1. Volume 117, Issue 1, Pages 173-180 (July 1999)
Nitric oxide increases hepatic arterial blood flow in rats with carbon tetrachloride– induced acute hepatic injury 
Noriko Tanaka*, Katsuaki Tanaka*, Yoji Nagashima‡, Masaaki Kondo*, Hisahiko Sekihara* 
Gastroenterology 
Volume 117, Issue 1, Pages (July 1999)
DOI: /S (99)
Copyright © 1999 American Gastroenterological Association Terms and Conditions

2. Fig. 1
Photomicrographs of the liver 24 hours after AG injection in CCl4-treated and control rats. (A) Normal control rats and (B) AG-treated control rats had no significant changes in the liver. (C) Centrilobular hepatocytes were damaged in CCl4-treated rats, and (D) AG treatment after CCl4 administration showed more severe hepatocyte degeneration with inflammatory infiltrates (H&E; original magnification 179×).
Gastroenterology  , DOI: ( /S (99) )
Copyright © 1999 American Gastroenterological Association Terms and Conditions

3. Fig. 1
Photomicrographs of the liver 24 hours after AG injection in CCl4-treated and control rats. (A) Normal control rats and (B) AG-treated control rats had no significant changes in the liver. (C) Centrilobular hepatocytes were damaged in CCl4-treated rats, and (D) AG treatment after CCl4 administration showed more severe hepatocyte degeneration with inflammatory infiltrates (H&E; original magnification 179×).
Gastroenterology  , DOI: ( /S (99) )
Copyright © 1999 American Gastroenterological Association Terms and Conditions

4. Fig. 1
Photomicrographs of the liver 24 hours after AG injection in CCl4-treated and control rats. (A) Normal control rats and (B) AG-treated control rats had no significant changes in the liver. (C) Centrilobular hepatocytes were damaged in CCl4-treated rats, and (D) AG treatment after CCl4 administration showed more severe hepatocyte degeneration with inflammatory infiltrates (H&E; original magnification 179×).
Gastroenterology  , DOI: ( /S (99) )
Copyright © 1999 American Gastroenterological Association Terms and Conditions

5. Fig. 1
Photomicrographs of the liver 24 hours after AG injection in CCl4-treated and control rats. (A) Normal control rats and (B) AG-treated control rats had no significant changes in the liver. (C) Centrilobular hepatocytes were damaged in CCl4-treated rats, and (D) AG treatment after CCl4 administration showed more severe hepatocyte degeneration with inflammatory infiltrates (H&E; original magnification 179×).
Gastroenterology  , DOI: ( /S (99) )
Copyright © 1999 American Gastroenterological Association Terms and Conditions

6. Fig. 2
Reverse-transcription PCR analysis of nNOS, iNOS, and eNOS in liver tissue from control and CCl4-treated rats 1 day after CCl4 administration. Ethidium bromide–stained agarose gel of the amplification products showed uniform levels of glycerol-3-phosphate dehydrogenase expression, used as a control (data not shown). Band sizes (arrows) were as follows: nNOS, 353 base pairs; iNOS, 675 base pairs; eNOS, 806 base pairs.
Gastroenterology  , DOI: ( /S (99) )
Copyright © 1999 American Gastroenterological Association Terms and Conditions