitary corticotrophs to return blood levels to normal preventing prolonged elevations of CRH, ACTH and cortisol  Chronic stress and maladapted responses to stress alters this mechanism and causes longterm cortisol dysregulation and even ‘cortisol resistance’

20 19 Hypothalamus Blood/Brain Barrier Cortisol “Glucocorticoid” Parvocellular neurons of the Paraventricular Nuclei release CRH in response to perceived stress Corticotrophin Releasing Hormone ACTH Adrenocorticotropic Hormone Paraventricular Nuclei Median Eminence Neurohypophysis Adrenal Cortex Anterior Pituitary “Corticotrophs” Cortisol elevation provides negative feedback to paraventricular nuclei decreasing CRH Promotes Aldosterone release “mineralocorticoid” Androgenic hormones Androstenidione, testosterone, DHT, progesterone Cortisol Resistance Adrenal Complex Tyrosine Reduce cortisol resistance Cortisol Activation

21 20 Modulating Cortisol  Symplex, Hypothalmex/us – HPA general support  Androgen up-regulation  Adrenal Complex – 2-4/day licorice & rehmannia  Allergen removal  Drenamin – 6/day  Dessicated Adrenal – 2-4/day for acute activation  Eleuthero – 2-4/day  Withania Complex – 2/day  Vitanox 2-4/day  Detoxification  Change of thinking  Neuro-emotional release

22 21 Modulating Cortisol  Adrenal Complex (1-2) has exploded on the scene and represents another MediHerb homerun  Introduced in 02/09 it has backordered multiple times as Americans have grasped its value as an idea whose time has come  Licorice (250 mg of 7:1 extract) contains 25 mg of glycyrrhizin the active component that assists cortisone (a less active storage form of cortisol) to convert to cortisol (more active form)  Rehmannia (150 mg of 5:1 extract) provides immune modulation  Expect modulation in WHR, concentration, sleep quality, reduced muscle tension, relaxability, reduced anxiety  Contraindicated when hypertension results

23 22 Gut Brain Interface

24 23 The Bidirectional Gut-Brain Axis Grenham S, Clarke G, Cryan JF, Dinan TG. Brain-gut-microbe communication in health and disease. Front Physiol. 2011;2:94. Epub 2011 Dec 7. PubMed PMID: ; PubMed Central PMCID: PMC Brain-gut-microbe communication in health and disease.

25 24 The GALT: Gut Associated Lymph Tissue Forsythe P, Sudo N, Dinan T, Taylor VH, Bienenstock J. Mood and gut feelings. Brain Behav Immun Jan;24(1):9-16.Mood and gut feelings. Ochoa-Repáraz J, Mielcarz DW, Begum-Haque S, Kasper LH. Gut, bugs, and brain: role of commensal bacteria in the control of central nervous system disease. Ann Neurol Feb;69(2):240-7.Gut, bugs, and brain: role of commensal bacteria in the control of central nervous system disease. Korecka A, Arulampalam V. The gut microbiome: scourge, sentinel or spectator? J Oral Microbiol. 2012;4.The gut microbiome: scourge, sentinel or spectator? 70% of our immune cells reside in the GI tract. The development of the intestinal immune system is largely dependent upon exposure to microorganisms. The gut produces ¾ of the body’s neurotransmitters. The gut has greater metabolic activity than the liver.

26 25 Microbiota Regulate HPA-Axis Development Commensal microbiota regulate the development of the HPA axis. “The series of events in the gastrointestinal tract following postnatal microbial colonization can have a long-lasting impact on the neural processing of sensory information regarding the endocrine axis.” This concept, based on in vivo findings [in mice], provides evidence of a novel link between indigenous microorganisms and the nervous system and shows a new aspect of the brain-gut axis. Sudo N, Chida Y, Aiba Y, Sonoda J, Oyama N, Yu XN, Kubo C, Koga Y. Postnatal microbial colonization programs the hypothalamic-pituitary-adrenal system for stress response in mice. J Physiol Jul 1;558(Pt 1):263-75Postnatal microbial colonization programs the hypothalamic-pituitary-adrenal system for stress response in mice.

27 26 Commensal Microbiota Drives Immune Homeostasis This tissue has the dual task of selectively absorbing nutrients from the intestinal lumen, while preventing microbial entry, infection, or immune activation. We are so focused on the immune system responding to things, that we forget that 99.9% of the time, its job is NOT to respond to things. “The gut handles more antigenic material in a single day than the rest of the immune system processes its entire lifetime.” Michael Ash Handley C. Should auld acquaintance be forgot… EMBO Reports Vol 5, No 12, 2004Arrieta MC, Finlay BB. The commensal microbiota drives immune homeostasis. Front Immunol. 2012;3:33. The commensal microbiota drives immune homeostasis.

