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Pulse. The pulse has been studied for centuries. Information gained: Information gained: Frequency, regularity Frequency, regularity Patency of peripheral.

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Presentation on theme: "Pulse. The pulse has been studied for centuries. Information gained: Information gained: Frequency, regularity Frequency, regularity Patency of peripheral."— Presentation transcript:

1 Pulse

2 The pulse has been studied for centuries. Information gained: Information gained: Frequency, regularity Frequency, regularity Patency of peripheral arteries Patency of peripheral arteries Characteristics of the arterial pressure pulse wave Characteristics of the arterial pressure pulse wave The arterial pulse contour changes to the periphery: The arterial pulse contour changes to the periphery: Resistance: viscosity, vessel geometry opposes flow; HR independent Resistance: viscosity, vessel geometry opposes flow; HR independent Inertia: mass opposes rate of change of flow, HR dependent Inertia: mass opposes rate of change of flow, HR dependent Compliance: distensibility opposes changes of blood volume, HR-dependent Compliance: distensibility opposes changes of blood volume, HR-dependent

3 Location of Pulses Carotid – similar to central aortic pulse (delay of 20msec) Carotid – similar to central aortic pulse (delay of 20msec) Radial – used to assess the volume and consistency of the peripheral vessels. Radial – used to assess the volume and consistency of the peripheral vessels. Brachial Brachial Femoral Femoral Posterior tibial Posterior tibial Dorsalis pedis Dorsalis pedis Look for any differences in the pulse amplitude, contour, or upstroke. Look for any differences in the pulse amplitude, contour, or upstroke.

4 Factors Influencing the Pulse Stroke volume Stroke volume Rate of ejection Rate of ejection Distensibility of peripheral arteries Distensibility of peripheral arteries Peripheral resistance Peripheral resistance Pulse rate Pulse rate Pulse pressure Pulse pressure Size of the vessel Size of the vessel Distance from the heart Distance from the heart

5 Normal Pulse (1/2) The normal central aortic pulse wave is characterized by a fairly rapid rise to a somewhat rounded peak. The normal central aortic pulse wave is characterized by a fairly rapid rise to a somewhat rounded peak. The anacrotic shoulder, present on the ascending limb, occurs at the time of peak rate of aortic flow just before maximum pressure is reached. The anacrotic shoulder, present on the ascending limb, occurs at the time of peak rate of aortic flow just before maximum pressure is reached. The less steep descending limb is interrupted by a sharp downward deflection, coincident with AV closure, called incisura. The less steep descending limb is interrupted by a sharp downward deflection, coincident with AV closure, called incisura. The pulse pressure is about mmHg. The pulse pressure is about mmHg.

6 Normal Pulse (2/2) As the pulse wave is transmitted peripherally, the initial upstrokes becomes steeper, the anacrotic shoulder becomes less apparent, and the incisura is replaced by the smoother dicrotic notch. As the pulse wave is transmitted peripherally, the initial upstrokes becomes steeper, the anacrotic shoulder becomes less apparent, and the incisura is replaced by the smoother dicrotic notch.

7 Abnormal Pulses Inequality in the amplitude of the peripheral pulses: Inequality in the amplitude of the peripheral pulses: Obstructive arterial diseases, most commonly artherosclerosis Obstructive arterial diseases, most commonly artherosclerosis Aortic dissection Aortic dissection Aortic aneurysm Aortic aneurysm Takayasu arteritis (pulseless disease) Takayasu arteritis (pulseless disease) Supravalvular aortic stenosis (fixed form of LVOT caused by narrowing of ascending aorta) Supravalvular aortic stenosis (fixed form of LVOT caused by narrowing of ascending aorta)

8 Pulsus Parvus The pressure is diminished, and the pulse feels weak and small, reflecting decreased stroke volume (e.g. heart failure), restrictive pericardial disease, hypovolemia, mitral stenosis, and increased peripheral resistance (e.g. exposure to cold, severe CHF). The pressure is diminished, and the pulse feels weak and small, reflecting decreased stroke volume (e.g. heart failure), restrictive pericardial disease, hypovolemia, mitral stenosis, and increased peripheral resistance (e.g. exposure to cold, severe CHF). Pulsus Parvus et Tardus (weak and delayed): →Aortic Stenosis Pulsus Parvus et Tardus (weak and delayed): →Aortic Stenosis

9 Aortic Stenosis (1/2) LVOT obstruction LVOT obstruction Reduces SV, prolongs LV total ejection time, and retards the rate of initial stroke output into the aorta and distal arterial system. Reduces SV, prolongs LV total ejection time, and retards the rate of initial stroke output into the aorta and distal arterial system. Anacrotic character (anacrotic pulse) Anacrotic character (anacrotic pulse) Interruption of the upstroke of the carotid pulse. AS is likely to be hemodynamically significant when the anacrotic notch is felt immediately after the onset of the upstroke. Interruption of the upstroke of the carotid pulse. AS is likely to be hemodynamically significant when the anacrotic notch is felt immediately after the onset of the upstroke.

