1. Abdominal Hemangiomas: A Pictorial Review of Unusual, Atypical, and Rare Types 
Vijayanadh Ojili, MD, Sree Harsha Tirumani, MD, Gowthaman Gunabushanam, MD, Arpit Nagar, MD, Venkateswar Rao Surabhi, MD, Kedar N. Chintapalli, MD, John Ryan, MD, FRCPC 
Canadian Association of Radiologists Journal 
Volume 64, Issue 1, Pages (February 2013)
DOI: /j.carj
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2. Figure 1
Atypical sclerosing hepatic hemangioma. (A, B) Axial contrast computed tomography image of the liver in the portal venous phase, showing an irregular hypodense tumour (arrows), with heterogenous enhancement and central calcification. Biopsy of the lesion showed cavernous hemangioma.
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3. Figure 2
Giant multiloculated hepatic hemangioma. (A-C) Axial postcontrast T1-weighted magnetic resonance images in arterial (A), venous (B), and 2-minute delayed (C) phases, showing large septated hypointense lesion (asterisks) with nodular (white arrows) enhancing septae.
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4. Figure 3
Splenic hemangioma. (A) Ultrasound image of the spleen, showing a well-marginated hyperechoic tumour (asterisk). (B) Axial contrast computed tomography image, showing heterogenous enhancing lesion in the portal venous phase (asterisk).
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5. Figure 4
Splenic hemangioma. (A, B) Axial dynamic contrast-enhanced T1-weighted magnetic resonance images in venous (A) and 5-minute delayed (B) phases, showing a hypointense splenic lesion with progressive centripetal filing (arrows). Note the incidental hepatic cysts (asterisks).
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6. Figure 5
Gastric hemangioma in 2 different patients. (A, B) Axial contrast-enhanced computed tomography (CT) images in arterial (A) and venous (B) phase, showing circumferential thickening of body of the stomach (asterisks) with progressive enhancement. (C) Axial contrast-enhanced CT image of another patient in the venous phase, showing a large exophytic tumour with nodular enhancement arising from the gastric wall (asterisk).
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7. Figure 6
Renal hemangioma in 2 different patients. (A, B) Axial contrast computed tomography (CT) images in arterial (A) and venous (B) phases, showing a peripheral nodular enhancing tumour (arrowheads) arising from the renal papilla. (C) Coronal reformatted CT image of another patient in late arterial phase, showing a large enhancing tumour with phleboliths (asterisk) filling the renal pelvis.
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8. Figure 7
Perirenal hemangioma. (A, B) Axial contrast computed tomography (CT) images in the portal venous phase, demonstrating an enhancing tumour (asterisks), with phleboliths in relation to the upper pole of the right kidney. (C, D) Coronal and sagittal reformatted CT images, showing the tumour (asterisks) separate from the normal right adrenal gland (arrows).
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9. Figure 8
Gastrosplenic ligament Hemangioma. (A, B) Axial contrast computed tomography in venous (A) and delayed (B) phases, showing a well-defined hypodense lesion (asterisks) in the gastrosplenic ligament with nodular enhancement. (C) Coronal true fast imaging with steady state precession magnetic resonance image (MRI), showing the tumour to be a T2 hyperintense tumour (asterisk). (D) Axial contrast-enhanced T1-weighted MRI in venous phase, showing nodular enhancement in the tumour (asterisk).
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10. Figure 9
Retroperitoneal hemangioma. (A) Axial unenhanced computed tomography (CT) sections, showing a well-defined lesion (arrow) adjacent to the inferior vena cava. (B, C) Axial contrast-enhanced CT images in arterial and venous phases, showing peripheral enhancement and progressive central filling (arrows).
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11. Figure 10
Psoas muscle hemangioma. (A) Axial non–fat-saturated T1-weighted magnetic resonance images (MRI), demonstrating a well-defined hyperintense lesion in the left psoas (arrow). (B) Axial T2-weighted MRI, showing the lesion to be hyperintense (arrow).
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12. Figure 11
Hemangioma of the inferior vena cava (IVC). (A) Ultrasound image of intrahepatic IVC, showing a well-defined echogenic lesion in the wall (arrow). (B) Coronal reformatted contrast computed tomography image, showing the lesion as a filling defect in IVC with mild enhancement (arrow). (C, D) Axial T2-weighted and postcontrast T1-weighted magnetic resonance images, showing a hyperintense lesion with enhancement (arrows).
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13. Figure 12
Hemangioma of abdominal wall. Axial computed tomography image in the arterial phase, showing serpiginous vascular channels (arrow) in the left lateral abdominal wall.
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14. Figure 13
Inguinal canal hemangioma. Axial contrast-enhanced computed tomography sections of pelvis, showing enhancing irregular soft-tissue lesion (arrow) with phleboliths along the spermatic cord.
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15. Figure 14
Klippel-Trénaunay syndrome. (A) Ultrasound image of the spleen, showing multiple hyperechoic lesions (white arrows) in the spleen, which represent hemangiomas. (B, C) Axial and (D) coronal reformatted contrast computed tomography images, showing hemangiomas in the liver (long white arrows), spleen (black arrow heads), left adrenal gland (black arrow), and right upper thigh (white arrow head). Thickened rectosigmoid with phleboliths (small white arrows) represents colorectal hemangiomatosis. (E) Axial true fast imaging with steady-state precession magnetic resonance images show T2 hyperintense hemangiomas in the liver (thick white arrows), left adrenal gland (thin white arrow), and spleen (black arrow heads).
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16. Figure 15
Giant hepatic hemangioma causing Kasabach-Meritt syndrome. (A, B) Axial contrast-enhanced computed tomography images in arterial and venous phases, showing a large nodular enhancing lesion (arrows), replacing almost the entire right lobe of liver. The patient developed consumptive coagulopathy.
Canadian Association of Radiologists Journal  , 18-27DOI: ( /j.carj )
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