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LYSTRA WILSON-CELESTINE, FACOG May 21th 2015

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Presentation on theme: "LYSTRA WILSON-CELESTINE, FACOG May 21th 2015"— Presentation transcript:

1 LYSTRA WILSON-CELESTINE, FACOG May 21th 2015
EMS OBGYN OVERVIEW LYSTRA WILSON-CELESTINE, FACOG May 21th 2015

2 OBJECTIVES Review female anatomy and reproductive system
Normal pregnancy, labor and delivery Assessing a pregnant patient Common complications and emergencies of pregnancy Newborn care Review of case scenarios

3 Definition of Terms Gravity: # of pregnancies
Parity: # of pregnancies >20wk Nulliparous: never pregnant Primagravid: first pregnancy Gravity- this includes ectopic and miscarriages, abortion In parity a twin pregnancy is counted as a single pregnancy

4 Definition of Terms Presentation: leading part in birth canal- crown, rump, face, arm. Term : 37 to 42wks. Preterm : <37wks Post term:>42wks Abortus: Fetus /embryo delivered <20wk/500gm

5 External Genitalia

6 Pelvic Anatomy

7 Reproductive Organs

8 Physiology of Pregnancy
Genital Tract Vagina, perineum: Increased vascularity, hyperemia, edema Increased secretions (thick white discharge) Acidic pH( 3.5-6) Increased vaginal wall length Chadwick’s sign- violet color of vagina/vulva For a successful pregnancy major adaptions are required in maternal anatomy, physiology and metabolism. It is necessary to understand the normal changes to recognize the abnormal.

9 Normal cervix

10 Chadwick’s sign and leucorrhea

11 Chadwick sign- pregnant

12 Physiology of Pregnancy Uterus
fold increase in size Wt. at term +/- 1100gm Out of pelvis by end of 12th wk. Dextrorotated Blood flow increases from 100 to 650ml/min Limited auto regulation Increase in size due to stretching and hypertrophy of muscle cells caused by estrogen and POC venous compression, supine hypotension, Uterine artery dilates due to hormone effect leading to decreased resistance to flow. Vessels are maximally dilated to improve blood flow but when maternal cardiac output declined blood shifts from uteroplacental bed to maternal brain, kidney and heart.

13 Physiology of Pregnancy Uterus
Limited Auto regulation Maximum uterine vessel dilation leave little auto regulation to improve flow during perfusion pressure changes Decreased maternal cardiac output blood flow shift away from placenta to maternal brain, kidney and heart. Uterine Hypertrophy Venous compression fall in venous return, fall in cardiac output Compensation: Supine hypotension syndrome, nausea, dizziness, syncope, relief by position change Increased size due to stretching and hypertrophy of muscles caused by estrogen and POC

14

15 Physiology of Pregnancy Cervix
Thickened mucus Chadwick sign Eversion of columnar cervical glands

16 Physiology of Pregnancy Ovaries
Suspended follicular maturation Enlarged ovarian veins Single corpus luteum Functional 4-5wks post ovulation Produces progesterone, relaxin

17 Physiology of Pregnancy Skin
Vascular Spider angiomas Palmar erythema- also seen thyroid disease, lung CA or inherited Striae gravidarum Genetic disposition

18 Palmar erythema

19

20 Spider Angioma

21 Striae gravidarum

22 Physiology of Pregnancy Skin
Increased pigmentations due to estrogen, progesterone, melanocytes simulating hormones Linea negra Chloasma/Melasma gravidarum

23 Linear Negra

24 Molasma Gravidarum

25 Physiology of Pregnancy Breast
Tender/tingling sensation in early preg Nipple enlarges, broader areolae with increased pigmentation Increase size from ductal growth and alveolar hyperplasia Colostrum production

26 Physiology of Pregnancy Musculoskeletal
Lumbra lordosis low back pain Relaxation of pubic symphysis and sacroiliac joints Relaxed muscles leading to hernia and easily strained muscles All compounded by weight gain.

