Takashi Nitta, MD, Masataka Mitsuno, MD, Chris K

Slides:



Advertisements
Similar presentations
Date of download: 6/25/2016 Copyright © The American College of Cardiology. All rights reserved. From: Mechanism, localization and cure of atrial arrhythmias.
Advertisements

Date of download: 6/26/2016 Copyright © The American College of Cardiology. All rights reserved. From: Mechanism and Location of Atrial Flutter in Transplanted.
Date of download: 9/18/2016 Copyright © The American College of Cardiology. All rights reserved. From: Characteristics of electrograms recorded at reentry.
Volume 4, Issue 1, Pages (January 2007)
Radiofrequency Ablation for Atrial Tachycardia and Atrial Flutter
Reentrant and Focal Activations During Atrial Fibrillation in Patients With Atrial Septal Defect  Takashi Nitta, MD, PhD, Shun-ichiro Sakamoto, MD, PhD,
Mark D. Rodefeld, MDa §, Burt I. Bromberg, MDb §, Richard B
Circ Arrhythm Electrophysiol
Volume 7, Issue 1, Pages (January 2010)
Clinical Intracardiac Electrophysiologic Testing: Technique, Diagnostic Indications, and Therapeutic Uses  STEPHEN C. HAMMILL, M.D.  Mayo Clinic Proceedings 
Fei Lü, MD, PhD, FACC, FHRS, Taibo Chen, MD, Kenneth K
Safety and efficacy of pericardial endoscopy by percutaneous subxyphoid approach in swine heart in vivo  Takehiro Kimura, MD, Shunichiro Miyoshi, MD,
Jennifer S. Lawton, MD, Thomas A. D'Amico, MD 
Connexins and the atrioventricular node
Volume 9, Issue 1, Pages (January 2012)
Congenital and surgically acquired Wolff-Parkinson-White syndrome in patients with tricuspid atresia  Alfred Hager, MD, Bernhard Zrenner, MD, Silke Brodherr-Heberlein,
James L. Cox, MD, John P. Boineau, MD, Richard B
Left-sided atrial flutter: Characterization of a novel complication of pediatric lung transplantation in an acute canine model  Sanjiv K. Gandhi, MDa*,
Jeffrey P. Moak, MD, Marco A. Mercader, MD, Dingchao He, MD, TK
Risk Factors of Recurrence of Atrial Fibrillation (AF) After AF Surgery in Patients With AF and Mitral Valve Disease  Yosuke Ishii, MD, PhD, Shun-ichiro.
Shun-ichiro Sakamoto, MD, Richard B. Schuessler, PhD, Anson M
Radial approach: a new concept in surgical treatment for atrial fibrillation. II. Electrophysiologic effects and atrial contribution to ventricular filling 
Peter Lukac, MD, PhD, Vibeke E. Hjortdal, MD, PhD, Anders K
Wolff-Parkinson-White syndrome due to a left atrial appendage–to–left ventricular connection: A case of a successful pathway elimination from inside of.
Regional distribution of atrial electrical changes induced by stimulation of extracardiac and intracardiac neural elements  Pierre L. Pagé, MDa,b, Nazih.
Concurrent multiple left atrial focal activations with fibrillatory conduction and right atrial focal or reentrant activation as the mechanism in atrial.
Radial approach: a new concept in surgical treatment for atrial fibrillation I. Concept, anatomic and physiologic bases and development of a procedure 
Incessant bundle branch reentrant ventricular tachycardia in a patient with corrected transposition of the great arteries  Ken Kato, MD, Daigo Yagishita,
Federico Milla, MD, Nikolaos Skubas, MD, William M
Volume 11, Issue 1, Pages (January 2014)
Volume 13, Issue 10, Pages (October 2016)
Volume 15, Issue 1, Pages (January 2018)
Peri–coronary sinus atrial flutter associated with prior slow pathway ablation  Mitsunori Maruyama, MD, PhD, FHRS, Shunsuke Uetake, MD, PhD, Yasushi Miyauchi,
Assessment of a conduction-repolarisation metric to predict Arrhythmogenesis in right ventricular disorders  C.A. Martin, M. Orini, N.T. Srinivasan, J.
Andreu Porta-Sánchez, MD, Andrew C. T
Successful catheter ablation of recipient left ventricular tachycardia 26 years after heterotopic heart transplantation  Mikael Laredo, MD, Shaida Varnous,
A case of scar-related ventricular tachycardia demonstrating termination with nonglobal capture at the site of concealed entrainment with dual slow conduction.