10 Aortic Stenosis (2/2) Delayed upstroke of the ascending limb (pulsus tardus) Delayed upstroke of the ascending limb (pulsus tardus) Prolonged LV ejection time. Prolonged LV ejection time. Appreciated by simultaneous palpation of carotid pulse and auscultation fo the interval between S1 and S2. Appreciated by simultaneous palpation of carotid pulse and auscultation fo the interval between S1 and S2. Normally the carotid pulse occurs closer to S1; in severe AS closer to S2. Normally the carotid pulse occurs closer to S1; in severe AS closer to S2. Thrill (carotid shudder) Thrill (carotid shudder)

11 Bisferiens Pulse Increased arterial pulse with a double systolic peak. Increased arterial pulse with a double systolic peak. Second peak, the tidal wave, presumed to represent a reflected wave from the periphery. Second peak, the tidal wave, presumed to represent a reflected wave from the periphery. Causes: hemodynamically significant AR Causes: hemodynamically significant AR HCM: rarely palpable; rapid LV ejection during early systole, rapid decline due to LVOT obstruction, and tidal wave from periphery. HCM: rarely palpable; rapid LV ejection during early systole, rapid decline due to LVOT obstruction, and tidal wave from periphery.

12 Bigeminal Pulse Normal beat alternating with a premature contraction. Normal beat alternating with a premature contraction. SV of the premature beat diminished, and pulse varies in amplitude accordingly. SV of the premature beat diminished, and pulse varies in amplitude accordingly. May masquerade as pulsus alternans May masquerade as pulsus alternans Causes: decreased BP (e.g. severe HF, hypovolemic shock, cardiac tamponade) and peripheral resistance (e.g. fever), s/p aortic valve replacement. Causes: decreased BP (e.g. severe HF, hypovolemic shock, cardiac tamponade) and peripheral resistance (e.g. fever), s/p aortic valve replacement. Present in normal individuals after exercise. Present in normal individuals after exercise.

13 Pulsus Alternans (1/2) Variation in pulse amplitude occurring with alternate beats due to changing systolic pressure. Variation in pulse amplitude occurring with alternate beats due to changing systolic pressure. When the cuff pressure is slowly released while taking BP, phase I Korotkoff sounds are initially heard only during the alternate strong beats; with further release of cuff pressure, the softer sounds of the weak beat also appear. When the cuff pressure is slowly released while taking BP, phase I Korotkoff sounds are initially heard only during the alternate strong beats; with further release of cuff pressure, the softer sounds of the weak beat also appear. Degree of pulsus alternans can be quantitated by measuring the pressure difference between the strong and the weak beat. Degree of pulsus alternans can be quantitated by measuring the pressure difference between the strong and the weak beat.

14 Pulsus Alternans (2/2) Causes: Causes: Left ventricular failure – usually accompanied by a left- sided S 3. Left ventricular failure – usually accompanied by a left- sided S 3. May be seen in patients with severe AR May be seen in patients with severe AR Frequently precipitated by ectopic beats (bigeminal pulse) Frequently precipitated by ectopic beats (bigeminal pulse) Mechanisms: Mechanisms: Alternating preload and afterload, incomplete relaxation, Alternating preload and afterload, incomplete relaxation, Change in ventricular contractility, causing changes in end- diastolic volume and pressure. Change in ventricular contractility, causing changes in end- diastolic volume and pressure.

15 Pulsus Paradoxus (1/2) Pressure drop > 20 mmHg during inspiration. Pressure drop > 20 mmHg during inspiration. Normally, systolic arterial pressure falls 8-12 mmHg during inspiration. Normally, systolic arterial pressure falls 8-12 mmHg during inspiration. Evaluated with sphygmomanometer: Evaluated with sphygmomanometer: when the cuff is slowly released the systolic pressure at expiration is first noted. With further slow deflation of the cuff, the systolic pressure during inspiration can also be detected. when the cuff is slowly released the systolic pressure at expiration is first noted. With further slow deflation of the cuff, the systolic pressure during inspiration can also be detected.