28 27 A Closer Look Artis D. Epithelial-cell recognition of commensal bacteria and maintenance of immune homeostasis in the gut. Nat Rev Immunol Jun;8(6): Epithelial-cell recognition of commensal bacteria and maintenance of immune homeostasis in the gut. IEC Nucleus M Cell Glycocalyx DC Lymphoid Follicle Microvillar Extension Peyer’s Patch Commensal and pathogenic microorganisms Antimicrobial peptides Transcytosed microbes are acquired by DCs which activate adaptive immune response Direct sampling of microbial antigen in lumen

29 28 Paracellular Transport Artis D. Epithelial-cell recognition of commensal bacteria and maintenance of immune homeostasis in the gut. Nat Rev Immunol Jun;8(6): Epithelial-cell recognition of commensal bacteria and maintenance of immune homeostasis in the gut.

30 29 Transcellular Pathways Degradative route 90% Non-degradative route <10%

31 30 The Gut in Action Artis D. Epithelial-cell recognition of commensal bacteria and maintenance of immune homeostasis in the gut. Nat Rev Immunol Jun;8(6): Epithelial-cell recognition of commensal bacteria and maintenance of immune homeostasis in the gut. Intestinal lumen Epithelium Lamina propria

32 31 Just the Right Balance Antigen Pathogen/food-derived lipid/protein/nucleic acid ^^^^^ TLR4/CD14/MD2 T cell (Th0) Antigen presentation Th2 Th1 Treg Differentiation IL-10 TGF-β Il-4 IFN-γ IL-12 IFN-γ TNF-α IL-1β IL-6 IL-8 DC Macrophage NK cell Cell-mediated adaptive immune response B cell Humoral adaptive immune response Innate immunity Macrophage Inflammatory cytokines Interferon Phagocytosis Thomas S, Przesdzing I, Metzke D, Schmitz J, Radbruch A, Baumgart DC. Saccharomyces boulardii inhibits lipopolysaccharide-induced activation of human dendritic cells and T cell proliferation. Clin Exp Immunol Apr;156(1):78-87Saccharomyces boulardii inhibits lipopolysaccharide-induced activation of human dendritic cells and T cell proliferation.

33 32 TJ Disassembly Resulting in Unremitting Immune Activation via Leaky Gut Fasano A. Leaky gut and autoimmune diseases. Clin Rev Allergy Immunol Feb;42(1):71-8.Leaky gut and autoimmune diseases.

34 33 Leaky Gut’s One-Two Punch Maes M, Kubera M, Leunis JC. The gut-brain barrier in major depression: intestinal mucosal dysfunction with an increased translocation of LPS from gram negative enterobacteria (leaky gut) plays a role in the inflammatory pathophysiology of depression. Neuro Endocrinol Lett Feb;29(1): The gut-brain barrier in major depression: intestinal mucosal dysfunction with an increased translocation of LPS from gram negative enterobacteria (leaky gut) plays a role in the inflammatory pathophysiology of depression. O&NO damage Translocation of microbial (LPS) or food antigens (gluten, casein) through gut Leaky gut Antibodies confuse self with pathogenic antigens (antigen mimicry) Antibodies and autoimmunity Local and system inflammation

35 34 Food Allergens

36 35 Determining Food Allergies  Blood type sensitivities Eat For Your Blood Type, D’Amatto  Most food allergies are delayed sensitivity reactions – difficult to objectively determine  Elisa Act lymphocyte response assay Dr. Russell Jaffe Serammune Labs, Virginia, 800/  Elimination is the most accurate and labor intensive - 2 week elimination then reintroduce and watch for 4 days for reactions  Histaminic Reactions (rash, red eyes, serous secretions) vs. Immune Activity (fever, catarrhal, lymphatic congestion, aching)  Basic 4 allergies that most complicate healing process – wheat (gluten), corn, soy, milk (casein) –Additionally suspect chocolate, peanuts, tomatoes, beef

37 36 Food Allergies – Now & Later Immediate response within hours or next day Delayed response onset 2-7 days later Histaminic Immunological – viral, bacterial, parasitic Red, burning eyes, serous secretions (clear) Colds & Flu – WBC mediated response Tiredness, sleepinessAchiness HeadachesCatarrhal, phlegm (colored) Mood changes, irritabilityFever Rashes, hivesEczema Nausea, cramps, diarrheaEmesis Loss mental accuity Elevated C-reactive protein, SED rate, AA:EA ratio

