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Jugular venous pressure and waveforms
Dr Bijilesh
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Jugular venous pulse is the oscillating top of the the distended proximal portion of the internal jugular vein and represents volumetric changes that faithfully reflect the pressure cahnges in the right heart
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Right atrial pressure during systole and right ventricular filling pressure during diastole
Window into the right heart, providing critical information regarding its hemodynamics. Right atrial pressure during systole and right ventricular filling pressure during diastole are producing pulsations and pressure waves in jugular veins . Evaluation of the jugular venous pulse offers a window into the right heart, providing critical information regarding its hemodynamics.
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“In the study of venous pulse we have often the direct means of observing the effects of systole and diastole of right auricle and systole and diastole of right ventricle.” James Mackenzie Ging back to a little bit of history.In 1902 james mc established jvp as an important part of cvs examination. According to him jvp provides a good opportunity to observe the effects of systole and diastole of right auricle and systole and diastole of right ventricle.”
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“Precise analysis of the cervical venous pulse and measurement of the height of each wave is not only possible at the bedside but highly desirable” Paulwood Despite this ,analysis of jvp was lacking in the examination till 1950 wen paulwood reawakened the interest eplaining the hemodynamics
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Anatomy JV pressure measurement Causes of elevated JVP Normal wave pattern Abnormal wave pattern Kussmaul s sign and hepatojugular reflux Specific conditions
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Jugular veins Internal jugular vein External jugular vein
Two jugular veins int and ext can be used 4 assessing jv pulse and pressure, though internal one is the preffered one
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The internal jugular vein begins just medial to the mastoid process at the base of the skull. The internal jugular vein runs directly inferior from the mastoid process,. it joins the subclavian vein, to form r innominate which continue as superior vena cava and then into the right atrium.
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Lateral to carotid artery & deep to sternomastoid muscle.
External jugular is superficial to sternomastoid The internal jugular vein is
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Shows the vertical course of the ijv which psses inbetween the two heads of the st mastoid under the medial end of clavicle
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Examination of JVP Right IJV is usually assessed both for waveform and estimation of venous pressure Transmitted pulsations to overlying skin between two heads of sternocleidomastoid Unlike ejv pulsations it is not possible to see ijv pulsations directly as it is deep. We actually see the transmitted pulsations to the overlying skin b/w the two heads of st mast.
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Right IJV Preferred :Why?
Straight line course through innominate vein to the svc and right atrium Less likely extrinsic compression from other structures in neck Why not EJV No or less numbers of valves in IJV than EJV Usually rijv s pref 4 jvp examination. RIJV is less likely affected by extrinsic compression from other structures in neck. Left inno compresssed by arch f aorta and presence of left svc can falsely elevate the jvp on left side. So in conditions with increased symp tone ejv pulsations less visible. Left IJV drains into left innominate vein, which is not in straight line from RA
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Differences between IJV and Carotid pulses
Superficial and lateral in the neck Better seen than felt Has two peaks and two troughs Descents >obvious than crests Digital compression abolishes venous pulse Jugular venous pressure falls during inspiration Abdominal compression elevates jugular pressure Deeper and medial in the neck Better felt than seen Has single upstroke only Upstroke brisker and visible Digital compression has no effect Do not change with respiration Abdominal compression has no effect on carotid pulse
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Estimation of Venous Pressure
Measuring jugular venous pressure Hepatojugular reflux Examining the veins on the dorsum of the hand Assessment of jugular venous pressure at bed side reflect mean right atrial pressure Venous pressure can be roughly assessed
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Measurement of JV Pressure
Sternal angle or angle of Louis - reference point Found approximately 5 cm above the center of the right atrium Sternal angle – RA Fixed relationship Sternal angle or angle of Louis is a surface anatomical mark reference point used for JVP measurement Distance between sternal angle and center of right atrium remains relatively constant regardless of position of the thorax
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In an adult with anatomically normal chestthe sternal angle is found to be 5 cm above the centre of ra in siiting supine or varios reclining angle inbetween. Since tha angle of louis has a fixed relationship with the centre of ra this landmark is used for measuresment os jvp.
