1. Regional Myocardial Perfusion After Experimental Subarachnoid Hemorrhage
by Jonathan G. Zaroff, Guy A. Rordorf, James S. Titus, John B. Newell, Nicholas J. Nowak, David F. Torchiana, H. Thomas Aretz, and Michael H. Picard
Stroke
Volume 31(5):
May 1, 2000
Copyright © American Heart Association, Inc. All rights reserved.

2. Example of CBN from an SAH dog.
Example of CBN from an SAH dog. The characteristic pattern of myofibrillar degeneration is noted. This lesion is histologically distinct from coagulative necrosis, which is characteristic of myocardial infarction.
Jonathan G. Zaroff et al. Stroke. 2000;31:
Copyright © American Heart Association, Inc. All rights reserved.

3. Example of ECG changes from an SAH dog.
Example of ECG changes from an SAH dog. Baseline tracings (left) have isoelectric ST segments. Tracings on the right demonstrate inferior ST depression and lateral ST elevation, which developed 4 hours (4°) after SAH.
Jonathan G. Zaroff et al. Stroke. 2000;31:
Copyright © American Heart Association, Inc. All rights reserved.

4. Example of myocardial contrast echocardiography from a SAH dog.
Example of myocardial contrast echocardiography from a SAH dog. Left, Precontrast image. RV indicates right ventricle. Right, Contrast-enhanced image demonstrates homogeneous perfusion. Arrow indicates a region of transient attenuation that showed normal perfusion in subsequent frames.
Jonathan G. Zaroff et al. Stroke. 2000;31:
Copyright © American Heart Association, Inc. All rights reserved.

5. Global MBF as determined by radiolabeled microspheres vs time in SAH and control dogs.
Jonathan G. Zaroff et al. Stroke. 2000;31:
Copyright © American Heart Association, Inc. All rights reserved.

6. Ratio of subendocardial (subendo)/subepicardial (subepi)MBF vs time in SAH and control dogs.
Jonathan G. Zaroff et al. Stroke. 2000;31:
Copyright © American Heart Association, Inc. All rights reserved.