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Perspectives in Nutrition 5th ed. Gordon M.Wardlaw, PhD, RD, LD, CNSD

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Presentation on theme: "Perspectives in Nutrition 5th ed. Gordon M.Wardlaw, PhD, RD, LD, CNSD"— Presentation transcript:

1 Perspectives in Nutrition 5th ed. Gordon M.Wardlaw, PhD, RD, LD, CNSD
PowerPoint Presentation by Dana Wu Wassmer, MS, RD

2 Chapter 13: Energy Balance and Weight Control

3 Energy Balance “State in which energy intake, in the form of food and /or alcohol, matches the energy expended, primarily through basal metabolism and physical activity” Positive energy balance Energy intake > energy expended Results in weight gain Negative energy balance Energy intake < energy expended Results in weight loss

4 Energy Balance (Fig. 13-1) Copyright © The McGraw-Hill Companies, Inc. Permission required for reproduction or display.

5 Estimating Kcal Content in Food
Copyright © The McGraw-Hill Companies, Inc. Permission required for reproduction or display. Estimating Kcal Content in Food Bomb calorimeter (Fig. 13-2) Burns food inside a chamber surrounded by water Heat is given off as food is burned The increase in water temperature indicates the amount of energy in the food

6 Macronutrients and Fat Storage
Most fat is stored directly into adipose tissue Body has unlimited ability to store fat (as fat) Limited CHO can be stored as glycogen Most CHO is used as a energy source Excessive CHO will be synthesized into fat (for storage)

7 Macronutrients and Fat Storage
Protein is primarily used for tissue synthesis Adults generally consume more protein than needed for tissue synthesis Excess protein is used as a energy source Some protein will be synthesized into fat (for storage)

8 Macronutrients and Fat Storage
Body prefers to use CHO as energy source Only excess intake of CHO and protein will be turned into fat Fat will remain as fat for storage Physical activity encourages the burning of dietary fat

9 Energy In Vs. Energy Out NEAT Basal Metabolism
Dietary Intake Physical Activity Thermic Effect of food

10 Basal Metabolism The minimum energy expended to keep a resting, awake body alive ~60-70% of the total energy needs Includes energy needed for maintaining a heartbeat, respiration, body temperature Amount of energy needed varies between individuals

11 Influences On Basal Metabolism
Body surface area (weight, height) Gender Body temperature Thyroid hormone Age Kcal intake Pregnancy Use of caffeine and tobacco

12 Physical Activity Increases energy expenditure beyond BMR
Varies widely among individuals More activity, more energy burned Lack of activity is the major cause of obesity

13 Thermic Effect of Food (TEF)
Energy used to digest, absorb, and metabolize food nutrients “Sales tax” of total energy consumed ~5-10% above the total energy consumed TEF is higher for CHO and protein than fat Less energy is used to transfer dietary fat into adipose stores

14 Nonexercise Activity Thermogenesis
Nonvoluntary physical activity triggered by overeating Fidgeting Over eating increases sympathetic nervous system activity Resists weight gain

15 Measurement of Body’s Energy Needs
Direct calorimetry Measures heat output from the body using an insulated chamber Expensive and complex Indirect calorimetry Measures the amount of oxygen a person uses A relationship exists between the body’s use of energy and oxygen

16 Harris-Benedict Equation
Estimates resting energy needs Considers height, weight, age, and gender For men: x(kg) + 5x(cm) - 6.8x(age in yr.) For women: x(kg) + 1.8x(cm) - 4.7x(age in yr.)

17 Sample Calculations Man: 21 yr., 5’10” (171 cm), 155# (70 kg)
x(70kg) + 5x(171cm) x(21) = 1745 kcal/day Woman: 21 yr., 5’10” (171 cm), 155# (70kg) x(70kg) + 1.8x(171cm) - 4.7x(21)= 1536 kcal/day

18 Why Do You Eat? Hunger Appetite Physiological (internal) drive to eat
Controlled by internal body Appetite Psychological (external) drive to eat Often in the absence of hunger e.g., seeing/smelling fresh baked chocolate chip cookies

19 Satiety Regulator The hypothalamus Sympathetic nervous system
When feeding cells are stimulated, they signal you to eat When satiety cells are stimulated, they signal you to stop eating Sympathetic nervous system When activity increases, it signals you to stop eating When activity decreases, it signals you to eat

20 Influences of Satiety (Fig. 13-4a)
Copyright © The McGraw-Hill Companies, Inc. Permission required for reproduction or display.

21 Influences of Satiety (Fig. 13-4b)
Copyright © The McGraw-Hill Companies, Inc. Permission required for reproduction or display.