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Position of Patient Patient should lie comfortably and trunk is inclined by an angle Elevate chin and slightly rotate head to the left Neck and trunk should be in same line When neck muscles are relaxed ,shine the light tangentially over the skin and see pulsations Simultaneous palpation of the left carotid artery or apical impulse aids in timing of the venous pulsations in cardiac cycle . Or at an angle which permits best view of the upperlevel of the venouspulsations
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Iclination is required as level go below mandible in high jvp or below clavi in low pr. So start with 45 then tilt pt appropriately up or down . In patients with low jugular pressure , a lesser (<30‘) inclination is desirable In patients with high jugular pressure ,a greater (60-90‘) inclination is required to obtain visible pulsations
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Measurement of JVP Two scale method is commonly used
Normally JV pressure does not exceed 3- 4 cm above the sternal angle Since RA is approximately 5 cm below the sternal angle , the jugular venous pressure corresponds to 9 cm =7mmhg Elevated JVP : JVP of >4 cm above sternal angle . (9 cm column of water / 1.3 =6.9)
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.A horizontal scale at the top of the oscillating venous column in IJV cuts the vertical scale at the sternal angle gives JV pressure in cm of water Add 5 cm to measurement since right atrium is 5 cm below the sternal angle
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Elevated JVP Increased RVP and reduced compliance: Pulmonary stenosis
Pulmonary hypertension Right ventricular failure RV infarction RV inflow impedance: Tricuspid stenosis / atresia RA myxoma Constrictive pericarditis
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Elevated JVP Circulatory overload : Renal failure Cirrhosis liver
Excessive fluid administration SVC obstruction
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Kussmaul's sign Mean jugular venous pressure increases during inspiration Constrictive pericarditis Severe right heart failure RV infarction Restrictive cardiomyopathy Impaired RV compliance. Normally mean jvp falls during insp ..as a result of impaired RV compliance, so the increased venous return cannot be accomodated by the rv, resulting in an elevated jvp
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Abdominal -Jugular Reflux
Hepatojugular reflux – Rondot (1898) Apply firm pressure to periumbilical region sec Normally JV pressure rises transiently to < 1cm while abdominal pressure is continued If JV pressure remains elevated >1cm until abdominal pressure is continued: Positive AJR. Apply firm pressure to periumbilical region for sec with patient lying comfortably and breathing quietly while observe JVP ABRAMS
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Abdominal compression forces venous blood into thorax.
A failing/dilated RV not able to receive venous return without rise in mean venous pressure. Positive AJR Incipient and or compensated RVF Tricuspid regurgitation COPD Copd sudden disproportionate increase in intrathoracic pr due to altered intrathoracic pr relation ships in copd
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Hepatojugular reflux
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Gaertner's method Measurement of JVP by examining the veins on the dorsum of the hand When patient sitting or lying at a 45‘ elevation , arm slowly and passively raised from dependant position until the vein collapses Height of the limb above the level of sternal angle at which vein collapses represents the venous pressure When venous pressure is normal , veins of hand collapse at the level of sternal angle
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Normal JVP Normal JVP reflects phasic pressure changes in RA during systole and RV during diastole Two visible positive waves ( a and v) and two negative troughs ( x and y) Two additional positive waves can be recorded C wave interrupts x descent and h wave and h wave precedes the next a wave
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Normal JVP Waveform Consists of 3 positive waves a,c & v
And 3 descents x, x'(x prime) and y
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a Wave First positive presystolic a wave is due to right atrial contraction Effective RA contraction is needed for visible a wave Dominant wave in JVP and larger than v It precedes upstroke of the carotid pulse and S1, but follow the P wave in ECG First positive presystolic a wave is due to right atrial contraction results in retrograde blood flow in to svc and jugulars
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x Descent Systolic x descent is due to atrial relaxation during atrial diastole X descent is most prominent motion of normal JVP which begins during systole and ends just before S2 It is larger than y descent X descent more prominent during inspiration
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C Wave Not usually visible. Two different causes
- Transmitted carotid artery pulsations. - Upward bulge of closed Tricuspid valve in isovolumic systole Second positive wave recorded in JVP which interrupts the x descent
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x` Descent x`descent is systolic trough after c wave Due to
Fall of right atrial pressure during early RV systole Downward pulling of the TV by contracting right ventricle Descent of RA floor
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v Wave Begins in late systole ends in early diastole
Rise in RA pressure due to continued RA filling during ventricular systole when tricuspid valve closed Roughly synchronous with carotid upstroke and corresponds S2 . Third positive wave in JVP which begins in late systole and ends in early diastole
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y Descent Diastolic collapse wave (down slope v wave)
It begins and ends during diastole well after S2 Decline of RA pressure due to RA emptying during early diastole when tricuspid valve opens
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h wave Small brief positive wave following y descent just prior to a wave Described by Hieschfelder in 1907 It usually seen when diastole is long With increasing heart rate, y descent immediately followed by next a wave . Small brief positive wave following y descent just prior to a wave during period of diastasis It usually seen when diastole is long (as in slow heart rates)
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Prominent a Wave Forceful atrial contraction when there is resistance to RA emptying or increased resistance to ventricular filling RV inflow obstruction: Tricuspid stenosis or atresia RA mxyoma Decreased ventricular compliance: Pulmonary stenosis Pulmonary hypertension of any cause RV infarction RV cardiomyopathy (HOCM) Acute pulmonary embolism 3. Increased RVEDP. RV infarction , Acute pulmonary embolism
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Large a wave
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Cannon Waves Whenever RA contracts against closed TV valve during RV systole Regular cannon waves: Junctional rhythm VT with 1:1 retrograde conduction Isorhythmic AV dissociation Irregular cannon waves : Complete heart block Ventricular tachycardia Ventricular pacing or ventricular ectopics . Venous corrigan by paul wood
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Rgular cannon waves
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Absent a Wave When no effective atrial
contraction as in atrial fibrillation In sinus tachycardia , when a wave may fuse with preceding v wave , especially when the PR interval is prolonged .
