Presentation is loading. Please wait.

Presentation is loading. Please wait.

Fig. 5. Onecut transcription factors are important for the correct generation of the mdDA neuronal population.(A) Schematic representation of the region.

Published bySuparman Kusnadi Modified over 5 years ago

Similar presentations

Presentation on theme: "Fig. 5. Onecut transcription factors are important for the correct generation of the mdDA neuronal population.(A) Schematic representation of the region."— Presentation transcript:

1 Fig. 5. Onecut transcription factors are important for the correct generation of the mdDA neuronal population.(A) Schematic representation of the region analyzed for Th immunohistochemistry as shown in (B). Onecut transcription factors are important for the correct generation of the mdDA neuronal population.(A) Schematic representation of the region analyzed for Th immunohistochemistry as shown in (B). (B) Sagittal sections showing the organization of the mdDA neuronal population through the presence of Th protein. The arrow indicates a region where a severe flattening of the neuronal population is present in the Oc1−/− and Oc1/2−/− mice. (C) Coronal sections from rostral (R) to caudal (C) encompassing the mdDA neuronal population as shown by Th protein expression. The flattening of the mdDA neuronal pool as observed in the sagittal sections (B) is confirmed in these sections as indicated by arrows. Koushik Chakrabarty et al. Biology Open 2012;bio © Published by The Company of Biologists Ltd

Download ppt "Fig. 5. Onecut transcription factors are important for the correct generation of the mdDA neuronal population.(A) Schematic representation of the region."

Similar presentations

Structure of abbreviated dystrophins.

Structure of abbreviated dystrophins.
Fig. 2. Effect of endurance training on gene expression, and protein content and activity in heart muscle. Effect of endurance training on gene expression,

Fig. 2. Effect of endurance training on gene expression, and protein content and activity in heart muscle. Effect of endurance training on gene expression,
Fig. 2. Outline of the two types of stimulus sequences employed in the analysis.(A) Environment information stimuli; (B) adaptation stimuli. Outline of.

Fig. 2. Outline of the two types of stimulus sequences employed in the analysis.(A) Environment information stimuli; (B) adaptation stimuli. Outline of.
Fig. 3. External work and kinetic and potential energy of the whole body for the mean individual (individual 10) in the mean population (population 1)

Fig. 3. External work and kinetic and potential energy of the whole body for the mean individual (individual 10) in the mean population (population 1)
Representative illustrations of a sagittal lung section stained with H&E in a control and a CS-exposed mouse after 6 months. Representative illustrations.

Representative illustrations of a sagittal lung section stained with H&E in a control and a CS-exposed mouse after 6 months. Representative illustrations.
Fig. 2. Morphological changes of cultured adherent fibroblastic cells after OA treatment related to actin microfilament reorganization.(A) Cells observed.

Fig. 2. Morphological changes of cultured adherent fibroblastic cells after OA treatment related to actin microfilament reorganization.(A) Cells observed.
Fig. 7. Motion adaptation increases time-dependent response modulations (TDRM) relatively to the average cell response.TDRM normalized to the value obtained.

Fig. 7. Motion adaptation increases time-dependent response modulations (TDRM) relatively to the average cell response.TDRM normalized to the value obtained.
Fig. 2. Proportion of motile objects and track length

Fig. 2. Proportion of motile objects and track length
Fig. 1. Muscle-specific PITX1 over-expression in the Pitx1 transgenic mice.(A) Detection of Pitx1 mRNA expression in muscles of the Pitx1 transgenic mice.

Fig. 1. Muscle-specific PITX1 over-expression in the Pitx1 transgenic mice.(A) Detection of Pitx1 mRNA expression in muscles of the Pitx1 transgenic mice.
Fig. 4. A primary screen based on scrape closure

Fig. 4. A primary screen based on scrape closure
Fig. 6. Cross-section of the stomach wall and spiral intestine of the embryo, stained with PAS. (A) Surface of the stomach wall (SW) and ingested material.

Fig. 6. Cross-section of the stomach wall and spiral intestine of the embryo, stained with PAS. (A) Surface of the stomach wall (SW) and ingested material.
Fig. 6. The cholinergic receptor subunits a6 (Chrna6) and b3 (Chrnb3) are (subset) specifically expressed in mdDA neurons during development.(A) The Chrna6.

Fig. 6. The cholinergic receptor subunits a6 (Chrna6) and b3 (Chrnb3) are (subset) specifically expressed in mdDA neurons during development.(A) The Chrna6.
Fig. 6. Comparison between the response against transformed tissues and capsule formation.At the cellular level the two responses share many similarities.

Fig. 6. Comparison between the response against transformed tissues and capsule formation.At the cellular level the two responses share many similarities.
Fig. 7. Knockdown of Meis1 abolishes CR4. 2-GFP expression

Fig. 7. Knockdown of Meis1 abolishes CR4. 2-GFP expression
Fig. 2. Mapping of the interaction domain on coilin for association with the dyskerin complex. Mapping of the interaction domain on coilin for association.

Fig. 2. Mapping of the interaction domain on coilin for association with the dyskerin complex. Mapping of the interaction domain on coilin for association.
Embryonic corneal defects in E18.5 AP-2β NCC KO mutant embryos.

Embryonic corneal defects in E18.5 AP-2β NCC KO mutant embryos.
Fig. 2. Body weight and size analysis of A1/A2-KO mice

Fig. 2. Body weight and size analysis of A1/A2-KO mice
Fig. 6. Schematic diagram showing distribution and dynamics of four E-cadherin populations within the ROI of a FRAP experiment. Schematic diagram showing.

Fig. 6. Schematic diagram showing distribution and dynamics of four E-cadherin populations within the ROI of a FRAP experiment. Schematic diagram showing.
Fig. 1. Lack of Hmga1 and Hmga2 expression in A1/A2-KO mice

Fig. 1. Lack of Hmga1 and Hmga2 expression in A1/A2-KO mice
Fig. 4. The model of malate metabolism in fruit cells under different K level conditions. The model of malate metabolism in fruit cells under different.

Fig. 4. The model of malate metabolism in fruit cells under different K level conditions. The model of malate metabolism in fruit cells under different.

Similar presentations

Ads by Google