38 37 Allergic Events schematic Blood/lymph fluids Tissue/cell structures Gut lining Gut lumen Allergens Foreignness Immune response Viron Infectious process Irritation leading to infestation

39 38 Generalization of allergen  Milk allergy is primarily casein protein intolerance commonly seen in respiratory and atopic symptoms  Wheat allergy is primarily a gluten protein intolerance commonly effecting GI symptoms and hyper tension & siderosis  Corn allergy is primarily a zein protein intolerance commonly effecting neurological symptoms  Soy allergy is more acquired and therefore can be unlearned commonly effecting acne rosacea and paranasal rashes  Zypan or Betaine HCL (2-3/meal) will reduce food allergen effects

40 39 Plenty of Mediators to Measure Maes M et al. Endocrinol Lett. 2008;29(1): ; Haroon E et al. Neuropsychopharmacology. 2012;37(1):

41 Many nutrients & botanicals inhibit the activation of NF-KappaB inflammatory gene activation. Omega 3 EFA’s & GLA Grape Seed Extract Vitamin D Propolis Curcumin/Turmeric Resveratrol Lipoic Acid Cholagogues Green Tea Vitamin C Complex Rosemary

42 41 Immuno-inflammatory Influence

43 42 Innate & Acquired Immunity  Primary roles of the healthy immune system are: Identify potentially injurious and infectious substances Distinguish self antigens (non-threatening) from non-self (threatening) Assess the potential level of threat posed by infectious, toxic, or non-self antigens Mount a response that is appropriate to the level of threat Repair any damage that ensues from adversarial encounters  Too much response = inflammatory cascades  Too little response = tolerance of danger  WBC is optimal 6-8, outside optimal range may suggest acute or chronic immune burden, under 4 indicates bone marrow fatigue

44 43 Immune System – 2 Parts  Generally recognized that there are 2 parts of the immune system  Innate Immune System – Inborn initial response to eliminate microbes and infections, immediately or within hours – it is not in any locale or organ, it is in the WBC »Each cell is equipped with different mechanisms that allow it to attack and eliminate pathogens from the body demonstrating immune versatility »Non-specific defense against pathogens, activates the complement system of inflammatory response »Identifies self vs. non-self, complement system triggers inflammation and identifies foreign substances, and activates the adaptive immune system –Innate Immune Cells include: »Mast Cells »Natural Killer Cells »Phagocytes – Monocytes, Macrophages, Dendritic cells »Ranulocytes – Neutrophils, Eosinophils, Basophils  Adaptive Acquired Immune System – Learned response precisely addressing threat requiring 5-7 days for adaptive immune modulation to reach full activity and specific lymphocyte presence »Results in TH1 cellular phagocytosis or TH2 humoral antibodies »TH1 responds to living things bacteria, fungus, virus »TH2 responds to non-living things (and parasites) including food, pollens, bad fats, heavy metals

45 44 Common TH1 & Th2 Cytokines  TH1  Il-12  IFN – gamma  TNF – alpha  IL-2  GM – CSF  TH2  IL-4  IL-5  IL-10  IL-13  IL-1 and IL-6 (and others) can show both TH1 and Th2 influences

46 45 Efflux pumps and bacteria  Milk thistle and Berberine have been found to inhibit the active efflux pump in certain bacteria (Staph) and thus inhibit the germ’s resistance to remediation by drugs and theoretically host immune response as well

47 46 Cytokines – Immune Messages  Immune response results in the release of cytokines meant to direct local and distant immune function  These cytokine messenger molecules also drive HPA status and thus determine global brain status  Cytokines subsequently cause the release of WBC inflammatory mediators to direct the inflammatory process of repair  Therefore immune status and activity determine HPA/brain settings  Hypervigilant or depressed immune states reflect in brain states

48 47 Someone you met Immuno-competent Cell (Macrophage) Something you et IL-1β, TNF-α, IL-6 NE GCs Ach Cytokines signal in the brain: -Saturable transporters -BBB leakiness -Localized production -Binding to receptors on afferent nerve fibers -Recruitment of activated cells Cytokines and DOCs NF-κB Raison CL, Lowry CA, Rook GA.. Arch Gen Psychiatry Dec;67(12):

