Transmissible Spongiform Encephalopathies (Prion disorders)

Slides:



Advertisements
Similar presentations
Distinctive characteristics
Advertisements

Prion Disease Transmissible spongiform encephalopathy (TSE)
TAYLOR GOLDBECK Brain Trust Chapters 8 & s- Farmers report sheep with scrapie-like symptoms Creutzfeldt observes a patient with CJD 1918-
Prions Sly Richards.
Presented by Béla Reiz Supervisor: Dr. Liang Li
Mad Cow Disease 袁聖甯 黃竹瑄 鄧雯心. What is Mad Cow Disease ? A kind of transmissible spongiform encephalopathies (TSE) Occurs in many mammals, including human.
Transmissible Spongiform Encephalopathy TSE. Center for Food Security and Public Health Iowa State University Overview Organism History Epidemiology.
Do Now: Complete the chart on your notes as completely as possible.
1 How did viruses originate? “Viral Mutation Evolved” Media: Pastel on paper WALTER DUPRIEST acanthusarts.org.
1 Transmissible Spongiform Encephalopathies. 2 Kuru Since the early 1900’s the Fore people of New Guinea have honored their dead by cooking and consuming.
Viruses Discovered in 1898 as the cause of hoof-and-mouth disease in cattle. The disease-causing organism was much smaller than bacteria—it could pass.
Bovine Spongiform Encephalopathy Luke VanNatter Carrie Pell Amy Richwine Scott Inskeep Kristina Anderson.
Prions Fact or Science Fiction?. Stanley Prusiner, 1982 Born in Des Moines, Ia. Suggested that spongiform encephalopathies in animals and humans are caused.
L and D isomers of amino acids. Ionization state as a function of pH.
Prions Alicia Arguelles, Jerry Wang May 4, What are prions? proteinaceous infectious particle an infectious agent made only of protein, containing.
Mad Cow Disease. Effects of Mad Cow disease Mad cow disease, or bovine spongiform encephalopathy (BSE), is a fatal brain disorder that occurs in cattle.
© Elsevier, 2011.Principles of Molecular Virology Subviral Agents Satellites and viroids – parasites of parasites! Prions - infectious protein molecules.
Prion Diseases Microbes and Society Fall What is a Prion? Prion- small proteinaceous infectious particles which resist inactivation by procedures.
Mad Cow Disease The Past History of Mad Cow Disease
Topics in ten…. Bacteria Size (500nm – several um) Genetics (HGT) Gram type (peptidoglycan) Metabolism Reproduction Pathogenesis.
Viruses Living or Not ???????.
Transmissible Spongiform Encephalopathies (TSEs) a.k.a. Prion Diseases Transmissible  can be spread Spongiform  resembling a sponge Encephalopathies.
Transmissible Spongiform Encephalopathy Prion Protein Diseases Lisa Kennedy, Dylan Bradford, Madi Hoagland Henefield, Anders Ohman Advisor: Dr. Todd Livdahl.
By Shon Augustine. Mad cow disease is an incurable, fatal brain disease that affects cattle and possibly some other animals, such as goats and sheep.
Prions: Proteins Gone Bad
What do the following have in common?.
1 An Introduction to the Viruses. 2 Viral Components All viruses have capsids- protein coats that enclose & protect their nucleic acid Viruses may have.
Labs start today, or Thursday Inquiry 1 proposal due 9/9 or 9/13 Class communication via Blackboard, webpage, and/or .
PRIONS Kalina Estrada TA: Yu-Chen Hwang Thursday, 7-8pm.
EEE 105 Name: eee105 Password: prAIRie Agriculture.
Prions and Protein Misfolding BICH 107 GENE 105. Kuru Discovered in Papua New Guinea in early 1900's "Trembling with fear" Characterized by headaches,
U.S. Department of the Interior U.S. Geological Survey Chronic Wasting Disease Wildlife Agency Perspective Leslie Dierauf, V.M.D. USGS National Wildlife.
Chronic Wasting Disease (CWD) David Litts. What is it? Prions (infectious incomplete protein) Prions (infectious incomplete protein) Creutzfeldt-jakob.
 Foreign Sources of Infection To − Vi Nguyen. Foreign Infection  Preventable environmental source of infection  Remove infectious material, epidemic.
Prion. Similar to Viruses Atypical Agents  Small  Filterable  Need host cells  No machinery for energy generation of protein synthesis.