27 Lordosis of pregnancy

28

29 Physiology of Pregnancy Hematologic
50% increases in blood volume Plasma volume increases 50-70%; starts at 6wks RBC mass increase 20-35%: starts at 12wk Physiologic anemia Hemodilutional Anemia nadirs at 30-34wks

30 Physiology of Pregnancy Hematology
Iron Deficiency Anemia Increased iron requirements, supplements recommended term Hgb <10mg/dL due to deficiency rather than hemodilution Immune changes WBC increases to in 3rd TM Plt decrease slightly

31 Physiology of Pregnancy Hematology
Coagulation Fibrinogen increases 50% Changes in clotting factors and regulatory protein Cardiac output Begins to increase by 5th wk Peaks at 20-24wks Rises by 40% by 20-24wks Overall 50% increase

32 Physiology of Pregnancy Hematology
Initially increase in heart rate Reduced systemic vascular resistance CXR: displaced heart to left upward and pericardial effusion

33 Physiology of Pregnancy Test Interpretation
BP: SBP increases by 5-10mmHg; DBP by mmHg (before 24wks). Each contraction pushes ml from uterus to circulation Rise in arterial BP 10mmHg during Ctx.

34 Physiology of Pregnancy Respiratory
Estrogen hyperemic, edematous nasopharynx and increased mucous secretions. Symptoms: stuffiness, epistaxis, chronic cold.  chest circum. and transverse diameter; Diaphragm pushed up 4cm Changes in lung volumes and pulmonary function test. Oxygen consumption increases 15-20% BOTTOM LINE State of hyperventilation with chronic respiratory alkalosis

35 Physiology of Pregnancy Urinary
Mechanical Ureteric obstruction from uterus Incomplete bladder empting Vesicoureteral reflux Physiology 75%  renal blood flow with increase in GFR 50% Multiple trips to bathroom Glucosuria, Proteinuria

36 Physiology of Pregnancy Gastrointestinal
Increased appetite (300kcal/d) Ptyalism (1-2L/d) spitting Gingivitis Lower tone of Gastroesophageal sphincterreflux Delay gastric emptying (60% of meal emptied in 90mins for non-pregnant; doubled time for pregnant)

37 Physiology of Pregnancy Gastrointestinal
Increased small bowel transit time 58 vs 52hrs Stomach and intestinal displacement appendix at right flank Constipation/Hemorrhoids Gallbladder changes increased risk of stones

38 Normal Pregnancy Events
1st Trimester (LMP to 13wks) Nausea/Vomiting, fatigue, Food aversion or cravings, spotting, breast tenderness, increased sex drive Gain about 5-8lbs Complications- Miscarriage, Ectopic, blighted ovum

39 Normal Pregnancy Events
2nd Trimester (13-26wks) Feeling of well being, less fatigue. Round ligament pain, bladder pressure, round ligament pain, Braxton hicks Complications- fetal loss is minimal but can seen with labor, incompetent cervix, intrauterine death.

40 Normal Pregnancy Events
3rd Trimester (26wks to delivery) Feeling uncomfortable; pelvic/back pain and pressure Lower extremities swelling, varicosities, engagement, contractions,. Wt gain 1lbs/wk Complications: Rupture membranes, preterm labor, pregnancy induced hypertension, Urinary tract infection, Gestational diabetes

41 Complications of Pregnancy
Vaginal bleeding Spontaneous Miscarriage Ectopic Pregnancy Premature rupture of membranes with cord prolapse Pre eclampsia/Eclampsia Placental Previa

42 Complications of Pregnancy
Medical/surgical eg diabetes, ruptured appendix Abruptio Placenta Breech presentation and delivery Meconium Stained fluid Abnormal labor pattern Stressed Newborn

43 Labor Clinical diagnosis Onset of regular rhythmic contractions
Progressive cervical dilation and effacement 3 stages

44 Stages of Labor Stage 1 Interval between labor onset and full cervical dilation Latent phase- period btw labor onset to start of rapid change of cervical dilation Active phase- period from 6cm to 10cm

45 Stages of Labor Stage 2 Interval btw full dilation (10cm) to delivery of infant Nulliparous- push for max of 2hr without regional anesthesia(3hr with) Multiparous- push for max 1hr without anesthesia (2hr with)

46 Stages of Labor Stage 3 Refers to delivery of placenta and fetal membranes Make take up to 30mins What are the active interventions if >30mins?