49 48 Immune Tolerance ”Don’t be so Reactive”  If it weren’t for tolerance we would constantly fighting a war with the foreignness everywhere  Complex feedback system developed through reactor and moderator substances activating and suppressing immune/ inflammatory response creating an immune capacity of tolerance  Net reactor chemistry x net moderator chemistry = immune tolerance  Especially strategic to the autoimmune circumstance – goal is to reduce immune burdens and promote immune tolerance and thus reduce immune reactivity  Infections, infestations, toxicities, allergens, injuries, inoculations, etc. create a burden teasing out intolerance and excessive reactions

50 49 Immune mechanisms – schematic Nuclear Membrane Cell Membrane Cytoplasm Nuclear genetic code Gene Activation Foreignness Tolerance mechanisms moderating acquired immune activation Reactor mediators Activate acquired immune response s

51 50 Sequential Immune Up-Regulation  Especially under the teeth, diverticulosis, severe infections near or in bone, body cavities like sinus, ears, pelvic, intestinal  Sequential immune bolstering protocols for one month each at therapeutic dosage – “deep cleaning”  Up regulate immune system gradually beginning with Sesame Oil Perles (6/day), followed by Thymex (10/day), then Immuplex (6/day), Congaplex (15/day), Allerplex (15/day), Echinacea (4/day), Astragulus (4/day)  Clear infestations with Zymex II (6/day), Multizyme (4/day), Wormwood Complex (4/day) – also treats mycoplasmic infections  Finally use Chaparral with high concentration of NDGO (strongest known antioxidant) – will clear systemic infection including bowel dysbiosis and infections

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56 Establish a Differential Diagnosis: Table 2.1: Symptoms and Associated Medical Conditions on the MSIDS Map, page 68 “Why Can’t I Get Better?” SymptomsPossible Medical ConditionsLaboratory Testing to Consider Unexplained fevers, sweats, chills, or flushing Lyme disease (chronic and other bacterial, viral, parasitic, and fungal infections) Babesiosis Malaria Brucellosis Hyperthyroidism Hormonal failure (early menopause) Tuberculosis* Non- Hodgkin’s lymphoma* Panic disorders Autoimmune disorders Inflammation CBC with a white cell count CMP with liver functions Giemsa stain and malarial smears Babesia IFA Babesia WA- 1/duncani titers Babesia FISH and PCR Thyroid function tests (TFT’s) Sex hormone levels Chest X-ray/PPD Antinuclear antibody (ANA), Rheumatoid factor (RF) Erythrocyte sedimentation rate (ESR),C-reactive protein (CRP) Cytokine panel

57 Overview: Lyme & Chronic Disease in the US 1) According to the CDC, chronic disease accounts for 70% of the deaths and 75% of the health care costs in the United States 1) According to the CDC, chronic disease accounts for 70% of the deaths and 75% of the health care costs in the United States 2) Lyme disease is the number one spreading vector borne epidemic worldwide, and mimics other chronic diseases accounting for an increased burden of those suffering with chronic illness (CDC figures 2103:10 fold increase in cases ) 2) Lyme disease is the number one spreading vector borne epidemic worldwide, and mimics other chronic diseases accounting for an increased burden of those suffering with chronic illness (CDC figures 2103:10 fold increase in cases ) 3) There is confusion regarding the diagnosis and treatment of LD due to 2 standards of care in the US 3) There is confusion regarding the diagnosis and treatment of LD due to 2 standards of care in the US 4) Defining Chronic Lyme Disease: What is it?-the difference between the “surveillance” definition and “real life” in the doctors office. Proposing a new definition: Lyme-MSIDS (Multi- Systemic Infectious Disease Syndrome) 4) Defining Chronic Lyme Disease: What is it?-the difference between the “surveillance” definition and “real life” in the doctors office. Proposing a new definition: Lyme-MSIDS (Multi- Systemic Infectious Disease Syndrome) 5) Diagnosing Lyme Disease: Problems with serology and seronegative infection 5) Diagnosing Lyme Disease: Problems with serology and seronegative infection