16 Pulsus Paradoxus (2/2) Causes: Causes: Cardiac Tamponade Cardiac Tamponade COPD, hypervolemic shock COPD, hypervolemic shock infrequently in constrictive pericarditis and rescrictive cardiomyopathy. infrequently in constrictive pericarditis and rescrictive cardiomyopathy. Mechanism: Mechanism: Decreased LV-SV due to an increased RV-EDV and decreased LV-EDV during inspiration. Decreased LV-SV due to an increased RV-EDV and decreased LV-EDV during inspiration. In cardiac tamponade, the interventricular septum shifts toward the LV cavity during inspiration (reverse Bernheim’s effect) b/c of increased venous return to RV, decreasing the LV preload. In cardiac tamponade, the interventricular septum shifts toward the LV cavity during inspiration (reverse Bernheim’s effect) b/c of increased venous return to RV, decreasing the LV preload. Decrease in pulmonary venous return to the LV during inspiration also contributes to decreased LV preload. Decrease in pulmonary venous return to the LV during inspiration also contributes to decreased LV preload.

17 Bounding Pulses (1/2) A.K.A. water-hammer pulse or the Corrigan pulse. A.K.A. water-hammer pulse or the Corrigan pulse. Most commonly in chronic, hemodynamically significant AR. Most commonly in chronic, hemodynamically significant AR. Seen in many conditions associated with increased stroke volume: PDA, large arteriovenous fistula, hyperkinetic states, thyrotoxicosis anemia, and extreme bradycardia. Seen in many conditions associated with increased stroke volume: PDA, large arteriovenous fistula, hyperkinetic states, thyrotoxicosis anemia, and extreme bradycardia. Not seen in acute AR, since SV may not have increased appreciably. Not seen in acute AR, since SV may not have increased appreciably.

18 Bounding Pulses (2/2) Physical signs of aortic insufficiency are related to the high pulse pressure and the rapid decrease in blood pressure during diastole due to the AI: Physical signs of aortic insufficiency are related to the high pulse pressure and the rapid decrease in blood pressure during diastole due to the AI: Lighthouse sign (blanching & flushing of forehead) Lighthouse sign (blanching & flushing of forehead) de Musset's sign (head nodding in time with the heart beat) de Musset's sign (head nodding in time with the heart beat) Ladolfi's sign (alternating constriction & dilatation of pupil) Ladolfi's sign (alternating constriction & dilatation of pupil) Becker's sign (pulsations of retinal vessels) Becker's sign (pulsations of retinal vessels) Müller's sign (pulsations of uvula) Müller's sign (pulsations of uvula) Corrigan's pulse (rapid upstroke and collapse of the carotid artery pulse) Corrigan's pulse (rapid upstroke and collapse of the carotid artery pulse) (Watson's) Water-hammer pulse (Watson's) Water-hammer pulse Quincke's sign (pulsation of the capillary bed in the nail) Quincke's sign (pulsation of the capillary bed in the nail) Mayen's sign (diastolic drop of BP>15 mm Hg with arm raised) Mayen's sign (diastolic drop of BP>15 mm Hg with arm raised) Rosenbach's sign (pulsatile liver) Rosenbach's sign (pulsatile liver) Gerhardt's sign (enlarged spleen) Gerhardt's sign (enlarged spleen) Duroziez's sign (systolic and diastolic murmurs heard over the femoral artery when it is gradually compressed) Duroziez's sign (systolic and diastolic murmurs heard over the femoral artery when it is gradually compressed) Hill's sign (A ≥ 20 mmHg difference in popliteal and brachial systolic cuff pressures, seen in chronic severe AI) Hill's sign (A ≥ 20 mmHg difference in popliteal and brachial systolic cuff pressures, seen in chronic severe AI) Traube's sign (a double sound heard over the femoral artery when it is compressed distally) Traube's sign (a double sound heard over the femoral artery when it is compressed distally) Lincoln sign (pulsatile popliteal) Lincoln sign (pulsatile popliteal) Sherman sign (dorsalis pedis pulse is quickly located & unexpectedly prominent in age>75 yr) Sherman sign (dorsalis pedis pulse is quickly located & unexpectedly prominent in age>75 yr)


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