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Volume 117, Issue 1, Pages 173-180 (July 1999) Nitric oxide increases hepatic arterial blood flow in rats with carbon tetrachloride– induced acute hepatic injury Noriko Tanaka*, Katsuaki Tanaka*, Yoji Nagashima‡, Masaaki Kondo*, Hisahiko Sekihara* Gastroenterology Volume 117, Issue 1, Pages 173-180 (July 1999) DOI: 10.1016/S0016-5085(99)70565-2 Copyright © 1999 American Gastroenterological Association Terms and Conditions

Fig. 1 Photomicrographs of the liver 24 hours after AG injection in CCl4-treated and control rats. (A) Normal control rats and (B) AG-treated control rats had no significant changes in the liver. (C) Centrilobular hepatocytes were damaged in CCl4-treated rats, and (D) AG treatment after CCl4 administration showed more severe hepatocyte degeneration with inflammatory infiltrates (H&E; original magnification 179×). Gastroenterology 1999 117, 173-180DOI: (10.1016/S0016-5085(99)70565-2) Copyright © 1999 American Gastroenterological Association Terms and Conditions

Fig. 1 Photomicrographs of the liver 24 hours after AG injection in CCl4-treated and control rats. (A) Normal control rats and (B) AG-treated control rats had no significant changes in the liver. (C) Centrilobular hepatocytes were damaged in CCl4-treated rats, and (D) AG treatment after CCl4 administration showed more severe hepatocyte degeneration with inflammatory infiltrates (H&E; original magnification 179×). Gastroenterology 1999 117, 173-180DOI: (10.1016/S0016-5085(99)70565-2) Copyright © 1999 American Gastroenterological Association Terms and Conditions

Fig. 1 Photomicrographs of the liver 24 hours after AG injection in CCl4-treated and control rats. (A) Normal control rats and (B) AG-treated control rats had no significant changes in the liver. (C) Centrilobular hepatocytes were damaged in CCl4-treated rats, and (D) AG treatment after CCl4 administration showed more severe hepatocyte degeneration with inflammatory infiltrates (H&E; original magnification 179×). Gastroenterology 1999 117, 173-180DOI: (10.1016/S0016-5085(99)70565-2) Copyright © 1999 American Gastroenterological Association Terms and Conditions

Fig. 1 Photomicrographs of the liver 24 hours after AG injection in CCl4-treated and control rats. (A) Normal control rats and (B) AG-treated control rats had no significant changes in the liver. (C) Centrilobular hepatocytes were damaged in CCl4-treated rats, and (D) AG treatment after CCl4 administration showed more severe hepatocyte degeneration with inflammatory infiltrates (H&E; original magnification 179×). Gastroenterology 1999 117, 173-180DOI: (10.1016/S0016-5085(99)70565-2) Copyright © 1999 American Gastroenterological Association Terms and Conditions

Fig. 2 Reverse-transcription PCR analysis of nNOS, iNOS, and eNOS in liver tissue from control and CCl4-treated rats 1 day after CCl4 administration. Ethidium bromide–stained agarose gel of the amplification products showed uniform levels of glycerol-3-phosphate dehydrogenase expression, used as a control (data not shown). Band sizes (arrows) were as follows: nNOS, 353 base pairs; iNOS, 675 base pairs; eNOS, 806 base pairs. Gastroenterology 1999 117, 173-180DOI: (10.1016/S0016-5085(99)70565-2) Copyright © 1999 American Gastroenterological Association Terms and Conditions