58 Overview: Lyme & Chronic Disease in the US 6) Treating Lyme disease: evidence of persistent/chronic borrelial infection & the need for longer treatment courses 6) Treating Lyme disease: evidence of persistent/chronic borrelial infection & the need for longer treatment courses 7) Chronic infection from Lyme and associated tick-borne diseases drives inflammation. 7) Chronic infection from Lyme and associated tick-borne diseases drives inflammation. 8) Inflammation is the number one common denominator in all chronic disease. 8) Inflammation is the number one common denominator in all chronic disease. 9) Diagnosing and treating the 3 I's: chronic infection, inflammation and immune dysfunction helps individuals recover their health. 9) Diagnosing and treating the 3 I's: chronic infection, inflammation and immune dysfunction helps individuals recover their health. 10) Detoxification also plays an important role in improving clinical outcomes. 10) Detoxification also plays an important role in improving clinical outcomes. 11) A multifactorial model for chronic disease called MSIDS (Multiple Systemic Infectious Disease Syndrome) will be discussed, showing how multiple factors on the MSIDS map can be addressed to help decrease inflammation and reduce disabling symptoms. 11) A multifactorial model for chronic disease called MSIDS (Multiple Systemic Infectious Disease Syndrome) will be discussed, showing how multiple factors on the MSIDS map can be addressed to help decrease inflammation and reduce disabling symptoms.

59 Defining Chronic Lyme Disease: Lyme-MSIDS  Infections: a)Bacterial: Lyme disease, Ehrlichiosis, Bartonella, Mycoplasma, Chlamydia, RMSF, Typhus, Tularemia, Q-Fever, Tick paralysis b) Parasites: Babesiosis and other piroplasms, filiariasis, amebiasis, giardiasis…  c) Viruses: EBV, HHV-6, CMV, W Nile, Heartland v., Powassan encephalitis and other viral encephalopathies  d) Candida and other fungi 2) Immune dysfunction: ANA+, RF+ ↑ HLA DR-4 3) Inflammation: ↑ IL-1, IL-6, TNF-α→ “Sickness syndrome” 4) Toxicity: Multiple Chemical Sensitivity, Environmental Illness, Heavy Metals, Mold, and Neurotoxins 5) Allergies: foods, drugs, environmental… 6) Nutritional & Enzyme Deficiencies/ functional medicine abnormalities in biochemical pathways 7) Mitochondrial dysfunction 8) Psychological disorders 9) Neurological dysfunction 10) Endocrine disorders 11) Sleep disorders 12) ANS dysfunction +/- POTS 13) G.I. disorders 14) Elevated LFT’s 15) Pain syndromes 16) Deconditioning

60 59 Protocol for stealth pathogens Sequential Immune Up-Regulation – graduated immune sparing due to reduction of immune burdens Neuro-endocrine support – HPA support – Symplex F 3 bid, Hypothalamex 1 bid, Black Currant 1 bid Ongoing hemopoietic and lymph support to assist in the processing of cellular debris and infection remediation Final amplification of nutritional therapy to promote immune aggression toward sequestered stealth pathogens This would include the following possibly: Cat’s Claw Complex 2 bid Immuplex 3 bid Sesame Oil 3 bid Ostrophin 2 bid Whey Pro Complete 2 scoops daily (source of IgG found in colostrum) Berberine 2 bid or sourced from Gut Flora or Golden Seal Myrrh tincture 3 droppers twice daily from Weed Botanical Enzymes to promote cellular cleansing – Multizyme 2 tid, Zypan 2 tid Ongoing liver support – Livco 2 bid, betacol 2 bid

61 How do we think without proof - It would be possible to describe everything scientifically, but it would make no sense; it would be without meaning, as if you described a Beethoven symphony as a variation of wave pressure. Albert Einstein

62 61 Principles at work Sufficient clinical observation allows mechanisms to be revealed that will remove the idiopathic mystery of hypertension and return it to a simple physiological modulation and resultant augmentation in function, balance, tissue fortification and promotes healthy genetic expression This allows the symptom resolution to occur as a result of system ‘mosaic’ change, and then of course the downstream events occur The longing in the public is for this sort of detective work to find the cause and make the correction – increasingly food is seen as medicine and people are asking more and more for what foods will change their health patterns

63 Sequential Intervention  By giving hope through discussion of therapeutic rationale and then accountably determine if the therapy had efficacy it is possible to initiate activity that may assist a person to make the changes that result in healing  Sequential intervention and accountable follow-up can show what has worked and what may still need to be employed  Promote an understanding of intervention that creates evolutions in individual physiology and show the effect of that intervention  Allow every condition to become a strategic consideration of possible etiology and therapeutic rationale – people are in search of experts – reveal yourself  The comprehensive nature of nutritional therapy means there is always more physiology to optimize and support leaving an individual constantly refining as long as they wish to further improve their status  If the practitioner is accountable s/he will be allowed to experiment with reasonable ideas

64 63 Change the world It wants to Change the world It wants to


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