 Support Events  Foot (Heel) Strike  Foot Flat  Midstance  Heel Off  Foot (Toe) Off  Swing Events  Pre swing  Midswing  Terminal swing.

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Presentation transcript:

 Support Events  Foot (Heel) Strike  Foot Flat  Midstance  Heel Off  Foot (Toe) Off  Swing Events  Pre swing  Midswing  Terminal swing

Initial Contact Beginning of Loading Foot Position may vary, but is generally supinated Represents end of single support on the opposite side

Maximum Impact Loading occurs Controlled by the Tibialis Anterior Foot rapidly moves into pronation Weight has been shifted to the support leg Coincides with end of the Initial period of Double Support on the Opposite side

Single Support Balance Critical All weight supported by single leg Foot remains pronated initially then re-supinates Late mid-stance is the period of max propulsion Swing occurring on opposite

Un-loading of limb and preparation for swing Foot Strike on Opposite Side Weight Shift to opposite side begins

Weight transition to opposite side completed Hip flexion has been initiated to facilitate swing Coincides with beginning of single support on the opposite side

Leg shortened (ankle Dorsiflexion) and hip elevated (abducted) to facilitate swing Mid-stance on the opposite side C. Of G. directly over opposite supporting foot

Hip flexion stopped and knee extended Foot supinated and positioned for foot strike The Sequence Begins Again

 Determinants on Gait: (Saunders, Inman, Whittle, etc.)  Knee Flexion During Stance  Pelvic Rotation (transverse plane)  Pelvic Lateral Tilt (Obliquity)  Ankle Mechanism (Dorsiflexion)  Ankle Mechanism (Plantarflexion)  Step Width

Lengthens the leg during stance

Lengthens Leg During Swing, prior to foot contact

Narrowing the base during double stance reduces lateral motion

 Center of Gravity (CG):  midway between the hips  Few cm in front of S2  Least energy consumption if CG travels in straight line

CG

B. Lateral displacement:  Rhythmic side-to-side movement  Lateral limit: mid stance  Average displacement: 5cm  Path: extremely smooth sinusoidal curve

 (1) Pelvic rotation:  Forward rotation of the pelvis in the horizontal plane approx. 8 o on the swing-phase side  Reduces the angle of hip flexion & extension  Enables a slightly longer step-length w/o further lowering of CG

 (2) Pelvic tilt:  5 o dip of the swinging side (i.e. hip adduction)  In standing, this dip is a positive Trendelenberg sign  Reduces the height of the apex of the curve of CG

 (3) Knee flexion in stance phase:  Approx. 20 o dip  Shortens the leg in the middle of stance phase  Reduces the height of the apex of the curve of CG

 (4) Ankle mechanism:  Lengthens the leg at heel contact  Smoothens the curve of CG  Reduces the lowering of CG

 (5) Foot mechanism:  Lengthens the leg at toe-off as ankle moves from dorsiflexion to plantarflexion  Smoothens the curve of CG  Reduces the lowering of CG

 (6) Lateral displacement of body:  The normally narrow width of the walking base minimizes the lateral displacement of CG  Reduced muscular energy consumption due to reduced lateral acceleration & deceleration

 Forces which have the most significant Influence are due to: (1) gravity (2) muscular contraction (3) inertia (4) floor reaction

 The force that the foot exerts on the floor due to gravity & inertia is opposed by the ground reaction force  Ground reaction force (RF) may be resolved into horizontal (HF) & vertical (VF) components.  Understanding joint position & RF leads to understanding of muscle activity during gait

 Muscle activation patterns are also cyclic during gait  In normal individuals, agonist- antagonist co activation is of relatively short duration  The presence of prolonged or out-or-phase agonist antagonist co activation during gait in individuals with pathology may indicate skeletal instability as well as motor control deficiencies