22 Influenced By Body Composition
Leptin A hormone produced by the adipose tissue Increases with larger fat mass (and decrease desire to eat) Decreases with lower fat mass (and enhance desire to eat) Acts to decrease activity of neuropeptide Y Neuropeoptide Y Increases food intake Reduces energy expenditure

23 Hormonal Influence Endorphins CCK Serotonin
Natural body tranquilizer that can prompt you to eat CCK Along with gastrointestinal distention, decreases hunger (and desire to eat) Serotonin Neurotransmitter that is released as a result of CHO intake High levels appear to decrease desire to eat CHO and induce calmness

24 Hormonal Influence Nutrient receptors In small intestine
Elicit feeling of satiety Communicate with the brain via nerves Inform brain of the presence of nutrients in the small intestine Feeling of satiety with the infusing of CHO or fats in the small intestine

25 Nutrients Influence Presence of energy yielding nutrient registers satiety in the brain Apolipoprotein A-IV on the chylomicrons signals satiety in the brain Absence of these nutrients will signal hunger

26 Why We Eat Appetite is affected by a variety of external forces
Combination of internal and external signals drive us to eat Not a perfect system; desire to eat can be overwhelming

27 What is a Healthy Body Weight?
Based on how you feel, weight history, fat distribution, family history of obesity-related disease, current health status, and lifestyle Current height/weight standards only provide guides

28 Body Mass Index (BMI) The preferred weight-for-height standard
Calculation: Body wt (in kg) OR Body wt (in lbs) x 703.1 [Ht (in m)] [Ht (in inches)]2 Health risks increase when BMI is > 25

29 Estimation of Healthy Weight
For men: 106 pounds for the first 5 feet add 6 pounds per each inch over five feet A man who is 5’10” should weigh 166 lbs. For women: 100 pounds for the first 5 feet add 5 pounds per each inch over five feet A women who is 5’10” should weigh 150 lbs.

30 Obesity Excessive amount of body fat
Women with > 30-35% body fat Men with > 25% body fat Increased risk for health problems Are usually overweight Measurements using calipers

31 Estimation of Body Fat Underwater weighing (Fig. 13-5) Most accurate
Copyright © The McGraw-Hill Companies, Inc. Permission required for reproduction or display. Estimation of Body Fat Underwater weighing (Fig. 13-5) Most accurate Fat is less dense than lean tissue Fat floats

32 Estimation of Body Fat Bioelectrical impedance
Copyright © The McGraw-Hill Companies, Inc. Permission required for reproduction or display. Estimation of Body Fat Bioelectrical impedance Low-energy current to the body that measures the resistance of electrical flow Fat is resistant to electrical flow; the more the resistance, the more body fat you have X-ray photon absorptiometry An X-ray body scan that allows for the determination of body fat Infrared light Assess the interaction of fat and protein in the arm muscle

33 Body Fat Distribution Upper-body (android) obesity--”Apple shape”
Associated with more heart disease, HTN, Type II Diabetes Abdominal fat is released right into the liver Fat affects liver’s ability to clear insulin and lipoprotein Encouraged by testosterone and excessive alcohol intake Defined as waist to hip ratio of >1.0 in men and >0.8 in women

34 Body Fat Distribution (Fig.13-9)
Copyright © The McGraw-Hill Companies, Inc. Permission required for reproduction or display.

35 Body Fat Distribution Lower-body (gynecoid) obesity--”Pear shape”
Encouraged by estrogen and progesterone After menopause, upper-body obesity appears Less health risk than upper-body obesity

36 Overweight and Obesity
Underweight = BMI < 18.5 Healthy weight = BMI Overweight = BMI Obese = BMI Severely obese = BMI >40

37 Juvenile-Onset Obesity
Develops in infancy or childhood Increase in the number of adipose cells Adipose cells have long life span and need to store fat Makes it difficult to loose the fat (weight loss)

38 Adult-Onset Obesity Develops in adulthood
Fewer (number of) adipose cells These adipose cells are larger (stores excess amount of fat) If weight gain continues, the number of adipose cells can increase

39 Causes of Obesity Nature debate
Identical twins raised apart have similar weights Genetics account for ~40% of weight differences Genes affect metabolic rate, fuel use, brain chemistry Thrifty metabolism gene allows for more fat storage to protect against famine

40 Causes of Obesity Nurture debate
Environmental factors influence weight Learned eating habits Activity factor (or lack of) Poverty and obesity Female obesity is rooted in childhood obesity Male obesity appears after age 30

41 Nature and Nurture Obesity is nurture allowing nature to express itself Location of fat is influenced by genetics A child with no obese parents has a 10% chance of becoming obese A child with 1 obese parent has a 40% chance A child with 2 obese parents has a 80% chance

42 Nature Vs. Nurture Those at risk for obesity will face a lifelong struggle with weight Gene does not control destiny Increased physical activity, moderate intake can promote healthy weight

43 Set Point Theory Weight is closely regulated by the body
Genetically predetermined body weight Body resists weight change Leptin assists in weight regulation Weight returns after weight loss Reduction in energy intake results in lower metabolic rate Ability to shift the set point weight

44 Why Diets Don’t Work Obesity is a chronic disease
Treatment requires long-term lifestyle changes Dieters are misdirected More concerned about weight loss than healthy lifestyle Unrealistic weight expectations

45 Why Diets Don’t Work Body defends itself against weight loss
Thyroid hormone concentrations (BMR) drop during weight loss and make it more difficult to lose weight Activity of lipoprotein lipase increases making it more efficient at taking up fat for storage

