Pathogenesis and Management of Acute Heart Failure and Cardiogenic Shock: Role of Inotropic Therapy A. Iain McGhie, M.D., Richard A. Goldstein, M.D. CHEST Volume 102, Issue 5, Pages 626S-632S (November 1992) DOI: 10.1378/chest.102.5_Supplement_2.626S Copyright © 1992 The American College of Chest Physicians Terms and Conditions
Figure 1 Effect of compliance on the left ventricular (LV) pressure and volume curves. Point A represents a patient with a pressure-volume curve and normal ventricular compliance; B represents a patient with an identical ventricular volume that is associated with higher ventricular pressure because of reduced compliance. CHEST 1992 102, 626S-632SDOI: (10.1378/chest.102.5_Supplement_2.626S) Copyright © 1992 The American College of Chest Physicians Terms and Conditions
Figure 2 Effect of a sudden elevation in left ventricular (LV) filling pressure on the water content of the lung. CHEST 1992 102, 626S-632SDOI: (10.1378/chest.102.5_Supplement_2.626S) Copyright © 1992 The American College of Chest Physicians Terms and Conditions
Figure 3 Effect of different left ventricular filling pressures (LVFP) on the relationship between coronary flow and diastolic arterial pressure; see text for further details. CHEST 1992 102, 626S-632SDOI: (10.1378/chest.102.5_Supplement_2.626S) Copyright © 1992 The American College of Chest Physicians Terms and Conditions
Figure 4 Delayed clearance of water from lungs resulting in the “phase lag” between the left ventricular filling pressure and clinical or radiologic findings; see text for further details. CHEST 1992 102, 626S-632SDOI: (10.1378/chest.102.5_Supplement_2.626S) Copyright © 1992 The American College of Chest Physicians Terms and Conditions