We think you have liked this presentation. If you wish to download it, please recommend it to your friends in any social system. Share buttons are a little bit lower. Thank you!
Presentation is loading. Please wait.
supports HTML5 video
Published byTaryn Spears
Modified over 4 years ago
Fluid and Hemodynamic Disorders
Where’s my water? Intracellular IonsIon specific gates in cell membrane Cellular proteins Extracellular Interstitial (between the cells) Lymph Intravascular Blood Lymphatic fluid
Movement of water in the vascular systemHydrostatic, the pumping pressure Heart Skeletal muscle action Oncotic or osmotic, holds fluid in. Proteins such as albumin Cellular elements such as RBCs
Intracellular & Extracellular Water
Things can go wrong Heart failure Kidney failure Myocardial infarctionPulmonary emobolus Tissue congestion Edema
Edema Too much extracellular fluid. Swelling Localized or Generalizedtumor Localized or Generalized Dependent action of gravity
Tansudate or Exudate? Exudate Inflammatory waterPart of the inflammatory reaction Rubor, dolor, calor, tumor Purposeful and intentional Localized
Tansudate or Exudate? Transudate Leakage, not part of healingIncreased hydrostatic pressure Heart failure Lymphatic obstruction Decreased oncotic pressure Decreased albumin
Congestive Heart Failure
Congestive Heart Failure
Chronic Passive Congestion, Nutmeg Liver
Chronic Passive Congestion
Water in Hollow Spaces Hydrothorax Hydropericardium HydroperitoneumAscites
Healthy Blood ClottingPlatelets Vessels Clotting Proteins
Thrombosis A pathological clotA clot forming in the fixed vascular system.
Thrombosis Endothelial damage Stasis and clotting factor activationClotting factor abnormalities Too many clotting proteins Pregnancy Cancers Too little inhibition Abnormal factors Leiden Factor (abnormal V)
Thrombosis Arterial Side Thrombi Venous Side ThrombiPlatelet activation Endothelial cell injury Venous Side Thrombi Stasis Clotting factor activation
Coronary Artery ThombosisAngiogram
Acute Myocardial Infarction
Aneurysm with Thrombus
Deep Leg Vein Thrombosis
Airplane Travel Gunner turret
Outcomes of a DVT
Embolus Space occupying mass moving in the fixed vascular systemBlood clot Bone Fragments Amniotic Fluid Air
Infarction Anemic Hemorrhagic End artery supply No blood WhiteVenous occlusion Loose tissues Dual blood supply Red
Shock Poor perfusion Tissue hypoxia Tissue acidosis Many causesPoor pumping by heart Low blood volume Loss of fluid Overwhelming infections
Types of Shock Cardiogenic Hypovolemic Anaphylaxis SepticDecreased output Hypovolemic Blood loss Fluid loss Anaphylaxis IgE and histamine Septic Gram negative rods Toxins
What Happens Next? Compensated Decompensated Irreversible Fluid shiftsProgression possible Irreversible No recovery
The Shock Spiral
Summary Fluid shifts Excessive tissue water Clot formation ThrombosisOncotic & Hydrostatic Pressures Excessive tissue water Exudate vs. Transudate Clot formation Vessels, platelets & proteins Thrombosis Pathological clot Arterial = endothelial damage & platelet activation. Venous = stasis and factor activation
Summary Infarction Ischemic = end artery organHemorrhagic = venous or dual blood supply Tissue vulnerability Brain Kidney muscle
The Physiology of Shock
Tissue Fluid Formation and Oedema
Edema, Hyperemia and Congestion
Hemodynamic Disorders, Thrombosis & Shock
Chapter 3 Disorders of Vascular Flow Yiran Ni M.D
Capillary Circulation & Edema Formation
Hemodynamic Disorders Dr. Raid Jastania. Intended Learning Outcomes 1.Students should be able to define edema, congestion, hemorrhage, thrombosis and.
Hemodynamic Disorders. Fluid Distribution ~60% of lean body weight is water ~2/3 is intracellular ~1/3 is extracellular (mostly interstitial) ~5% of total.
Hemodynamics & Cellular Phases Types of Exudates (Role of Chemical mediators in inflammation)
2nd Year Pathology 2010 Vascular Disturbances I Oedema, Hyperaemia and Haemorrhage.
60% of lean body weight = water (2/3) intracellular. (1/3)extracellular (interstitial fluid) 5% blood plasma. edema = an accumulation of interstitial.
Edema Excess fluid in the tissues Intracellular Edema Extracellular Edema.
Hemodynamic Disorders, Thrombosis, and Shock Dr Hisham Alkhalidi.
Weeks 4 and 5 Disturbances of Blood Flow Dr.İ.Taci Cangül Bursa-2008.
Care of Patients with Shock
Anatomy Overview THE CIRCULATORY AND LYMPHATIC SYSTEMS.
Interstitial Fluid Formation and Oedema By DR QAZI IMTIAZ RASOOL.
Pulmonary Vascular Disease. Pulmonary Circulatuion Dual supply Pulmonary arteries Bronchial arteries Low pressure system Pulmonary artery receives.
© 2019 SlidePlayer.com Inc. All rights reserved.