47 Cardinal Movement of Labor
Engagement- passage to widest diameter of presenting part below plan of pelvic inlet Descent- downward passage of presenting part through pelvis Flexion- passive flexion of head on to chest

48 Cardinal Movements of Labor
Internal Rotation- vertex moves from transverse to anteroposterior position Extension – fetus head is at level of introitus; base of occiput is at inferior margin of pubic symphysis External Rotation- or restitution- return of head to correct anatomical position- LOA or ROA Explusion- delivery of rest of fetus

49 Demonstration of Delivery Method.
xCo

50 Field Obstetric Assessment
Determine if delivery is imminent Remain calm Ask few questions Closed ended Simple answers Perform visual exam (with permission) Evaluate vitals

51 Obstetrics Assessment
Things you want to know Due date Number of pregnancies delivered in past Length of labor in past Is there vaginal bleeding or did she break her water Is there a feelings to have a bowel movement

52 Obstetrics Assessment
If delivery is imminent- What are the signs? Crowning or bulging She screams “I need to take a dump “or “its coming” or “I have to push” What to do! Remain calm, place patient supine in safe location. Disrobe undergarment – have pt/husband/ SO do it. Visual check of perineum- blood loss, fetal parts, bag Abdominal palpation for contractions-duration, interval

53 Obstetrics Assessment Field Delivery
Anticipate exposure of large amount of blood and body fluids Full personal protection is recommended Don’t assume absence or presence of disease by appearance of patient or situation.

54 Sterile OB Kit Content Sterile exam gloves Disposable scalpel
Maternity pad Plastic lined under pad Receiving blanket Disposable towels Gauze sponges Disposable bulb syringe Disposable plastic apron Plastic bag to hold placenta Twist ties O.B. towelettes Umbilical cord clamp

55 Obstetrics Assessment Field Delivery
You are ready for a delivery!!!

56 Crowning/Extension

57 External Rotation

58 External Rotation

59 Delivery of Anterior Shoulder

60 Delivery of Posterior Shoulder

61 Double cord clamping and cutting

62 Case Scenario #1

63 Case Scenario #1 Post partum hemorrhage risk factors:
Grand multiparous, rapid labor, prolonged labor, augmented labor History of postpartum hemorrhage, episiotomy, especially mediolateral, preeclampsia, Overdistended uterus (macrosomia, twins, hydramnios), operative delivery, Asian or Hispanic ethnicity, chorioamnionitis

64 Case Scenario #2

65 Case Scenario #2 Cord Prolapse True emergency
Need to release pressure of head against cord Sterile vaginal exam check for cord pulsation and push up on vertex. Keep hand in vagina until OB team takes over. Emergency cesarean section with general anesthesia is fastest way to deliver.

66 Case Scenario #2

67 Case Scenario #3

68 Case Scenario #3 Abruptio Placenta
Premature separation of normal placenta from uterine wall secondary to decidual bleeding. 1/86 to 1/206 cases. Risks factors: Hypertensive disease, Advanced maternal age and parity Drug use (eg smoking, cocaine) Trauma Uterine anomalies eg fibroids Sudden decompression eg ROM

69 Placental Abruption

70 Case Scenario #3 Abruptio Placenta
Classic Signs: vaginal bleeding, abdominal pain, uterine contractions and tenderness Abruption can be concealed with no evidence of vaginal bleeding (10-20%) Size of hemorrhage predictive of fetal survival >60ml associated with >50% fetal mortality.

