1. Lab 2
أعزائي الطلبة هذه الورقة تحتوي فقط على ما قيل عن هذه الصور زيادة عن النظري وأي معلومة ذكرت في النظري وتكررت في العملي لن تجدوها مذكورة هنا ولكنكم مطالبون بمعرفتها بالتأكيد

2. Fibrinoid necrosis
Chronic inflammatory cells attacking the vessel wall
Robbins

3. TB-caseating necrosis
Cheese-like
Robbins
Emphysematous changes

4. Fibrillary background due to glial cell processes
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Macrophages in liquefactive necrosis of brain (brain infarction)
Neurons (large nucleus, prominent nucleolus and bulky cytoplasm)
Small nuclei are glial cells

5. Brain atrophy
Narrowed gyri
Widened sulci
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Changes are mainly frontally (Alzheimer case)

6. Kidney coagulative necrosis
Robbins
glomerulus

7. Alzheimer
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Tau proteins are the components of these “neurofibrillary tangles”

8. Degeneration and absence of nucleiMI
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Inflammatory cells
Degeneration and absence of nuclei

9. Cardiac hypertrophy
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10. Barret esophagus
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Columnar cell metaplasia
..may be like stomach foveolar epithelium
or goblet cells like intestine (intestinal metaplasia)
glands

11. Calcification (arrows)
Stroma (connective tissue)
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Glands of stomach body/fundus (zymogenic granules in chief cells)
vessel

12. Lipid-laden macrophages in bone marrow (storage disease)
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13. Tingible body macrophages eating apoptotic fragments of lymphocytes in thymus
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14. Robbins

15. Fatty liver (steatosis)
Non-alcoholic steatohepatitis (NASH)
Alcoholic steatohepatitis (ASH)
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16. Mallory hyaline inclusions (keratin protein) (characteristic but not only in alcoholic liver disease)
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17. Perl’s or Prussian blue stain
..Iron…blue
Hemosiderosis is due to hereditary hemochromatosis or other causes such as multiple blood transfusions (as in thalassemia major cases)
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18. Lipofuscin in liver…atrophy or autophagy…etc.
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Needs Perl’s stain to exclude iron
Liver (trabeculae of polygonal hepatocytes)

19. Hepatocyte atrophy with lipofuscin
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