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Dr. Raj Patel OD - Vancouver Vision Clinic

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Presentation on theme: "Dr. Raj Patel OD - Vancouver Vision Clinic"— Presentation transcript:

1 Dr. Raj Patel OD - Vancouver Vision Clinic
Vision & Perception

2 What we will cover today…
Eye Anatomy Accommodation Rods & Cones Convergence Edge Enhancement (Herman Grid Illusion) Contralateral Processing Cases

3 Photoreceptors: Rods & Cones 160K/mm in humans, 1 Billion/mm in hawk
LE 49-18 Photoreceptors: Rods & Cones 160K/mm in humans, 1 Billion/mm in hawk Sclera Choroid Retina Ciliary body Suspensory ligament Fovea (center of visual field) Cornea Iris Optic nerve Pupil Aqueous humor Lens Central artery and vein of the retina Vitreous humor Optic disk (blind spot)

4 Retinal image (? Eye)

5 Retinal Detachment

6 Cataract

7 Cataract surgery

8 LASIK surgery

9 Accomodation The lens inside the eye changes shape to focus at different distances

10 Near vision (accommodation)
LE 49-19 Front view of lens and ciliary muscle Choroid Lens (rounder) Retina Ciliary muscle Suspensory ligaments Near vision (accommodation) Lens (flatter) Distance vision

11 What happens after 40! Presbyopia

12 How do we see ? Lets go back to the retina …

13 Rods: Cones: 1. More sensitive to light – function better in dim light
2. Become bleached in bright light 3. Absorb all wavelengths of visible light, so they give monochrome vision 4. Groups (up to 200) pass impulses to same sensory neuron of optic nerve, therefore less visual acuity 5. More widely dispersed, giving a wider field of vision LE 49-22 Dark Responses Light Responses Cones: 1. Less sensitive to light 2. Don’t bleach in bright light, function well in bright light 3. 3 types of cone cells: sensitive to red, green & blue light 4. Have their own individual neuron through which messages can be sent to brain, therefore better visual acuity 5. Very concentrated near the fovea, giving one acute area of field of vision Rhodopsin inactive Rhodopsin active Na+ channels open Na+ channels closed Rod depolarized Rod hyperpolarized Glutamate released No glutamate released Bipolar cell either depolarized or hyperpolarized, depending on glutamate receptors Bipolar cell either hyperpolarized or depolarized, depending on glutamate receptors

14 General Organization Retina is only part of CNS visible
3 layers of neurons

15 More Detail of Cell Types
Receptive field, convergence, resolution

16 So how do we process all this?

17 Edge Enhancement

18 background Provides greater contrast
Many rods -> single ganglia = lower acuity Cones have lower ratio with ganglia = higher acuity Receptive fields. If smaller then higher acuity Large fields in peripheral retina / smallest in fovea

19 The concept Lateral Inhibition 2 other cells (horizontal and amacrine)
Below is 1 to 1 mapping (fovea)

20 Drum roll …

21 Herman Grid Illusion Due to edge enhancement white areas of grid look whiter if they are next to black area Optical illusion: grey areas appear at the intersections of the white lines which are not real. If all of grid is covered up apart from one white line, the grey areas disappear.

22 Still Awake

23 Contralateral Processing
Collect - integrate - perceive

24 3. Contralateral processing of Visual Stimuli
LE 49-24 3. Contralateral processing of Visual Stimuli *Both retinas receive stimuli from left & right fields of vision *Left & right optic nerves cross in optic chiasma *Right field of vision is processed in left side of brain (vice versa) *Neurons from optic nerve synapse with neurons in visual cortex of Cerebrum (occipital lobe) *Allows brain to Have perception of depth, distance & sizes Left visual field Right visual field Left eye Right eye Optic nerve Optic chiasm Lateral geniculate nucleus Primary visual cortex

25 An interesting case

26 Where is the problem located?

27 Thank You


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