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Prominent x descent Presence of atrial relaxation with intact tricuspid valve and good RV contraction Causes : Cardiac tamponade Constrictive pericarditis is needed for prominent x descent.
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Reduced x descent Moderate to severe TR: early sign
Atrial fibriillation Obliteration of x descent with early large systolic wave
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Prominent v wave Increased RA volume during ventricular
systole produce prominent v wave Severe TR : giant v wave Giant v wave sometimes causes : systolic movement of ear lobe head bobbing with each systole systolic pulsation of liver pulsatile exophthalmos (Lanci sign) – ventricularization of atrial / jugular pressure
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cv wave
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PROMINENT V WAVE ASD with mitral regurgitation
VSD of LV to RA shunt (Gerbode's defect) RV failure
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Rapid y Descent Severe TR
C .Pericarditis (Friedreich's sign): Early rapid ventricular filling Severe RV failure ASD with mitral regurgitation Elevated venous pressure from any cause. Constrictive pericarditis,(Friedreich’s sign) Severe TR, Severe RVF Severe RV infarction
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Slow y Descent When RA emptying and RV filling are impaired y descent is slow and gradual Tricuspid stenosis Right atrial tumours Pericardial tamponade( y descent may even be absent).
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Respiratory influences
Inspiration – increased visibility of venous pulse. Mean venous pressure falls , but the wave forms are accentuated during inspiration.
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Waves more prominent during inspiration
X descent more brisk Increased venous return augment RA contraction and hence relaxation >> brisk x Also increased venous return augment RV volume and contraction > increased systolic descent of floor of RA>>brisk x’
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JUGULAR VENOUS PULSE IN ARRHYTHMIAS
‘a’ & ‘v’/c (carotid pulse correlates with P & QRS complex in ECG. Normal sinus rhythm is characterized by sequential a & v waves. Any disturbance in this wave form indicates rhythm abnormality. A wave occurring along 1st heart sound – normal PR interval.
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Rhythm Cannon waves Sinus a - v regular Absent I AV block
Pr0longed AC interval Wenckebach’s Gradual prolongation of A-C interval Mobitz II block Constant AC interval followed by sudden skipping of carotid pulse CHB Variable Present & irregular VPC JPC Early cycle Present APC VT Atrial tachycardia Normal
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Cardiac tamponade JVP is usually elevated
y descent is diminished or absent x wave is normal Kussmaul's sign usually negative
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Constrictive pericarditis
JVP is elevated a wave is usually normal v wave is usually equal to a wave x descent –prominent y descent – rapid descent Kussmauls sign is usually positive
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Restrictive cardiomyopathy
JVP is usually elevated Both a and v wave equal Kussmaul s may be positive
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Pulmonary Hypertension
Early RV decompensation : JVP may be elevated a wave is prominent Decompensated RVF: a and v wave prominent , v wave larger than a wave x descent is diminished or absent Rapid y descent due to TR
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JVP in ASD JVP is normal and equal a and v waves
Elevated JVP may seen in severe PAH and in RVF Prominent a wave with PS and MS Prominent v wave with PAH and RVF with TR Rapid y descent with RVF or TR
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JVP in VSD Elevated JVP with CHF Prominent v wave with Gerbode's shunt
In Eisenmenger complex : JV Pressure usually normal Normal a and v waves
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Ebstein Anomaly JVP is usually normal
Attenuated x descent and systolic v wave are not reflected in jugular pulse despite appreciable TR Damping effect of large capacitance RA and thin, toneless atrialized RV Unimpressive JVP is attributed to
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Thank you………
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