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Electrolyte abnormalities and ECG Elias Hanna, MD LSU Cardiology.

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Presentation on theme: "Electrolyte abnormalities and ECG Elias Hanna, MD LSU Cardiology."— Presentation transcript:

1 Electrolyte abnormalities and ECG Elias Hanna, MD LSU Cardiology

2 Hyperkalemia: T wave in hyperkalemia is typically tall and narrow, but does not have to be tall (may be just narrow and peaked pulling ST segment). Tall T means > 2 big boxes in the precordial leads or >1 small box in limb leads, or T wave taller than QRS.

3 Hypokalemia: Flat T with K~3 ST depression with prominent T (actually U) and prolonged QT when K<2.5-3

4 Hypokalemia: -T progressively flattens -U wave more and more prominent (looks like T) -ST-segment more and more depressed Large U wave simulates and hides T wave  with severe hypokalemia, the ST-T pattern may mimick: ST-segment depression with a flat or upright wide “T” wave (actually U) and a prolonged “QT” interval (actually QTU)

5 On the other hand, the pattern of T inversion is not seen with hypokalemia:

6 ECG changes of digoxin effect (digoxin therapy) simulate the changes seen with hypokalemia (U wave and ST depression), except that with digoxin therapy QT is not prolonged

7 Hypocalcemia: Long QT that is due to a long ST segment, which is different from long QT due to congenital long QT syndrome, drugs, or hypokalemia. T wave is not wide, there is no T wave abnormality.

8 Hypercalcemia: short QTc <390 ms. No significant ST or T wave abnormality

9 Hypomagnesemia is not associated with characteristic or specific ECG findings It is associated with a non-specific prolongation of QT and/or QRS intervals, and is often associated with hypokalemia and hypocalcemia. Therefore, changes related to the latter 2 abnormalities may be seen.


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