46 Why Diets Don’t Work Weight cycling (yo-yo dieting)
Typically weight loss is not maintained Weight lost consists of fat and lean tissue Weight gained after weight loss is primarily adipose tissue Weight gained is usually more than weight lost Associated with upper body fat deposition

47 Why Diets Don’t Work Weight gain in adulthood
Weight gain is common from ages 25-44 BMR decreases with age Inactive lifestyle Changes in body composition Fluid is usually the first weight lost Loss in lean body tissue means lowering the BMR Very little fat is lost during weight loss

48 Lifestyle Vs. Weight Loss
Prevention of obesity is easier than curing Balance energy in(take) with energy out(put) Focus on improving food habits Focus on increase physical activities

49 What It Takes To Lose a Pound
Body fat contains 3500 kcal per pound Fat storage (body fat plus supporting lean tissues) contains 2700 kcal per pound Must have an energy deficit of kcal to lose a pound per week

50 Do the Math To lose one pound, you must create a deficit of kcal So to lose a pound in 1 week (7 days), try cutting back on your kcal intake and increase physical activity so that you create a deficit of kcal per day - 500 kcal x 7 days = kcal = 1 pound of weight loss day week in 1 week

51 Sound Weight Loss Program
Meets nutritional needs, except for kcal Slow & steady weight loss Adapted to individuals’ habits and tastes Contains enough kcal to minimize hunger and fatigue Contains common foods Fit into any social situation Chang eating problems/habits Improves overall health See a physician before starting

52 Cutting Back Control calorie intake by being aware of kcal and fat content of foods “Fat Free” does not mean “Calories Free” (or “All You Can Eat”) Read food labels Estimate kcal using the exchange system Keep a food diary

53 Regular Physical Activity
Fat use is enhanced with regular physical activity Increases energy expenditure Duration and regularity are important Make it a part of a daily routine

54 Behavior Modification
Modify problem (eating) behaviors Chain-breaking Stimulus control Cognitive restructuring Contingency management Self-monitoring

55 Chain-Breaking Breaking the link between two behaviors
These links can lead to excessive intake Snacking while watching T.V.

56 Stimulus Control Alternating the environment to minimize the stimuli for eating Puts you in charge of temptations

57 Cognitive Restructuring
Changing your frame of mind regarding eating Replace eating due to stress with “walking”

58 Contingency Management
Forming a plan of action in response to a situation Rehearse in advance appropriate responses to pressure of eating at parties

59 Self-Monitoring Tracking foods eaten and conditions affecting eating
Helps you understand your eating habits

60 Weight Maintenance Prevent relapse Have social support
Occasional lapse is fine, but take charge immediately Continue to practice newly learned behavior Requires “motivation, movement, and monitoring” Have social support Encouragement from friends/ family/ professionals

61 Dieting Can Be Hazardous To Your Health
Weight regained consists of a higher percentage of body fat than before Less healthy than before dieting Weight loss diet should not be considered unless you are committed and motivated

62 Diet Drugs Amphetamine (Phenteramine) Sibutramine (Meridia)
Prolongs the activity of epinephrine and norepinephrine in the brain Decreases appetite Not recommended for long term use Sibutramine (Meridia) Enhances norepinephrine and serotonin activity Decreases appetite(eat less) Not recommended for people with HTN

63 Diet Drugs Orlistat (Xenical) Inhibits fat digestion
Reduces absorption of fat in the small intestine Fat is deposited in the feces with its side effects Must control fat intake Malabsorption of fat-soluble vitamins Supplements needed

64 Homeopathic drug Ephedrine (ma huang) St. John’s Wort
Linked to illnesses and death Associated with nervous and cardiovascular disorder St. John’s Wort Antidepressant Both taking together Not recommended until careful testing is done

65 Over-The-Counter Diet Aids
Phenylpropanolamine Epinephrine-like drug Cause a slight decrease in food intake Fiber “Filler” leading to satiety Causes stomach distention Benzocaine Numbs the tongue and taste buds

66 Very Low-Calorie Diets (VLCD)
Recommended for people >30% above their healthy weight kcal per day Low carbohydrates and high protein Causes ketosis Lose ~3-4 pounds a week Requires careful physician monitoring Health risks includes cardiac problems and gallstones

67 Gastroplasty - Stomach Stapling
Common surgical procedure for treating severe obesity Reduces the stomach size (from 4 cups) to half a shot glass size (1 oz) Overeating will result in rapid vomiting Smaller stomach promotes satiety earlier 75% will lose ~50% of excess body weight Costly Dumping syndrome

68 Gastroplasty (Fig ) Copyright © The McGraw-Hill Companies, Inc. Permission required for reproduction or display.

69 Underweight is Also a Problem
15-25% below healthy weight or BMI of <18.5 Associated with increased deaths, menstrual dysfunction, pregnancy complications, slow recovery from illness/surgery Causes are the same as for obesity but in the opposite route

70 Treatment for Underweight
Intake of energy-dense foods (energy input) Encourage meals and snacks Reduce activity (energy output) To gain a pound you need a total excess intake of kcal


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