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THE ASSESSMENT AND CARE FOR IMPAIRED URINARY ELIMINATION PN 134

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1 THE ASSESSMENT AND CARE FOR IMPAIRED URINARY ELIMINATION PN 134

2 ASSESSMENT Pain on urination Pattern of urination
Strength of urine stream Urgency, frequency, incontinence, hematuria, nocturia Intake and output Urine color, clarity, and odor

3 URINARY RETENTION Urinary retention Etiology/pathophysiology
The inability to void despite an urge to void Clinical manifestations/assessment Distended bladder Discomfort in pelvic region Voiding frequent, small amounts URINARY RETENTION Inability to void when there is an urge to void, acute or chronic This creates urinary stasis & increases the possibility of infection. Urine may overflow the bladder’s capacity, causing incontinence. Possible Causes Response to stress Obstruction of the urethra by calculi (concentration of mineral salts – stones) Tumor Infection Interference with the sphincter muscles during surgery Side effect of medications or perineal trauma Client may experience discomfort & anxiety Frequency and voiding small amounts may occur Distended bladder can be palpated above the symphysis. Can be chronic or acute; if chronic, bladder can overflow causing urinary incontinence Clinical manifestations: sometimes vague and overlooked Distended bladder Can be palpated above pubic symphysis Cause discomfort and anxiety Assessment: subjective: frequency with or with out burning, urgency, nocturia, occassional acute discomfort Treatments Analgesics & antispasmodics Urinary catheter Surgery – to remove obstacles Assessments Check for residual urine after voiding Residual should be less than 50 mL.

4 URINARY RETENTION Client may experience discomfort and anxiety.
Frequency of urination and voiding small amounts may occur. Treatment: urinary analgesics and antispasmotics. Urinary catheter may be used, or surgery if indicated. When client able to void, check for residual urine-should be less than 50mL. Residual urine: pt voids amount measured; pt catheterized after voiding and amount measured; amount remaining is residual; Help pt relax, sitz baths, privacy warm showers, warm beverages. Interventions pg 481.

5 URINARY RETENTION Stasis may lead to infection.
Distended bladder may result. Caused by stress, calculus obstruction, stones, tumor, infection, medications, trauma.

6 URINARY RETENTION Medical management/nursing interventions
Warm shower or sitz bath Natural voiding position if possible Urinary catheter Surgical removal of obstruction Analgesics

7 URINARY RETENTION Urinary Analgesics
Pyridium, Pyridate, phenazopyridine: Uses: relief of pain associated with lower genitourinary tract Adverse Reactions: headache, rash, pruritis, GI disturbances, discoloration of the urine, sclera and/or skin. Dosage range: 200 mg TID PO

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9 Urinary Incontinence Etiology/pathophysiology
Involuntary loss of urine from the bladder Total incontinence; dribbling; stress incontinence Secondary Infection; loss of sphincter control; sudden change in pressure in the abdomen Permanent or temporary

10 URINARY INCONTINENCE Stress: leakage of urine on straining.
Urge: sudden need to urinate. Overflow: full bladder leads to leakage. Total: no control of voiding. Nocturnal enuresis: night time incontinence. URINARY INCONTINENCE The involuntary loss of urine from the bladder May be a complication of urinary tract problems or neurologic disorders May be permanent or temporary Medications Sedatives Hypnotics Diuretics Anticholinergics Antipsychotics Alpha antagonists More than 13 million men & women in the U.S. experience incontinence Women twice as often as men Affects the client’s emotional, psychological, & social well-being Can occur with any age but more common in older adults All types of incontinence can be treated at any age. Keeping perineal area dry and intact is a goal for all clients Classifications Stress Urge Overflow Total Nocturnal enuresis STRESS INCONTINENCE Most common type Not a disease or a natural, inevitable effect of aging Anyone can be affect but usually women Leakage of urine when a person does anything that strains the abdomen, such as: Coughing Laughing Jogging Dancing Sneezing Lifting Making a quick movement Walking Medical Management Depends on underlying cause Often can be cured or alleviated Treatments may include: Bladder retraining Medicines, such as conjugated estrogens Surgery – to restore support of pelvic floor muscles or re-construct sphincter Collagen injections surrounding urethra This closes urethra enough to prevent urine from leaking out. Nonsurgical outpatient setting Support prostheses External barriers Kegel exercises (strengthens pelvic floor muscles) Practice 10 times, 7 or 8 times a day Planning a schedule of voiding URGE INCONTINENCE When a person is unable to suppress the sudden urge or need to urinate Sometimes urine leaks without warning. An irritated bladder is often the cause. Infection or very concentrated urine may irritate the bladder. Treatments Clearing up an infection, if present Encouraging client to have a fluid intake of 3,000 mL/day Less fluid does not prevent incontinence but may promote infection. OVERFLOW INCONTINENCE Bladder becomes so full & distended that urine leaks out Occurs when a blocked urethra or bladder weakness prevents normal emptying The blockage may be an enlarged prostate. The distended bladder cannot contract with enough force to expel a stream of urine. Occurs most often in persons who: Have diabetes Drink a large quantity of alcohol Have decreased nerve function Bladder retraining may alleviate the situation TOTAL INCONTINENCE When no urine can be retained in the bladder Client may be able to manage with an indwelling catheter. A neurologic problem is usually the cause. Surgery to make a temporary or permanent urinary diversion may be required. NOCTURNAL ENURESIS Occurs during sleep Limited fluid intake after 6 pm will help. However, total fluid intake for each 24 hours should remain the same. Nursing Management Identify impaired urinary elimination based on subjective & objective data Assess vital signs Encourage adequate fluid intake Teach Kegel exercises Initiate bladder retraining

