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Upper Gastrointestinal Tract KNH 411. Upper GI – A&P Stomach - Motility Filling, storage, mixing, emptying 50 mL empty – stretches to 1000 mL Pyloric.

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Presentation on theme: "Upper Gastrointestinal Tract KNH 411. Upper GI – A&P Stomach - Motility Filling, storage, mixing, emptying 50 mL empty – stretches to 1000 mL Pyloric."— Presentation transcript:

1 Upper Gastrointestinal Tract KNH 411

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3 Upper GI – A&P Stomach - Motility Filling, storage, mixing, emptying 50 mL empty – stretches to 1000 mL Pyloric sphincter © 2007 Thomson - Wadsworth

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6 Pathophysiology - Oral Cavity Nutrition Therapy/Evaluation Increase frequency of meals- tired quickly- 6 small feeding, high cal, high pro Bland foods served at room temp. Liberal use of fluids- be careful with water, make sure it is high energy density food Preference for cold and frozen foods- takes away some of the smell if ill (chemo) Oral hygiene- embarrassed maybe Monitor using food diary, observation, or kcal count- a lot of this done by computers but need to know how to do by hand Monitor weight gain or maintenance

7 Pathophysiology - Esophagus GERD - reflux of gastric contents into the esophagus Incompetence of LES Increased secretion of gastrin, estrogen, progesterone Hiatal hernia Cigarette smoking- can losen Use of medications Foods high in fat, chocolate, spearmint, peppermint, alcohol, caffeine (fried foods)

8 Pathophysiology - Esophagus GERD - symptoms Dysphagia- difficulty swallowing Heartburn- antiacids Increased salivation Belching Pain radiating to back, neck, or jaw Aspiration- refluxing of the contents of the stomach Ulceration Barrett’s esophagus- change in epithelial cells, abnormal pH- squamous cell carcinoma- cancer a concern

9 Pathophysiology - Esophagus GERD - Treatment Medical management- antiacids, histamine blocker, mucousal protectants Modify lifestyle factors Medications – 5 classes (in book) to strengthen LES Surgery- most severe Fundoplication- fundus, wrap it around the LES, tightens Stretta procedure- radiofrequency is energy is used, increases the function

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12 Pathophysiology - Esophagus GERD - Nutrition Therapy Identify foods that worsen symptoms- previously mentioned Assess food intake esp. those that reduce LES pressure, or increase gastric acidity Assess smoking and physical activity- smoking cessation Small, frequent meals- lessens the pressure Weight loss if warranted

13 Pathophysiology - Esophagus Dysphagia – difficulty swallowing Potential causes – GERD, Stroke Drooling, coughing, choking- could aspirate Weight loss, generalized malnutrition Aspiration to aspiration pneumonia- inhalation into the oral pharynx, constant oral problem Treatment requires health care team dg by bedside swallowing, videofluoroscopy, barium swallow

14 Pathophysiology - Esophagus Dysphagia – Nutrition Therapy Use acceptable textures to develop adequate menu National Dysphagia Diet 1,2,3 Use of thickening agents and specialized products Monitor weight, hydration, and nutritional parameters

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18 © 2007 Thomson - Wadsworth Hiatal Hernia

19 Pathophysiology - Stomach Gastritis Inflammation of the gastric mucosa Primary cause: H. pylori bacteria Alcohol, food poisoning, NSAIDs Symptoms: belching, anorexia, abdominal pain, vomiting Type A – automimmune- upper section of the section- antibodies of the peritoneal cells Type B – H. pylori- atropy Increases with age, achlorhydria- lack of HCl Treat with antibiotics and medications

20 Pathophysiology - Stomach Peptic ulcer disease - ulcerations of the gastric mucosa that penetrate submucosa Gastric or duodenal H. pylori NSAIDS, alcohol, smoking Certain foods, genetic link Increased risk of gastric cancer

21 Pathophysiology - Stomach Peptic Ulcer Disease - Nutrition Restrict only those foods known to increase acid secretion Black and red pepper, caffeine, coffee, alcohol, individually non- tolerated foods Consider timing and size of meal Do not lie down after meals Small, frequent meals

22 © 2007 Thomson - Wadsworth

23 Pathophysiology - Stomach Gastric Surgery - Nutrition Implications Reduced capacity Changes in gastric emptying & transit time Components of digestion altered or lost Decreased oral intake, maldigestion, malabsorption Alter their diet, chart about these

24 Pathophysiology - Stomach Gastric Surgery - Dumping Syndrome Increased osmolar load enters small intestine too quickly from stomach Release of hormones, enzymes, other secretions altered Food “dumps” into small intestine

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26 Pathophysiology - Stomach Gastric Surgery - Dumping Syndrome Early dumping – 10-20 min.; diarrhea, dizziness, weakness, tachycardia Intermediate - 20-30 min.; fermentation of bacteria produces gas, abdominal pain, etc. Late dumping - 1-3 hrs.; hypoglycemia

27 Pathophysiology - Stomach Gastric Surgery - Dumping Syndrome Other nutritional concerns: vitamin and mineral deficiencies, lack of intrinsic factor, iron deficiency, osteoporosis

28 Pathophysiology - Stomach Dumping Syndrome - Nutrition “Anti-dumping” diet Slightly higher in protein & fat Avoid simple sugars & lactose Calcium & vitamin D Liquid between meals Small, frequent meals Lie down after meals Assess for weight loss, malabsorption, and steatorrhea

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