1. Volume 118, Issue 6, Pages 1031-1038 (June 2000)
Epstein–Barr virus—associated gastric carcinomas: Relation to H. pylori infection and genetic alterations 
Ming–Shiang Wu*, Chia–Tung Shun‡, Chung–Chun Wu§, Tsuey–Ying Hsu§, Ming–Tsan Lin∥, Ming–Chu Chang*, Hsiu–Po Wang*, Jaw–Town Lin* 
Gastroenterology 
Volume 118, Issue 6, Pages (June 2000)
DOI: /S (00)
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2. Fig. 1
A representative example of microscopic features and in situ hybridization of LELC of the stomach. (A) Pleomorphic cancer cell nests separated by intervening lymphoid stoma (H&E stain; original magnification 200×). (B) Positive EBER-1 signals (black) in the nucleus of glandular and undifferentiated carcinomas (in situ hybridization; original magnification 200×).
Gastroenterology  , DOI: ( /S (00) )
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3. Fig. 2
Survival analysis of 3 different groups estimated by Kaplan–Meier method. Cum. Survival, cumulative survival.
Gastroenterology  , DOI: ( /S (00) )
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4. Fig. 3
(A) A representative example of positive p53 immunostaining in neoplastic cells with distinct nuclear staining (original magnification 100×). (B) Automatic sequencing data of a point mutation (C→T, arrow) of p53 in a case of gastric LELC with p53 overexpression. Upper and middle panels represent forward and reverse sequencing of tumor DNA, respectively, in contrast to nontumorous DNA in lower panel.
Gastroenterology  , DOI: ( /S (00) )
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5. Fig. 4
(A) A representative example of c-erbB-2 overexpression in gastric cancer (original magnification 100×). (B) E-cadherin expression in gastric cancer (original magnification 200×). Both show a predominantly membranous pattern in neoplastic cells.
Gastroenterology  , DOI: ( /S (00) )
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