11 Urinary Incontinence Clinical manifestations/assessment
Involuntary loss of urine Leaking with coughing, sneezing, or lifting Medical management/nursing interventions Treat underlying cause Surgical repair of bladder Temporary or permanent catheter Bladder training Kegal exercises

12 Urinary Tract Infections
Etiology/pathophysiology UTIs are caused by pathogens that enter the urinary tract – with or without presence of symptoms Bacteriuria (bacteria in the urine): the most common of all nosocomial infections; often associated with urinary catheters Common in older people r/t bladder obstruction, insufficient bladder emptying, decreased bactericidal secretions of the prostate, increased perineal soiling in women, sexual intercourse.

13 Urinary Tract Infections
Immobility, sensory impairment, and multiple organ impairment may increase the probability of infection in the older adult Females more susceptible because shorter urethra and proximity to vaginal and rectal area.

14 Urinary Tract Infections
Gram-negative microorganisms from the GI tract (e.g. E. Coli, Klebsiella, Proteus, or Pseudomonas) commonly cause UTIs. They ascend through the urinary meatus. Body usually keeps infections in check by washing them from the body through voiding. If there is incomplete emptying of the bladder or reflux of urine, the retained urine supports growth of bacteria.

15 Urinary Tract Infections
Clinical Manifestations Urgency, frequency, burning on urination Microscopic or gross hematuria Cloudy or blood-tinged urine Nocturia Abdominal discomfort, perineal or back pain Sudden onset incontinence or increased incontinence Type of infection depends on location: cystitis, urethritis, nephritis, etc.

16 Urinary Tract Infection
Treatment Pharmacology: antibiotics Common ones: Norflaxin (Noroxin) Nitrofurantoin (Furadantin) Sulfisoxazole (Gantrisin) Trimethoprim-Sulfamethoxazole ( Bactrim, Septra) Pharmacological Norfloxacin Nitrofurantoin Ciprofloxacin Sulfonamides Determine allergy to sulfonamides or penicillins Length of treatment related to type of cystitis, acute or chronic. Diet Fluids encouraged – 3 to 4 liters of non-caffeinated fluid per day Meats & whole grains – urine more acid Cranberry juice – tannins may prevent e. coli from sticking to cells of urinary tract. Activity Clients on bed rest – answer call light promptly Difficult to empty bladder completely with bedpan. Orders for bathroom privileges or commode are encouraged. Nursing Management Monitor vital signs Record I&O Encourage - Fluid intake, especially water & cranberry juice Voiding more frequently Women to void after intercourse Cotton-crotch undergarments Advise that Pyridium turns urine red-orange & stains clothing Change incontinence control product frequently NURSING PROCESS Assessment Subjective Data Frequency or urgency of urination or nocturia Becomes annoying & embarrassing, regardless of age or sex. Burning & pain when voiding Clients with indwelling catheter – may complain of dysuria, burning, & frequency. Clients often just do not feel well. Objective Data Perineal irritation Urine will small foul & appear cloudy Hematuria may be present. Elderly people may become anorexic & develop a low-grade fever.

17 Urinary Tract Infection
Diagnostic Tests: UA, C&S

18 INFECTIOUS DISORDERS (pp ) Infectious disorders of the urinary system are called urinary tract infections (UTIs). 2 types – upper & lower Lower UTIs – affect the bladder (cystitis) & urethra Upper UTIs – affect the kidneys (pyelonephritis, & acute & chronic glomerulonephritis) & ureters

