Download presentation
Presentation is loading. Please wait.
1
CCHD with Low PBF & No PAH
TOF TOF Equivalents DORV+ VSD+ PS D-TGA + VSD+ PS L-TGA + VSD+ PS Tricuspid Atresia + VSD + PS Single Ventricle + PS Truncus Arteriosus with small pulmonary arteries Pulmonary Atresia with IVS PS with ASD Ebsteins Anomaly Of TV
2
CCHD with Low PBF & PAH Eisenmenger’s Syndrome
3
CCHD with High PBF Inter circulatory mixing Venous level: TAPVC Atrial level : Single Atrium, Tricuspid Atresia, HLHS Ventricular level : Single ventricle Arterial level: Truncus Arteriosus Transposition Physiology D TGA Taussing bing anomaly
4
CCHD with Near Normal PBF
Pulmonary Arterio Venous Fistula Anomalous drainage of vena cava to Left Atrium Un roofing of coronary sinus in to Left Atrium
5
Factors affecting age of Presentation in CCHD
6
Factors affecting age of Presentation in CCHD
0-3 days : Changes in Transition Circulation 4-14 days : Closing ductus physiology 2-18 weeks: Low pulmonary vascular resistance Any age: Myocardial dysfunction, Metabolic and idiopathic
7
Factors affecting age of Presentation in CCHD : 0-3 days
Changes in Transition Circulation Parallel non mixing circulation TGA Critically obstructed series circulation- Obstructed TAPVC HLHS & HRHS Critical AS Interrupted aortic arch HRHS Critical PS Severe Ebsteins
8
Factors affecting age of Presentation in CCHD : 4- 14 days
Closing Ductus Physiology Obstructed Pulmonary circulation TOF (Severe) Tricuspid atresia TGA + VSD + PS Single ventricle + PS Obstructed Systemic circulation Severe CoA
9
Factors affecting age of Presentation in CCHD :2 weeks to 18 weeks
Low Pulmonary vascular resistance Shunt lesions with Left to Right shunts Complete AV Canal defect TOF with mild PS Large VSD TAPVC (Non obstructed) TGA + VSD without PA Single ventricle without PS ALCAPA
10
4- 12 months VSD (small to moderate) PDA (small to moderate) ASD
Mild CoA AS Mild to Mod PS Mild to Mod
11
Cardiac causes of CHF according to age
12
Cardiac causes of CHF according to age
IST Day of life HLHS Obstructed TAPVC Endocardial fibroelastosis Large AV fistula Arrythmia : PAT Congenital Severe TR or PR Premature infants with large PDA
13
Cardiac causes of CHF according to age
First week of life HLHS Complete TGA CoA Severe AS
14
Cardiac causes of CHF according to age
During infancy CoA with PDA or VSD Large VSD or PDA or AV Canal defect TAPVC Tricuspid atresia Persistent Truncus DORV TGA with Large VSD ALCAPA Myocarditis
15
CCHD Clinical clues to Diagnosis
16
CCHD with Bounding Pulse
Truncus Arteriosus Pulmonary Atresia with Large PDA or Large MAPCAS TOF with severe AR Eissenmenger PDA with Pulmonary Regurgitation
17
CCHD with Prominent A wave in JVP
TRICUSPID ATRESIA with Restrictive ASD CCHD with Cannon waves (Complete Heart Block) CCTGA Single Ventricle especially Inverted type
18
CCHD with Prominent V wave in JVP
Ebsteins anomaly Eisenmengers with severe TR Pulmonary atresia with IVS and severe TR
19
CCHD with Prominent LV Impulse
TRICUSPID ATRESIA SINGLE VENTRICLE OF LV TYPE TOF with significant AR PULMONARY ATRESIA with IVS TRUNCUS with Truncal Regurgitation to LV
20
CCHD with Wide Split S2 TAPVC EBSTEINS ANOMALY
21
CCHD with Prominent Pulsation in Second Left ICS
Eisenmengers Syndrome CCTGA with L Posed Aorta
22
CCHD with EDM TOF with AR TOF with Absent Pulmonary Valve
Eisenmenger with PR TRUNCUS ARTERIOSUS with Truncal Regurgitation
23
CCHD with Continous Murmer
Pulmonary Atresia with PDA/MAPCAS TOF : Post BT Shunt Supracardiac TAPVC with Venous Hum at Root of Neck
24
CCHD with Tricuspid Regurgitation
Ebsteins anomaly PULMONARY ATRESIA with IVS and SEVERE TR Eisenmenger with TR TOF with AV CANAL Defect, Severe TR
25
CCHD with Dominant Ventricle
26
CCHD with Right ventricle dominance
TOF DORV+VSD+PS D TGA+VSD+PS L TGA+VSD+PS PS+ASD HLHS
27
CCHD with Left Ventricle dominance
Tricuspid Atresia Double inlet Left Ventricle Pulmonary Atresia with IVS Ebsteins Anomaly with Hypoplastic Rt Ventricle Non Restrictive ASD
28
Biventricular Dominance
DORV+VSD D TGA+VSD Tricuspid Atresia with VSD Truncus Arteriosus
Similar presentations
© 2024 SlidePlayer.com Inc.
All rights reserved.