Right ventricular outflow tract resection for treatment of refractory ventricular tachycardia in a 2-year-old child  S. Kanaan, MD, K. Shamsuddin, MD,
Paul Knops, BSc, Charles Kik, MD, Ad J. J. C
Irregular wide QRS complex tachycardia in a patient with pulmonary hypertension: What is the mechanism?  Abigail Louise D. Te, MD, Fa-Po Chung, MD, Yenn-Jiang.
Dual-loop biatrial concomitant macroreentrant tachycardia in a patient without previous history of surgery or ablation  Song-Yun Chu, MD, Li-Bin Shi,
Yuichiro Kawamura, MDa,, Pierre L
Pseudo typical atrial flutter occurring after cavotricuspid isthmus ablation in a patient with a prior history of Senning operation  Naoki Yoshida, MD,
Isolation of the conduction between the Marshall bundle and distal coronary sinus and the entire coronary sinus for an atrial tachycardia after catheter.
Bradley D. Brochu, MD, Ahmed Abdi-Ali, MD, Jeffrey Shaw, MD, F
Christopher P. Lawrance, MD, Matthew C. Henn, MD, Jacob R
Finding the right pathway is the key to success
Adenosine-sensitive atrial tachycardia originating from the anterior mitral annulus  Dai Inagaki, MD, Rintaro Hojo, MD, Seiji Fukamizu, MD, PhD, Harumizu.
A case of successful termination of an atrial tachycardia ablated from the pulmonary artery during rapid ventricular pacing  Toshiya Kurotobi, MD, PhD,
Volume 16, Issue 4, Pages (April 2019)
Pseudo-conduction block at the mitral isthmus in a patient with epicardial impulse propagation through the vein of Marshall  Rintaro Hojo, MD, Seiji Fukamizu,
Yosuke Ishii, MD, PhD, Richard B. Schuessler, PhD, Sydney L
Cardiac arrest in an adolescent with Uhl’s anomaly: Two unusual cardiac arrhythmia mechanisms  Jeffrey P. Moak, MD, Gail D. Pearson, MD, ScD, Bradley.
Single-catheter validation of bidirectional block during atrial flutter ablation  Piotr Futyma, MD, Marian Futyma, MD, PhD, Konrad Dudek, MD, Piotr Kułakowski,
Macroreentrant form of an adenosine 5′-triphosphate–sensitive atrial tachycardia arising from the vicinity of the atrioventricular node involving the.
Volume 92, Issue 3, Pages (September 1987)
Dual intraventricular response after cardiac resynchronization
Anatomically based ablation of atrial flutter in an acute canine model of the modified Fontan operation  Mark D. Rodefeld, MDa, Sanjiv K. Gandhi, MDa,
Bundle branch reentry: A novel mechanism for sustained ventricular tachycardia in Chagas heart disease  Alvaro V. Sarabanda, MD, PhD, Wagner L. Gali,
Mapping of a postinfarction left ventricular aneurysm–dependent macroreentrant ventricular tachycardia  Elad Anter, MD, Jianqing Li, MD, Cory M. Tschabrunn,
Biatrial flutter circuit involving an anomalous insertion of the Bachmann bundle into the superior vena cava  Ely Gracia, MD, Roger Fan, MD, FHRS  HeartRhythm.
Circular mapping recordings in a persistent left superior vena cava during atrial tachycardia: Was isolation achieved?  Sandrine Venier, MD, Jason G.
A case of an incision-related single-loop intra-atrial reentrant tachycardia showing an eccentric atrial activation sequence and widely separate potentials.
Utility of entrainment pacing to clarify the circuit of macroreentrant tachycardia with dual early sites on activation maps  Koichi Nagashima, MD, PhD,
Radiofrequency ablation of ventricular tachycardia originating from a lipomatous hamartoma localized in the right ventricle cavity  Jin Xu, PhD, Yingmin.
Incisional atrial reentrant tachycardia: experimental study on the conduction property through the isthmus  Yosuke Ishii, MD, Takashi Nitta, MD, Shun-ichiro.
Management of Catheter-Related injuries to the coronary sinus
Intraoperative Electroanatomic Mapping
James L. Cox, MD  The Journal of Thoracic and Cardiovascular Surgery 
Intraoperative verification of conduction block in atrial fibrillation surgery  Yosuke Ishii, MD, Takashi Nitta, MD, Masaru Kambe, MD, Jiro Kurita, MD,
Presentation transcript:

Cryoablation of ventricular tachycardia guided by return cycle mapping after entrainment  Takashi Nitta, MD, Masataka Mitsuno, MD, Chris K. Rokkas, MD, Richard Lee, MD, Richard B. Schuessler, PhD, John P. Boineau, MD  The Journal of Thoracic and Cardiovascular Surgery  Volume 121, Issue 2, Pages 249-258 (February 2001) DOI: 10.1067/mtc.2001.111052 Copyright © 2001 American Association for Thoracic Surgery Terms and Conditions

Fig. 1 Difference between the CCP ablation and the EAS ablation. The upper panel illustrates the figure-of-8 reentry model. The CCP is a narrow isthmus between the lines of conduction block that are depicted as bold lines. The EAS locates at the exit from the pathway. The arrows indicate activation wave fronts. Each shaded region indicates the region to be ablated required to terminate reentry. The lower panel shows the electrocardiogram and the electrograms recorded at the CCP and at the EAS during VT in an animal in this study. Note that the electrograms at the CCP are low voltage and polyphasic, whereas the electrograms at the EAS have monophasic potentials with higher voltage and steep deflection. See text for detailed explanation. CCP, Central common pathway; EAS, earliest activation site. The Journal of Thoracic and Cardiovascular Surgery 2001 121, 249-258DOI: (10.1067/mtc.2001.111052) Copyright © 2001 American Association for Thoracic Surgery Terms and Conditions

Fig. 2 Mechanism of return cycle mapping after entrainment. Panel 1 illustrates the figure-of-8 reentry circuit and the activation pattern during entrainment from a region proximal to the CCP. The pacing site is denoted as a rectangle and the lines of block are denoted as bold lines. Arrows indicate the activation sequence. The antidromic activation of the Nth pacing collides with the orthodromic activation of the (N-1)th pacing. The fine lines connected to the lines of block are the lines of collision and the transition from Nth to (N-1)th activation. Panel 2 represents the activation sequence and the return cycle after entrainment at sites A, B, and C in panel 1. Panel 3 illustrates that the return cycle isochrone equal to the VTCL shifts as the stimulation site changes, but always converges on the lines of block irrespective of the stimulation site. Panel 4 shows that the potentials from the CCP are not necessarily required to have the return cycle isochrones converge on the lines of block. See text for detailed explanation. CCP, Central common pathway; VTCL, ventricular tachycardia cycle length; PCL, pacing cycle length. The Journal of Thoracic and Cardiovascular Surgery 2001 121, 249-258DOI: (10.1067/mtc.2001.111052) Copyright © 2001 American Association for Thoracic Surgery Terms and Conditions

Fig. 3 Location of 61 unipolar electrodes over the epicardium. The schema represents the polar view of the ventricles as if they are observed from the apex. Nineteen electrodes are distributed over the right ventricular epicardium, and 31 over the left. Eleven electrodes are located at the interventricular groove along the LAD. AO, Aorta; PA, pulmonary artery; RA, right atrium; LAA, left atrial appendage; IVC, inferior vena cava. The Journal of Thoracic and Cardiovascular Surgery 2001 121, 249-258DOI: (10.1067/mtc.2001.111052) Copyright © 2001 American Association for Thoracic Surgery Terms and Conditions