19 CYSTITIS Inflammation of the urinary bladder.
Caused by escherichia coli, candida albicans, coitus, prostatitis, diabetes mellitus. Culture, sensitivity testing, antimicrobial medication, urinary tract analgesic. Increase fluid intake, record I & O. CYSTITIS Inflammation of the urinary bladder More common in females – short urethra allows bacteria to ascend through the urethra from the vagina or rectum to the bladder. Also, bacteria from an infected kidney can descend through the ureter into the bladder Most UTIs are caused by Escherichia coli. Other common causes: Coitus Prostatitis Diabetes mellitus As women age, pelvic floor muscles relax, leading to decreased ability to empty the bladder completely. In men, usually secondary to another infection. Bacteria in bladder multiply, causing redness & swelling of bladder wall. Results in frequency, dysuria, pyuria, hematuria, sometimes burning & urgency Symptoms increase as bladder distends. Clean-catch midstream urinalysis showing >100,000 organisms/mL confirms diagnosis. Microscopic exam will also show hematuria & pus. Med-Surg Management Medical Medications & fluids Ineffective treatment can result in recurrence. C&S obtained before administration of antimicrobial

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21 CYSTITIS Encourage fluid intake. Should drink between 3-4 liters of non-caffeinated fluid a day. Intake of meats and whole grains makes the urine more acidic and may discourage growth of bacteria in the urinary bladder. Drinking cranberry juice Client Teaching See Nursing diagnosis for a client with cystitis (Impaired Urinary Elimination & Deficient Knowledge) pp Encourage large amount of fluid intake Pyridium – explain that it will stain Importance of taking all medication ordered Clean perineum from front to back Cotton crotch Tight jeans & long bike rides – may irritate the perineum Avoid perfumed perineal products Spermicidal contraceptives can be irritating Void more frequently Women void after sexual intercourse Teach elderly persons to change incontinence control products frequently

22 PYELONEPHRITIS Bacterial infection of renal pelvis, tubules, interstitial tissue of one or both kidneys. Also known as pyelitis or nephropyelitis. Usually associated with pregnancy, chronic health problems such as DM, polycystic or hypertensive kidney disease, insult to the urinary tract such as catheterization, infection, obstruction, or trauma PYELONEPHRITIS Bacterial infection of the: Renal pelvis Tubules Interstitial tissue - of one or both kidneys Also known as pyelitis or nephropyelitis Bacteria generally ascend from the urinary bladder through the ureter & enter the kidney in the area known as the renal pelvis. Bacteria can also enter from the blood & lymph. Can be secondary to: Ureterovesicular reflux When urine cannot drain from the pelvis of the kidney – obstruction May also occur: During pregnancy With prostatitis When bacteria are introduced during a cystoscopy or catheterizationTrauma of the UT Can be acute illness or chronic condition Leads to development of high blood pressure and/or chronic renal failure Escherichia coli – most often cultured Inflamed kidney becomes edematous – renal vessels congested Urine usually cloudy, containing mucus, blood, & pus.

23 PYELONEPHRITIS Kidney becomes edematous, inflamed; blood vessels congested Urine may be cloudy and contain pus, mucous, and blood Small abscesses may form in the kidney Symptoms of acute condition: chills, fever, flank pain, prostration

24 PYELONEPHRITIS Repeated episodes  chronic pyelonephritis and atrophy of the kidney with nephrons being destroyed. Destruction of nephrons  Azotemia: retention in the blood stream of excessive amounts of nitrogenous compounds Treat to prevent from becoming chronic

25 PYELONEPHRITIS Diagnostic tests could be: IVP, UA and C&S, CBC, BUN, serum creatinine. Collect urine specimens before administering antimicrobials Pharmacology: sulfonamides (Bactrim,Cipro); antipyretics if with fever, analgesics if in pain. Med-Surg Management Medical Diagnostic tests: IVP Urinalysis with C&S CBC BUN Serum creatinine Pharmacological Sulfonamides Antimicrobial ciprofloxacin hydrochloride Antipyretics Analgesics Diet Light diet during febrile stage Fluids increased to 3,000 m/L/day Activity Disease process will cause fatigue. Bed rest during acute phase Diversionary activities

26 Immunological Disorders of the Kidney
Nephrotic syndrome Etiology/pathophysiology Physiologic changes of the glomeruli interfere with selective permeability Clinical manifestations/assessment Proteinuria; hypoalbuminemia Generalized edema Anorexia Fatigue Oliguria

27 Nephrotic Sydrome

28 Immunological Disorders of the Kidney
Medical management/nursing interventions Corticosteroids Diuretics Diet Low sodium High protein

29 Immunological Disorders of the Kidney
Nephritis (acute glomerulonephritis) Etiology/pathophysiology: in taking a health hx., will usually find that an infectious disease process triggers an immune response. Frequently a beta-hemolytic streptococcus (2-3 weeks prior) The immune response  inflamed glomeruli excretion of RBCs and protein in the urine

30 Immunological Disorders of the Kidney
Clinical manifestations/assessment Edema of the face – esp. around eyes Pallor Malaise Anorexia Dyspnea with exertion Hematuria – “cola” colored  frank bleeding Changes in voiding patterns Oliguria; dysuria

31 Immunological Disorders of the Kidney
Diagnostic Tests: Blood tests will usually show: elevated BUN, serum Creatinine, potassium, ESR, and antistreptolysin-O titer. Urinalysis will show presence of RBCs, casts, and protein Treatment includes drug therapy, diet, and rest. Treat to prevent renal complications, cardiac complications, and complications to cerebral functioning.

