EKG REVIEW Dr. Srikanth Seethala MD,MPH. RBBB: 1.QRS duration more than 120 msec 2.rsr′, rsR′, or rSR′ in leads V1 or V2. The R′ or r′ deflection.

Slides:



Advertisements
Similar presentations
Texas Tech University Health Sciences Center
Advertisements

APPROACH TO WIDE QRS COMPLEX TACHYCARDIA
Differential Diagnosis of Wide QRS Complex Tachycardia
Differential Diagnosis of Tachycardias
Differential diagnosis of broad complex tachycardia
ECG TRAINING MODULE 4 BY BRAD CHAPMAN RCT.
By Dr.Ahmed Mostafa Assist. Prof. of anesthesia & I.C.U.
UNC Emergency Medicine Medical Student Lecture Series
EKG 101 Deborah Goldstein Georgetown University
Chapter Page, 12-Lead ECG for Acute and Critical Care Providers © 2006 by Pearson Education, Inc. Upper Saddle River, NJ 6 Wide Complex Tachycardia.
DIFFERENTIAL DIAGNOSIS OF WIDE COMPLEX TACHYCARDIA
Practice ECGs Part I Copyright © 2006 by Mosby Inc. All rights reserved.
“ Heart Blocks”.
ECG Interpretation Chapter 22.
ECG Interpretation Criteria Review
Wolff-Parkinson-White and Atrioventricular (AV) Heart Blocks
Heart Arrhythmia's Brandy Parker Brianne Negen Jeremy Grimm
Presentation Information
Dysrhythmia examples for residents Elias B Hanna, LSU New Orleans, Cardiology.
ECG Lecture Part 1 ECG Lecture Part 1 ECG Interpretation Selim Krim, MD Assistant Professor Texas Tech University Health Sciences Center.
Normal ECG: Rate and Rhythm
WIDE COMPLEX TACHYCARDIA
Fast & Easy ECGs, 2nd E – A Self-Paced Learning Program
Cardiovascular System Block Cardiac Arrhythmias (Physiology)
SHIVDA PANDEY, PGY-6 MARK VILLALON, PGY-6 BOSTON MEDICAL CENTER CARDIOVASCULAR FELLOWS ECG Master Session SENIOR RESIDENT EDITION.
For Dummies (ie: adult emerg guys like us)
Electrocardiogram Primer (EKG-ECG)
Normal Sinus Ryhthm Sinus Node Arrhythmias Atrial Arrhythmias
Liaoning Medical University Affiliated First Hospital
Fast & Easy ECGs, 2nd E – A Self-Paced Learning Program
LU6 Enhancement Lectures PGH IM Residents 2011
EKG Interpretation.
F. Propagation of cardiac impulse The Normal Conduction System.
ECG Tutorial: Rhythm Recognition Review – the systematic approach Rhythm – the hardest part! –Again – be systematic –Mind your p ’ s & q ’ s & follow the.
Garcia, Cholson Banjo E..  Conduction disturbance  Originate from: ◦ sinus node ◦ AV node ◦ bundle branch.
1 Case 7 Bradycardia © 2001 American Heart Association.
INTRAVENTRICULAR CONDUCTION DISTURBANCES AHA/ACCF/HRS RECOMMENDATIONS FOR THE STANDARDIZATION AND INTERPRETATION OF IVCD JACC 2009 VOL 53.
ECGs AFMAMS Resident Orientation March Lecture Outline ECG Basics Importance of systematically reading ECGs Rate Rhythm Axis Hypertrophy Intervals.
EKG Conduction Abnormalities Part II Sandra Rodriguez, M.D. January 15, 2008.
Adel Hasanin, MRCP (UK), MS (Cardiology)
“Advanced” EKG Reading Stefan Da Silva With special guest…. Dr. S. Weeks.
Gail Walraven, Basic Arrhythmias, Sixth Edition ©2006 by Pearson Education, Inc., Upper Saddle River, NJ Chapter 7 Heart Blocks.
ECG Part II. Rate-measure of frequency of occurrence of cardiac cycles(b/m) < 60 beats/min is a bradycardia beats/min is normal >100 beats/min.
The Normal EKG Eric J Milie D.O.. Sinus Rhythm P wave before every QRS complex P waves upright in II, negative in aVr Reproducibility of the R-R interval.
Introduction to EKG And then a little more. To get an accurate EKG, leads must be properly applied: I: RA(-) to LA(+) II RA(-) to LL(+) III:LA(-) to LL(+)
Q I A 6 Fast & Easy ECGs – A Self-Paced Learning Program QRS Complexes.
1 Nora Goldschlager, M.D. Cardiology – San Francisco General Hospital UCSF Disclosures: None ECG MIMICS OF MYOCARDIAL ISCHEMIA AND INFARCTION.
Aims The ECG complex Step by step interpretation Rhythm disturbances Axis QRS abnormalities Acute and chronic ischaemia Miscellaneous ECG abnormalities.
Electrocardiography – Abnormalities (Arrhythmias) 7
The normal ECG. Normal sinus rhythm –Each p wave followed by a QRS –Normal P waves –P wave rate bpm.
Introduction to Cardiac Arrythmias Arrythmia is a generalized term used to denote disturbances in the heart's rhythm. Normal sinus rhythm is characterized.
Putting It All Together
Wave, IntervalDuration (msec) P wave duration
Bundle Branch Blocks and Chamber Enlargement All EKGs in this presentation have been borrowed from: The Alan E. Lindsay ECG Learning Center ;
Fast & Easy ECGs – A Self-Paced Learning Program
EKG Rounds Rebecca Burton-MacLeod R4, Emerg Med July 20 th, 2006.
UCI Internal Medicine Mini-Lecture
EKG’s By: Robby Zehrung. Leads  In a 3-lead View there are two types of Leads:  Bipolar  Lead I: Right Arm to Left Arm  Lead II: Right Arm to Left.
Heart Blocks Leaugeay Webre BS, CCEMT-P, NREMT-P.
Dr Samira Arami General Cardiologist Conductive system.
Department of Medicine
Differential Diagnosis of Wide QRS Complex Tachycardia
ECG Conduction Abnormalities
ECG Strips of Cardiac Rhythms EYAS ALMOUSA,MD,FACC
Scott E. Ewing DO Lecture #9
What is the QRS axis? Is it normal or abnormal?
EKGs…The Basics for FP Residents
Dr Sing Khien Tiong GPST1
ECG Dr. Sara Al Abdulhadi.
Basic Rhythm Recognition
Presentation transcript:

EKG REVIEW Dr. Srikanth Seethala MD,MPH

RBBB: 1.QRS duration more than 120 msec 2.rsr′, rsR′, or rSR′ in leads V1 or V2. The R′ or r′ deflection is usually wider than the initial R wave. 3.S wave of greater duration than R wave or greater than 40 ms in leads I and V6 in adults. 4.Normal R peak time in leads V5 and V6 but greater than 50 ms in lead V1.

LBBB 1.QRS duration more than 120 msec 2.Broad notched or slurred R wave in leads I, aVL, V5, and V6 and an occasional RS pattern in V5 and V6 attributed to displaced transition of QRS complex. 3. Absent q waves in leads I, V5, and V6

1.ST elevation > 1mm in leads with a positive QRS complex in any lead(concordance in ST deviation) (score 5) 2.ST depression > 1 mm in V1-V3 (concordance in ST deviation) (score 3) 3.ST elevation > 5 mm in leads with a negative QRS complex (score 2) 4.A score of more than 3 has a specificity more than 90% 5.Can we apply for the pacing? Sgarbossa criteria

ST segment elevation ≥5 mm in leads with a negative QRS complex Two other criteria are 1.ST elevation ≥1 mm in leads with concordant QRS polarity 2.ST depression ≥1 mm in leads V1, V2, or, V3 GUSTO 1 trial

Other than unable to use V1-3 we can use rest of the leads RBBB and MI

Arrhythmias

Sinus rhythm

Sinus bradycardia with junctional escape

Sinus rhythm with a PAC

Sinus rhythm with 1st degree AVB

Mobitz type 1, Wenckebach : 1.Progressive PR interval prolongation for several beats preceding the non conducted P wave 2.PR interval after the dropped beat is always shorter than that before the non conducted P wave 3.Percentage of PR increase decrease 4.Progressive Shortening of R-R interval 5.R-R interval encompassing the non conducted P wave is less than twice the preceding R-R interval

Mobitz type 2 How to differentiate between Mobitz type 1 and 2 if it is 2:1 conduction 1.Short PR and Wide QRS- type 2 2.Atropine administration: Mobitz type 1 will be 1:1 conduction 3.Carotid sinus massage or adenosine administration: Mobitz type 1 will be 3:2 and paradoxical improvement in type 2

Complete heart block ( AV dissociation)

AV dissociation by default AV dissociation by usurpation Complete Heart block AV dissociation

High degree AV block, 3:1 conduction

Wandering pacemaker

Sinus rhythm with sinus arrest (junctional escapes)

Sinoatrial exit block

Wide complex tachycardia: A rhythm greater than 100/minute and has QRS duration more than 120 msec Ventricular Tachycardia: A WCT originating below the level of bundle his SVT: Tachycardia originating above the level of bundle of his Wide complex tachycardia

Differential Diagnosis 1.Ventricular tachycardia 2.SVT with aberrant conduction Wide complex tachycardia

1. Concordant Pattern 2. Concordance of the limb leads 3. Presence of Q waves 4. AV dissociation (AVD) 5. Fusion beats V tachycardia Vs SVT with aberrancy

6. The “precordial RS absent” criteria 7. V i /V t ratio 8. QRS during VT narrower than in baseline rhythm 9. Contralateral bundle branch block in baseline rhythm and WCT 10. QRS alternans 11. Presence of multiple WCT configurations

LBBB Ventricular Tachycardia SVT with Aberrant conduction V1 Initial R wave > msec Notching of the S wave (Josephson’s sign) R to S wave > msec V6 QS wave qR pattern No Q waves ( there could be minimal Q wave, but should not be pathological) LBBB pattern

RBBB Ventricular Tachycardia SVT with Aberrant conduction V1-2 Smooth monophasic R wave Notched downslope to the R wave — the taller left rabbit ear (= Marriott’s sign) A qR complex (small Q wave, tall R wave) in V1 RSR’ pattern V6 QS complex – a completely negative complex with no R wave (= strongly suggestive of VT) R/S ratio < 1 RBBB

Thank you Sri