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DERMATITIS AND ECZEMATOUS DISORDERS Dr. Abdulmajeed Alajlan Associate Professor Consultant Dermatologist & Laser surgeon Department of Dermatology- KSU.

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Presentation on theme: "DERMATITIS AND ECZEMATOUS DISORDERS Dr. Abdulmajeed Alajlan Associate Professor Consultant Dermatologist & Laser surgeon Department of Dermatology- KSU."— Presentation transcript:

1 DERMATITIS AND ECZEMATOUS DISORDERS Dr. Abdulmajeed Alajlan Associate Professor Consultant Dermatologist & Laser surgeon Department of Dermatology- KSU

2 Dermatitis Objectives  To know the definition & classification of Dermatitis/Eczema  To recognize the primary presentation of different types of eczema  To understand the possible pathogenesis of each type of eczema  To know the scheme of managements lines

3 Eczema  Definition: inflammation of the skin  Ezema vs. dermatitis

4  Classification: Types of classification Benefit of classification Eczema

5  Acute eczema: erosion, oozing and vesicles Eczema

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8  Chronic eczema: lichenification, drak pigmentation and thick papules and plaques Eczema

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11 Contact Dermatitis

12  Definition: dermatitis results from contact with external materials Contact Dermatitis

13  Pathogenesis:  Irritant vs. allergic  Common irritants: detergent, acids, dust, burning chemicals, etc  Common allergens: perfumes, hair dyes, nickels, leathers, metals, rubbers, latex, cosmetics, etc Contact Dermatitis

14  Clinical features  Prediliction sites:  Distribution & configuration Contact Dermatitis

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18  Management:  Identification  Patch testing: for allergic contact dermatitis not for irritant  Avoidance measures  Topical corticosteroids Contact Dermatitis

19 Atopic Dermatitis

20 Atopic Dermatits  Definition: chronic relapsing itchy skin disease in genetically predisposed patients. Associated diseases: bronchial asthma, allergic rhinitis, allergic congectivitis  Incidence: up to 15% in developed countries  Grow out tendency!

21  Pathogenesis: “Atopy”: genetic predisposition Dry skin (decrease production of moisturizing lipids; sebum) IgE ? (Epiphenomenon) T-Cell Allergy, increased tendency to certain allergens Atopic Dermatitis

22  Clinical Variants: Infantile AD Childhood AD Adult AD Atopic Dermatitis

23  Infantile AD: Distribution Presentation Behaviour Atopic Dermatits

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26  Childhood AD: Distribution Presentation Behaviour Atopic Dermatits

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30  Adult AD: Distribution Presentation Behaviour Atopic Dermatits

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33 Erythroderma: is a very rare complication of atopic dermatitis

34  Complications:  Secondary infections  Eczema herpeticum  Growth retardation  psychological Atopic Dermatits

35 Cellulitis

36 Impitigo: Bacterial infection

37 Eczema Herpiticum is a serious complicaiton that needs admission and systemic antiviral

38  Investigations: ???????? Atopic Dermatits

39  Management:  Education! Education! Education!  Support!  Skin care: moisturizing the skin  Topical therapy: (topical steroids, Tacrolimus, Pimecrolimus)  Phototherapy  Systemic therapy: steroids, Cyclosporin, Methotrexate, Azathioprine

40  AD and Food! minor role Atopic Dermatits

41 Seborrhoeic Dermatitis

42  Definition: Seborrhoeic Dermatits

43  Pathogenesis: increased Sebum! Tendency Pityrosporum ovale over growth Seborrhoeic Dermatits

44  Clinical features: Presentation Distribution Seborrhoeic Dermatits

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47  Investigations : ?????  Resistant cases think of: Histocytosis (langerhans cell neoplasm) Seborrhoeic Dermatits

48  Management:  Shampoo  Antigungals  Topicals  Combined therapy  Maintenance & recurrence Seborrhoeic Dermatits

49 Dermatitis Objectives  To know the definition & classification of Dermatitis/Eczema  To recognize the primary presentation of different types of eczema  To understand the possible pathogenesis of each type of eczema  To know the scheme of managements lines


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