 slowly progressive, degenerative, and fatal disease affecting the central nervous system of adult cattle.  abnormal version of a protein normally found.
Prions_Stanley B. Prusiner Wen-Chun Shaw Wen-Chun Shaw BTC SCIENTIFIC DISCOVERY BTC SCIENTIFIC DISCOVERY Dr. Vankley Dr. Vankley.
The History of Chronic Wasting Disease Dr. Trent Bollinger, CCWHC One World, One Health Symposium Sept. 29, 2004.
MICROORGANISMS: Viruses, Prions, Archaea. What do Archaea and Bacteria have in common? Single celled Microscopic No membrane bound nucleus Both essential.
Prion Propagation in Response to Temperature and Osmotic Stress Jess Dhillon Department of Biological Sciences, York College of Pennsylvania Introduction.
Labs start next week (first reflection due)
Kuru A PRION DISEASE.
Prions “Scrapie” “mad cow disease” Nobel Prize 1997
Microbes and Diseases Chapter 02. CREUTZFELDT-JAKOB DISEASE Prion.
What do the following have in common?.
Studying Biology: Start with a question. –For example: How? Why? When? Where? Etc? How do we get answers? –Strong Inference presents one method (article.
What do the following have in common?.
Mad Cow Disease By: Rylee and Cassidy. What is it? Mad Cow disease is a incurable, fatal brain disease that affects cattle and sometimes goats and sheep.
Dr. Stuart Reichler Bio 311D. DateSubjectTextbook Chapters Jan 14Introduction, Philosophy of Science 16Strong Inferencearticle on webpage 18, 23Meiosis12,
The Prion Diseases Stanley B. Prusiner.. Description & History Fifteen years ago I evoked a good deal of skepticism when I proposed that the infectious.
1 Chapter 13 Viruses, Viroids & Prions. 2 Viruses - General Characteristics obligate intracellular parasites (completely inert when not in a host cell)
By: Terry Bender and Dustin Rozier Students of Dr. James Corbett Lowndes County High School Valdosta, Georgia November, 2002.
Today’s Lecture: Bringing the disease to YOU! From the 1950s to NOW From Papua New Guinea to the U.S.A.
Prion diseases (transmissible spongiform encephalopathies) Dr. Mohammad Shakeeb, MD Specialist in clinical pathology/Microbiology and immunology.
History of Neurology Stanley B. Prusiner MD 1942-present May 8, 2017
Petra Jenišová Veronika Plačková Magdaléna Trojanová
MICROBIAL FOOD SAFETY A FOOD SYSTEMS APPROACH
Studying Biology: Start with a question. How do we get answers?
Copyright © 2017 American Academy of Pediatrics.
Rabies virus and Prion Dongli Pan
Brain Trust Chapt 22: Origins
Chronic Wasting Disease
Dr. Stuart Reichler Bio 325.
Studying Biology: Start with a question. How do we get answers?
Labs start today Inquiry 1 proposal due 9/10 or 9/14
Viruses Section 18-2.
PRIONS.
Viruses Living or Not ???????.
Studying Biology: Start with a question. How do we get answers?
Studying Biology: Start with a question. How do we get answers?
Presentation transcript:

Transmissible Spongiform Encephalopathies (Prion disorders)

Forms of Transmissible Spongiform Encephalopathy Scrapie Creutzfeldt-Jakob disease Gerstmann-Straussler-Scheinker syndrome Kuru BSE vCJD nvCJD All associated with PRIONS

Animals Effected by Transmissible Spongiform Encephalopathy Sheep, goats, deer, elk, mink, domestic cats, zoo species, cattle and humans

BSE First seen in UK in 1986 Linked to cattle food supplemented with rendered offal from slaughtered sheep Perhaps stimulated and amplified by the use of air embolus to slaughter cattle (captive bolt gun) Both are temporally related to the advent of BSE

Prions Proteinaceous infectious particle Discovered by Dr. Stanley Prusiner Prusiner awarded 1997 Nobel prize for physiology/medicine Protein composition Apparently capable of self reproduction Contains no DNA or RNA Very resistant to high temperatures and disinfection

Feedback: Privacy Policy Feedback
Do Not Sell My Personal Information
About project: SlidePlayer Terms of Service

© 2024 SlidePlayer.com Inc. All rights reserved.