Volume 97, Issue 2, Pages (February 1990)

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Volume 97, Issue 2, Pages 487-489 (February 1990) Fatal Pulmonary Edema due to Nitric Acid Fume Inhalation in Three Pulp-Mill Workers Rajendra Hajela, M.B., David T. Janigan, M.D., F.C.C.P., Paul L. Landrigan, M.D., F.C.C.P., Stephen F. Boudreau, M.D., Shanti Sebastian, M.B. CHEST Volume 97, Issue 2, Pages 487-489 (February 1990) DOI: 10.1378/chest.97.2.487 Copyright © 1990 The American College of Chest Physicians Terms and Conditions

Figure 1 Lung alveoli. Dark areas represent alveolar filling by proteinaceous edema fluid. This change is prominent around bronchioles (arrows) (hematoxylin-eosin, original magnification ×115). CHEST 1990 97, 487-489DOI: (10.1378/chest.97.2.487) Copyright © 1990 The American College of Chest Physicians Terms and Conditions

Figure 2 Lung alveoli after immunohistochemical staining for IgM with goat anti-human IgM antibody. The dark areas are deposits of IgM seen as linear “hyaline membranes” on alveolar epithelial surfaces and, in foci, within the interstitium (arrow) (original magnification × 2,500). CHEST 1990 97, 487-489DOI: (10.1378/chest.97.2.487) Copyright © 1990 The American College of Chest Physicians Terms and Conditions

Figure 3 Electron micrograph of part of an alveolar capillary with its lumen filled by a degranulating necrotic neutrophil with a pyknotic nucleus (N). It is in contact with a necrotic endothelial cell (E). The alveolar surface of the wall is covered by poorly preserved cytoplasm of a type 1 epithelial cell (arrow) (original magnification × 15,000). CHEST 1990 97, 487-489DOI: (10.1378/chest.97.2.487) Copyright © 1990 The American College of Chest Physicians Terms and Conditions