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BASIC HUMAN NEEDS ALTERATIONS IN SKIN INTEGRITY PRESSURE ULCERS

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Presentation on theme: "BASIC HUMAN NEEDS ALTERATIONS IN SKIN INTEGRITY PRESSURE ULCERS"— Presentation transcript:

1 BASIC HUMAN NEEDS ALTERATIONS IN SKIN INTEGRITY PRESSURE ULCERS

2 Skin Integrity Skin/Integumentary system is the body’s largest organ, 1/6th of TBW Protects against disease causing organisms Sensory organ for temp, pain, touch Synthesizes Vitamin D Injury to skin poses a risk to safety and triggers a complex healing process

3 Normal Integument 2 principle layers in relation to wound healing
Epidermis Dermis Separated by basement membrane

4 Epidermis Outer layer has several layers within it Stratum Corneum
Stratum Lucidem Stratum Granulosum Stratum Spinosum Basal cell layer

5 Dermis Inner layer of skin
Provides tensile strength & mechanical support & protection to underlying muscle, bones, and organs Contains mostly connective tissue Also includes blood vessels, nerves, sensory nerve cells, lymphatics, collagen

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8 Skin Functions Epidermis-functions to re-surface wounds & restore the barrier against bacteria Dermis-functions to restore structural integrity-collagen& physical properties of skin

9 Alarming Facts Pressure ulcers were the primary diagnosis in about 45,500 hospital admissions (2006) Among hospital admissions listing pressure ulcers as a primary diagnosis, 1 in 25 admissions ended in death. Pressure ulcer related hospitalizations are longer and more expensive than other hospitalizations. Avg 5 day hospitalization $10,000, average pressure ulcer related stay extends to 14 days and costs up to $20,000. Source: AHRQ (www.ahrq.gov)

10 Pressure Ulcers New NPUAP terminology (2007) www.npuap.org
A pressure ulcer is a localized injury to the skin and/or underlying tissue usually over a bony prominence, as a result of pressure, or pressure in combination with shear and/or friction. A number of contributing factors are also associated with pressure ulcers

11 Pressure Ulcers Tissues receive oxygen and nutrients and eliminates metabolic wastes via the blood Any factor that interferes with this affects cellular metabolism and cell life Pressure affects cellular metabolism by decreasing or stopping tissue circulation resulting in tissue ischemia

12 Causes of Pressure Ulcers
Pressure > ischemia > edema > inflammation > small vessel thrombosis > cell death Shear – trauma caused by tissue layers sliding across each other, results in disruption or angulation of blood vessels

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16 Pressure Ulcer Contributing Factors
Friction/Shear Poor Nutrition Incontinence Moisture Co-existing Medical Conditions

17 Pressure Tissue damage occurs when pressure exerted on the capillaries is high enough to close the capillaries Capillary closing pressure is the pressure needed to close the capillary > 32 mmHg After a period of ischemia light toned skin undergoes 2 hyperemic changes

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20 Hyperemia Normal Reactive Hyperemia-visible effect of localized vasodilatation (REDNESS) area will blanch with fingertip pressure and redness lasts less than 1 hour Abnormal Reactive Hyperemia-excessive vasodilatation and induration (edema) in response to pressure. Skin appears bright pink-red. Lasts 1 hour to 2 weeks

21 Risk Factors for Pressure Ulcer Development
Impaired Sensory Input Impaired Motor Function Altered Level of Consciousness Orthopedic Devices

22 Pathogenesis of Pressure Ulcers
Intensity of pressure and capillary closing pressure Duration and sustenance of pressure Tissue Tolerance

23 Pathogenesis of Pressure Ulcers
Bony prominences are most at risk (sacrum, heels, elbows, lateral malleoli, greater trochanter, ischial tuberosities Pressure ulcer forms as a result of time/pressure relationship Greater the pressure and duration of pressure, the greater the incidence of ulcer formation

