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ECG PRACTICAL APPROACH Dr. Hossam Hassan Consultant Emergency Medicine
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Objectives To emphasize simplicities Practical approach Interpretation & clinical scenario are inseparable Systematic approach
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Conduction System
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Nomenclature
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Magic numbers of Dr. Hossam
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Systematic approach Rate Rhythm axis P-wave PR interval QRS complex ST segment T-wave
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Rate The interval between 2 successive R-wave How many big squares? Divide 300 / # big squares Normal 60 – 100/min
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Rhythm Sinus Rhythm Every P=wave is followed by QRS complex P-wave is upright in lead II
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NSR
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Types of Sinus Rhythm NSR Sinus Tachycardia Sinus Bradycardia Sinus arrhythmia
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Sinus tachycardia
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Axis Normal axis Right axis deviation Left axis deviation
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RAD
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LAD
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P-wave Atrial depolarization Atrial contraction is a result Normally a dome-like structure
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Abnormalities of P-wave Peaked p-pulmonle –Pulmonary HTN –PE –Pulmonary valve stenosis M-shaped M-mitrale –Mitral valve stenosis –Left atrial hypertrophy Inverted 2 nd atrial / junctional ectopy
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P-pulmonale
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PR interval Definition From the start of P to beginning of QRS Represent the delay in transmission in AV node Normally 0.12 – 0.20 msec
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Abnormalities of PR interval Prolonged > 1 st degree HB Short < Pre-excitation syndromes –WPW Syndrome –LGL Syndrome Junctional rhythm
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QRS Complex Amplitute Duration Shape Q-wave R-wave
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QRS AMPLITUTE LVH By voltage criteria –S-wave in V 1 or V 2 + R-wave in V5 or V6
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LVH & STRAIN PATTERN
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Causes of LVH HTN Aortic stenosis HOCM Aortic regurgitation Mitral regurgitation
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QRS DURATION Ventricular depolarization Ventricular contraction is a result Normally < 0.12 msec < small squares
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Causes of wide QRS Ventricular tachycardia BBB –Left BBB –Right BBB
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L BBB
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R BBB
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Shape Upstroke & downstroke of R-wave Delta wave
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Q-wave 1 st negative deflection after the P-wave Normally 1mm wide & 2 mm deep Lead III, V5 & V6 Pathological Q-wave Wider & deeper >1/4 of the ensuing R-wave Old MI
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+ve R-wave in V1
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Causes +ve R-wave in V I RVH R BBB Posterior MI Type A WPW
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ST-Segment From the end of S-wave to the beginning of T-wave Normally iso-electric Abnormalities –Elevated –depressed
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Elevated ST segment Acute MI Pericarditis Early repolarization pattern in the young
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Infarct localization Inferior –Lead II, III, aVF Septal –V I, V II Anterior –V3, V4
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Lateral –Lead I, AVL,V5, V6 Posterior MI - Prominent R wave in V1,V2 with depressed ST segment
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Acute inf MI
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Anteroseptal MI
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Anterior MI
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Lateral MI
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Depressed ST Segment Unstable angina Left ventricular strain pattern
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LVH & strain pattern
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T-wave Ventricular repolarization Dome like structure Abnormalities –Peaked / tented t-wave Hyperkalaemia Subendocadial ischemia –Inverted LV Strain pattern Dynamic t-wave changes of ischemia
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DYNAMIC T-WAVE CHANGES
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Hay….. wake up we are done
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