Fig. 4 An example of return cycle mapping. The upper panel shows the electrocardiogram during cessation of entrainment of a VT induced in a 4-day-old canine infarct. The activation maps during the first and second activation after cessation of entrainment are shown in the middle panels. Boxed areas on the electrocardiogram are the data window analyzed to construct the activation maps. The VT was entrained from the right ventricular outflow tract (denoted as a rectangle ) at a pacing cycle length of 160 ms. After cessation of pacing, the VT resumed at a cycle length of 168 ms with the EAS located at the inferior left ventricle near the apex (denoted as an asterisk ). The activation times are represented as color codes with 20-ms increments. The left lower panel shows the return cycle map constructed from these activation maps, by subtracting the first activation time from the second activation time after cessation pacing. The return cycle was also represented as color codes with 10-ms increments. The return cycle was longest at the region around the pacing site and was shorter at the region near the earliest activation site. The blue indicates the region where the return cycle equaled the pacing cycle length, suggesting orthodromic activation by the preceding stimulus. The red line indicates the return cycle isochrone equal to the VTCL. As we changed the pacing site from the red to the blue rectangle, this isochrone shifted from the red to the blue line as shown in the right lower panel. These lines converged on a region 20 mm away from the earliest activation site and formed intersections. The VT was terminated by a cryothermia to the region between the intersections. ECG, Electrocardiogram; AO, aorta; PA, pulmonary artery; RA, right atrium; LAA, left atrial appendage; IVC, inferior vena cava; VTCL, ventricular tachycardia cycle length. The Journal of Thoracic and Cardiovascular Surgery 2001 121, 249-258DOI: (10.1067/mtc.2001.111052) Copyright © 2001 American Association for Thoracic Surgery Terms and Conditions

Fig. 5 Another example of return cycle mapping. The cycle length of the VT was 153 ms and the earliest activation site was at the anterior left ventricle adjacent to the LAD (denoted as an asterisk ). The VT was entrained at a pacing cycle length of 140 ms. The left panel represents the return cycle map after entrainment from a site at the anterior left ventricle. The site of stimulation is denoted as a rectangle. The return cycle is represented as color codes with 10-ms increments, and the red line indicates the return cycle isochrone equal to the VTCL. The right lower panel shows the return cycle isochrone equal to the VTCL in the return cycle maps after entrainment from two different pacing sites that were denoted as blue and red rectangles. The VT was terminated by a cryothermia applied to the region between the intersections of these isochrones. For abbreviations, see Fig 4. The Journal of Thoracic and Cardiovascular Surgery 2001 121, 249-258DOI: (10.1067/mtc.2001.111052) Copyright © 2001 American Association for Thoracic Surgery Terms and Conditions

Fig. 6 Electrocardiograms and activation maps of the anterior left ventricle during cryoablation guided by return cycle mapping. A total of 253 unipolar electrodes were distributed over the anterior left ventricle. The electrode patch was located from the basal left ventricle 1 to 2 cm away from the left atrioventricular groove to the inferior apex of the left ventricle. The left margin of the electrode array was adjacent to the LAD. The right margin was located at the obtuse margin. Boxed areas on the electrocardiogram are the data window analyzed to construct the activation maps. Panel 1 shows the baseline activation map during VT before the application of cryothermia. The cycle length of the VT was 153 ms. Cryothermia was applied at the region localized by return cycle mapping with 61 electrodes as shown in Fig 5. The mapped region was the entrance of the CCP between the lines of conduction block. Panels 2 and 3 show the activation maps 5 and 10 seconds after the start of cryothermia, respectively. As the diameter of the cryolesion became larger, the cycle length of the VT prolonged by degrees. Panel 4 shows the activation map during the last VT cycle before its termination after 20 seconds of cryothermia. The cryolesion connected the lines of block, blocked the rotating wave front, and resulted in termination of the VT. For abbreviations, see Fig 4. The Journal of Thoracic and Cardiovascular Surgery 2001 121, 249-258DOI: (10.1067/mtc.2001.111052) Copyright © 2001 American Association for Thoracic Surgery Terms and Conditions

Fig. 7 Distance between the earliest activation site and the sites where cryothermia successfully terminated the VT guided by return cycle mapping. The distance was plotted as a function of the cycle length of the VT (VTCL). The Journal of Thoracic and Cardiovascular Surgery 2001 121, 249-258DOI: (10.1067/mtc.2001.111052) Copyright © 2001 American Association for Thoracic Surgery Terms and Conditions