24 Pathogenesis of Pressure Ulcers
Skin and subcutaneous tissue can withstand some pressure Tissue will over time become hypoxic and ischemic injury will occur If the pressure is above 32mmHg and remains unrelieved to the point of tissue hypoxia, the vessel will collapse and thrombose

25 Pathogenesis of Pressure Ulcers
If circulation is restored before this critical point, circulation to tissue is restored (Reactive Hyperemia) Skin has a greater ability to tolerate ischemia than does muscle, hence true pressure ulcers begin at bone with pressure related to muscle ischemia eventually coming through to epidermis (Shear injury) Sacrum and heels most susceptible

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28 Pressure Ulcer Staging
Depth of destroyed tissue Does not indicate healing Ulcer covered by necrotic tissue or eschar cannot be staged until debrided NPUAP system used most clinically Other staging systems exist

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32 Stage 1 Pressure Ulcer Intact skin with non-blanchable redness of a localized area usually over a bony prominence. Darkly pigmented skin may not have blanching: its color may differ from the surrounding area The area may be painful, firm, soft, warmer or cooler as compared to adjacent tissue. Stage I may be difficult to detect in individuals with darker skin tones

33 Stage I Treatment Off-load pressure Transparent film dressing
Hydrocolloid dressing Moisture barrier

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36 Stage II

37 Stage 2 Pressure Ulcer Partial thickness skin loss involving the epidermis and/or dermis. The ulcer is superficial and presents clinically as an abrasion, blister, or shallow open ulcer Presents as shiny or shallow ulcer (red/pink wound bed) without slough or bruising. This stage should not be used to describe skin tears, tape burns, perineal dermatitis, maceration or excoriation

38 Stage II Treatment Hydrocolloid dressing: dressing of choice in minimally draining stage II ulcer Absorptive dressings (Foam) draining wounds Hydrogel: Healing wounds Off-load pressure

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42 Stage III

43 Stage III Full thickness skin loss involving damage or necrosis to subcutaneous tissue that may extend down to, but not through underlying fascia Ulcer presents as a deep crater with or without undermining or tunneling of adjacent tissue Slough tissue may be present but does not obscure the depth of tissue loss Depth varies by anatomical location

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45 Stage III Treatment Requires physician order for Stage III or IV
Draining vs. Non-draining Necrotic vs. Granulating Draining wounds-Absorptive dressings Granulating wounds-Hydrogel Necrotic wounds-Require debridement (Chemical. Mechanical, Autolytic, Sharp)

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48 Stage IV

49 Stage IV Full thickness skin loss with extensive destruction, tissue necrosis or damage to muscle, bone , or supporting structures (tendons, joint) Undermining and tunneling are often associated with Stage IV ulcers Slough or eschar may be present in some on some parts of the wound bed Depth of wound varies by anatomical location Exposed bone or tendon is visible or directly palpable

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54 Unstagable Wounds Full thickness tissue loss in which the base of the ulcer is covered by slough (yellow, tan, gray, green or brown) and/or eschar (tan, brown, black) in the wound bed The true depth of the wound cannot be determined until slough or eschar is removed, therefore stage cannot be determined. Stable eschars serve as the body’s natural biological cover and should not be removed

55 Unstageable

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58 Deep Tissue Injury Purple or maroon localized area of discolored intact or blood filled blister due to the damage of underlying soft tissue from pressure or shear. The area may be preceded by tissue that is painful, firm, mushy, boggy, warmer or cooler as compared to adjacent tissue May be difficult to detect with darker skin tones Evolution may include a thin blister over a dark wound bed

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62 Staging by Color Black Yellow Pink/Red Mixture of colors

63 Process of Wound Healing
Primary Intention Secondary Intention

64 Healing by Primary Intention
Inflammatory (Reaction) Proliferative (Regeneration) Maturation (Re-modeling)

65 Healing by Secondary Intention
Healing takes longer Wounds drain more fluids Inflammation phase is prolonged, chronic Wound becomes filled with fragile granulation tissue rather than collagen Wound Contraction takes place More susceptible to infection

