1. Common variation in the ADAM8 gene affects serum sADAM8 concentrations and the risk of myocardial infarction in two independent cohorts 
Emma Raitoharju, Ilkka Seppälä, Mari Levula, Pekka Kuukasjärvi, Jari Laurikka, Kjell Nikus, Ari-Pekka J. Huovila, Niku Oksala, Norman Klopp, Thomas Illig, Reijo Laaksonen, Pekka J. Karhunen, Jari Viik, Rami Lehtinen, Markku Pelto-Huikko, Matti Tarkka, Mika Kähönen, Terho Lehtimäki 
Atherosclerosis 
Volume 218, Issue 1, Pages (September 2011)
DOI: /j.atherosclerosis
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2. Fig. 1
Distribution of non-normalized soluble ADAM8 in the patients’ serum for the different genotypes of rs (A and B) and rs (C and D) and for the haplotypes created from them (E and F) in ANGES (A, C and E) and FINCAVAS (B, D and F). P-values calculated with the Kruskal-Wallis Test from non-normalized data are shown in the figures. ■ Median 25–75% non-outlier range.
Atherosclerosis  , DOI: ( /j.atherosclerosis )
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3. Fig. 2
ADAM8 protein (as expressed) in (A) the carotid artery of a major allele homozygotic patient (rs A/A and rs G/G); (B) the femoral artery of a major allele homozygotic patient; (C) the carotid artery of a heterozygotic patient (rs A/C and rs G/A); and (D) the femoral artery of a major allele heterozygotic patient. Arrows point to ADAM8 positive cells.
Atherosclerosis  , DOI: ( /j.atherosclerosis )
Copyright © 2011 Elsevier Ireland Ltd Terms and Conditions