Cardiovascular System Assessments

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Presentation transcript:

Cardiovascular System Assessments Chapter 6  Cardiovascular System Assessments

Cardiovascular System Assessments Overview of essential components of this chapter are the: Normal electrocardiogram (ECG) patterns Common heart arrhythmias Noninvasive hemodynamic monitoring assessments Invasive hemodynamic monitoring assessments Determinants of cardiac output

The Electrocardiogram (ECG)

Figure 6-1. Electrocardiographic pattern of a normal cardiac cycle. Insert Figure 6-1 Figure 6-1. Electrocardiographic pattern of a normal cardiac cycle.

Normal Heart Rates Adult: 60 to 100 bpm Infant: 130 to 150 bpm

Methods Used to Calculate Heart Rates When the rhythm is regular: Count the number of large boxes on the ECG strip between two QRS complexes and then divide by 300. For example, if an ECG strip consistently shows four large boxes between each QRS complex, the heart rate is 75 bpm. 300 ÷ 4 = 75

Methods Used to Calculate Heart Rates (Cont’d) When the rhythm is irregular: The heart rate can be determined by counting the QRS complexes on a 6-second strip and multiplying by 10.

Common Heart Arrhythmias

Sinus Bradycardia In sinus bradycardia the heart rate is less than 60 bpm. Bradycardia means “slow heart.” Sinus bradycardia has a normal P-QRS-T pattern, and the rhythm is regular.

Figure 6-2. Sinus bradycardia. Rate is about 37 beats per minute.

Sinus Tachycardia In sinus tachycardia the heart rate is greater than 100 bpm. Tachycardia means “fast heart.” Sinus tachycardia has a normal P-QRS-T pattern, and the rhythm is regular.

Figure 6-3. Sinus tachycardia. Rate is about 100 beats per minute.

Sinus Arrhythmia In sinus arrhythmia the heart rate varies by more than 10% from beat to beat. The P-QRS-T pattern is normal, but the interval between groups of complexes (e.g., R-R intervals) varies.

Figure 6-4. Sinus arrhythmia. Note the varying R-R interval.

Atrial Flutter In atrial flutter the normal P wave is absent and replaced by two or more regular saw-tooth waves. The QRS complex is normal, and the ventricular rate may be regular or irregular. The atrial rate is usually constant, 250 to 350 bpm, whereas the ventricular rate is in the normal range.

Figure 6-5. Atrial flutter Figure 6-5. Atrial flutter. Atrial rate is greater than 300 bpm; ventricular rate is about 60 bpm.

Atrial Fibrillation In atrial fibrillation the atrial contractions are disorganized and ineffective, and the normal P wave is absent. The atrial rate ranges from 350 to 700 bpm. The QRS complex is normal, and the ventricular rate ranges from 100 to 200 bpm.

Figure 6-6. Atrial fibrillation.

Premature Ventricular Contractions The premature ventricular contraction (PVC) is not preceded by a P wave. The QRS complex is wide, bizarre, and unlike the normal QRS complex. The regular heart rate is altered by a PVC. The heart rhythm may be very irregular when there are many PVCs.

Figure 6-7. Premature ventricular contraction.

Ventricular Tachycardia In ventricular tachycardia, the P wave is generally indiscernible and the QRS complex is wide and bizarre in appearance. The T wave may not be separated from the QRS complex. The ventricular rate ranges from 150 to 250 bpm. The rate may be regular or slightly irregular. Blood pressure is often decreased during ventricular tachycardia.

Figure 6-8. Ventricular tachycardia.

Ventricular Flutter In ventricular flutter the QRS complex has the appearance of a wide sine wave. Regular, smooth, rounded wave The rhythm is regular or slightly irregular. The rate is 250 to 350 bpm. There is usually not discernible peripheral pulse associated with ventricular flutter.

Figure 6-9. Ventricular flutter.

Ventricular Fibrillation Ventricular fibrillation is characterized by chaotic electrical activity and cardiac activity. The ventricles literally quiver out of control, with no perfusion beat-producing rhythm. The is no cardiac output or blood pressure. The patient will die in minutes without treatment.

Figure 6-10. Ventricular fibrillation and asystole.

Asystole (Cardiac Standstill) Asystole is the complete absence of electrical and mechanical activity. Cardiac output stops, and the blood pressure falls to zero. The ECG tracing appears as a flat line.

Figure 6-10. Ventricular fibrillation and asystole.

Noninvasive Hemodynamic Monitoring Assessments Hemodynamics are defined as forces that influence the circulation of blood. The general hemodynamic status of the patient can be monitored noninvasively at the bedside by assessing the: Heart rate Blood pressure Perfusion state

Invasive Hemodynamic Monitoring Assessments Invasive hemodynamic monitoring is used in the assessment and treatment of critically ill patients. Invasive hemodynamic monitoring includes: Intracardiac pressures and flows via a pulmonary artery catheter Arterial pressure via arterial catheter Central venous pressure via a venous catheter

Pulmonary Artery Catheter The pulmonary artery catheter (Swan-Ganz) is a balloon-tipped, flow-directed catheter that is inserted at the patient bedside. Directly measures the: Right atrial pressure Pulmonary artery pressure Left atrial pressure Cardiac output

Figure 6-11. Insertion of the pulmonary catheter.

Arterial Catheter The most commonly used mode of invasive hemodynamic monitoring Measures: Continuous systolic, diastolic, and mean arterial blood pressure Fluctuations in blood pressure Data for guidance of therapy decisions for hypotension or hypertension

Central Venous Pressure Catheter The central venous pressure catheter measures the central venous pressure (CVP) and right ventricular filling pressure.

Table 6-1. Hemodynamic Values Measured Directly

Table 6-2. Hemodynamic Values Calculated from Direct Hemodynamic Measurements

Table 6-3. Hemodynamic Changes Commonly Seen in Respiratory Diseases