71 Case Scenario #4

72 Case Scenario #4 Neonatal Resuscitation
Assessing a Newborn- 3 questions!! Is the baby term? Is the baby breathing or crying? Is the baby moving with good tone or is it flaccid? If YES to all, then Clamp and cut cord 7-8 inches from insertion site Place baby with mom Provide warmth, dry baby’s skin Record APGAR

73 Case Scenario #4

74 Case Scenario #4 Neonatal Resuscitation
If NO to any of the 3 questions, then Provide warmth Clear airways if necessary Stimulate baby Check HR: if <100- assist ventilation with bag valve mask Check breathing: if labored or cyanotic- clear airway Re evaluate HR and breathing after intervention

75 Case Scenario #4 Neonatal Resuscitation If HR <60 start compression
Revaluate HR and breathing. If no change consider intubation (hopefully you are in the ER dept) Establish access: umbilical vessels, IV, IO Medication use if condition deteriorates Consider possible narcotic use in mom- narcan for reversal. Pneumothorax, anomalies, cardiac or respiratory defects, blood sugar etc.

76 Case Scenario #5

77 Case Scenario #5 Ectopic pregnancy
Implantation of fertilized ovum outside uterine cavity 2% of all pregnancies in USA Most common cause of maternal mortality in 1st trimester

78 Case Scenario #5 Ectopic Pregnancy- Risk Factors Prior ectopic (15-5%)
Tubal surgery (15-20%) Tubal pathology (90%) PID history (6-9%) Infertility (5%) Sterilization (33%)

79 Case Scenario #5 Ectopic pregnancy Locations: Tubal 96% Ovarian <1%
Cervical<1% Abdominal 1.3%

80

81 Case Scenario #5 Ectopic Pregnancy Signs Abdominal tenderness 91%
1st TM bleeding 79% Tachycardia, low grade fever Cervical motion tenderness Tender pelvic or adnexal mass Chadwick sign Hypoactive bowel sound

82 Case Scenario #5 Ectopic Pregnancy- Symptoms Differential Diagnosis
Onset about 6-7wks after LMP Pelvic pain Vaginal bleeding N/V/D and dizziness Differential Diagnosis Appendicitis Threatened abortion Ruptured ovarian cyst

83 Case Scenario #5 Ectopic pregnancy- Differential Diagnosis Diagnosis
PID Endometritis Kidney stones Normal pregnancy UTI Diagnosis Beta HCG levels Ultrasound

84 Case Scenario #5 Ectopic Pregnancy Treatment Expectant management
Medical- Methotrexate( anti metabolite) Surgical

85 Case Scenario #6

86 Case Scenario #6 Preeclampsia- eclampsia
Form of hypertensive pregnancy specific disorder that occurs after 20wks Characterized by vasospasm, coagulation system activation, hyperreflexia Multitude of Symptoms Categorized: mild vs. severe preeclampsia

87 Case Scenario #6 Preeclampsia-eclampsia Mild pre eclampsia
BP >140/90 +1 urine dip protein or >300mg on 24hrs Severe Preeclampsia BP >160/110 Proteinuria >5g or 3-4+ urine dip Cerebral and visual disturbance Epigastric pain Pulmonary Edema

88 Case Scenario #6 Preeclampsia-eclampsia
Elevated liver enzymes HELLP Cause unknown; possible abnormal placentation or endothelial activation Prevention – no proven therapy Low ASA Calcium Antioxidant eg Vit A

89 Case Scenario #6 Preeclampsia-eclampsia Treatment
Delivery is ONLY known treatment Vaginal delivery unless otherwise indicated Delivery based on gestational age and severity of disease. Treatment Eclamptic Seizure prophylaxis/treatment- Magnesium sulfate IV Antihypertensive therapy SBP > DBP >110

90 Case Scenario #6 Preeclampsia-eclampsia Treatment Postpartum
Monitor coagulation factors and LFTs Aggressive fluid management, risk of pulmonary edema Monitor urine output Postpartum Continue Mg SO4 for 24hrs BP control, 40% recurrence rate.


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