66 Complications of Wound Healing
Hemorrhage Infection (Nosocomial) Dehiscence Evisceration Fistula Formation

67 Risk Assessment for Pressure Ulcers
Identify at risk population Norton Scale Gosnell Scale Braden Scale- most uses clinically, includes 6 subscales (sensory perception, moisture, activity, mobility, nutrition, friction & shear 6-23 score, <18 at risk in hospitalized patients Refer to P&P pg for Braden Scale

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69 Factors Affecting Pressure Ulcer Formation
Shearing force Friction Moisture Tissue Tolerance Factors Nutrition Infection Impaired Peripheral Circulation Age

70 Factors that Impair Wound Healing
Age Malnutrition Obesity Impaired oxygenation Smoking Diabetes (blood glucose level) Drugs Radiation Wound Stress

71 Nursing Process Assessment
Predictive measures-Risk assessment tools Skin assessment- any areas susceptible to pressure sources, (NG, oxygen tubes, casts, bony prominences) Tactile Assessment-Blanching red areas Assess mobility Assess nutritional status

72 Wound Assessment Location, Size, Stage Wound drainage
Wound bed, tissue type Wound edges Periwound skin Presence of undermining, tunneling

73 Wound Assessment Anatomical location Stage-NPUAP staging
Staging is for pressure ulcers only, other wounds are classified as partial or full thickness Size- Measure length, width, depth in centimeters

74 Wound Assessment Drainage Amount, color, consistency, odor
Scant, moderate, large Serous, serosanguinous, purulent, yellow, brown, green, clear Odor to wound may be indicative of infection

75 Wound Assessment Tissue Type
When describing wound bed include % of each tissue type (50% slough, 50% granulation) Necrotic tissue-nonviable Eschar- dry, leathery, black or brown Slough- stringy, cheesy, loose, yellow, tan Granulation- healthy, viable pink to beefy red Epithelialization-occurs along wound edges or as islands inside wound bed, pale pink resurfacing of wound

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80 Wound Assessment Periwound Area
Erythema-may mean infection Maceration-Whitish, wrinkled appearance Indicates presence of excessive moisture Rash- Macular or papular, may indicate fungal infection

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82 Wound Assessment Presence of Undermining/Tunneling
Document location and depth Use hands of clock as descriptor Measure with cotton tipped applicator

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85 Staging Limitations Difficult to identify stage I in dark skinned patients Unable to stage when obscured by eschar Reverse Staging/Downstaging

86 Nursing Process Diagnosis
You tell me!!!!!

87 Nursing Process Planning
Preventing pressure ulcers-early identification of those at risk (Braden, Norton, Gosnell scales) Prevention protocols by hospital Positioning Hygiene and skin care (incontinence care) Support surfaces Nutritional support Prevent friction and shear Education

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92 Nursing Process Acute Care Implementation
Management of Pressure Ulcers Culturing wound Cleansing wound Debridment of wound Moist Wound Healing Dressing selection Nutritional support Off-load pressure

93 Wound Dressing Selection
Goal: Promote moist wound healing Transparent dressing Hydrocolloid Hydrogel Calcium alginate Foam dressing Silver/Antimicrobial Collagen dressing Biological dressing (Regranex) Negative pressure wound therapy (VAC)

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110 Practice Scenario The nurse is assessing a bedridden client when a large erythemic area is noted on the client’s sacrum. In addition, the center of the injury looks like an abrasion with a shallow center. The nurse would classify this ulcer as: How will the nurse treat this type of pressure ulcer? What risk factors could have contributed to this patient developing a pressure ulcer?

111 Practice Question A nurse is working in a geriatric screening clinic. The nurse would expect that the skin of the normal elderly client will demonstrate which of the following characteristics? A. Dehydration causing skin to swell. B. Moist skin turgor. C. Skin turgor showing a loss of elasticity D. Overhydration causing skin to wrinkle.

112 Practice Question The nurse decides to treat a Stage II pressure ulcer with a hydrocolloid dressing. The nurse recognizes that the dressing will promote which type of wound debridement? A. Sharp B. Autolytic C. Chemical D. Mechanical


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