32 Immunological Disorders of the Kidney
Medical management/nursing interventions Antibiotics Treat primary symptoms Diuretics Antihypertensives Diet Protein and sodium restrictions Increase calories

33 Immunological Disorders of the Kidney
Pharmacology Prophylactic antimicrobial therapy possible Drug of choice is Penicillin Diuretic and antihypertensive drugs may be ordered Corticosteroids, chemotherapeutic drugs, and/or immunosuppressive drugs to control inflammatory response. Pharmacological Prophylactic antimicrobial therapy - penicillin Diuretic & antihypertensive medication Drugs to control inflammatory response Diet Fluid restriction due to fluid retention Adjusted according to I&O & weight Protein regulated to rest kidneys Potassium replaced Sodium restricted to prevent fluid retention Activity Physical & emotional rest are essential. Bedrest until inflammation has subsided, urinary flow increases, & as long as hematuria or proteinuria. Strict turning schedule – skin breakdown with edema

34 Immunological Disorders of the Kidney
Nursing Interventions Focus is on control of symptoms and prevention of complications Monitor level of consciousness if BUN is elevated VS , I/O Bedrest and fluid adjustments are guided by urine output until diuresis is adequate Level of Activity: depends on the degree of edema, BP, proteinuria, and hematuria – all of which increase with excessive activity ACUTE GLOMERULONEPHRITIS A condition that can affect one or both kidneys In both acute & chronic disease, the glomerulus within the nephron unit becomes inflamed. Predominantly a disease of children & young adults – when the cause is bacterial. Viral form can affect all ages. Prognosis For most clients – full recovery Some may develop chronic glomerulonephritis Approximately 1% to 2% of people with end-stage renal disease from acute glomerulonephritis Acute glomerulonephritis during childhood is known as Bright’s disease. Mechanism Most clients develop symptoms 1 to 3 weeks after: An upper respiratory infection (tonsillitis or pharyngitis with fever) Skin infection caused most commonly by group A -hemolytic streptococcus Infection triggers an autoimmune response & glomeruli are attacked by antibodies at the site of the glomerular basement membrane, resulting in inflammation. Some clients are asymptomatic. A nephrotoxic drug or systemic disease such as diabetes or lupus may also be a cause. Med-Surg Management Medical Drug therapy Diet Rest Focus on prevention of renal, cardiac & cerebral complications Diet: salt not restricted usually If BUN elevated: protein restricted, liberal carbs, may have sodium restriction if low urine output indicates impaired renal function Potassium according to individual needs…if oliguria becomes severe, renal clearance of potassium is impaired Water: according to urine output, may not need restriction

35 Immunological Disorders of the Kidney
Patient Teaching Nature of illness Effect of diet and fluids on fluid balance and sodium retention Diet: prescribed sodium and fluid restriction Info on protein restriction/ CHO sources for energy Medication Pacing daily activities Avoiding trauma and infections S/Sx that require medical attention Importance of medical follow up

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37 Immunological Disorders of the Kidney
Nephritis /Chronic Glomerulonephritis) Etiology/pathophysiology Slow, progressive destruction of glomeruli Commonly caused by other chronic illnesses Diabetes mellitus Systemic lupus erythematosus

38 Immunological Disorders of the Kidney
Clinical manifestations/assessment Malaise; morning headaches Dyspnea with exertion Visual and digestive disturbances Generalized edema Weight loss Fatigue Hypertension Anemia Proteinuria

39 Immunological Disorders of the Kidney
Chronic Glomerulonephritis (cont.) Medical management/nursing interventions Same as acute glomerulonephritis Bedrest, dietary modification, medication Goal: prevent further renal damage; prevent cerebral and cardiac complications Renal dialysis Kidney transplant

40 Immunological Disorders of the Kidneys
Pharmacology Antimicrobial therapy given prophylactically Diuretics and antihypertensive drugs ordered Pharmacological Diuretic & hypertensive medications are ordered. Antimicrobial therapy – prophylactically Diet Fluid intake is adjusted to urinary output. Protein in diet regulated Sodium & potassium restriction determined by serum electrolyte levels Carbohydrates increased to provide energy Activity Bed rest if hematuria or albuminuria Nursing Management Assist client -With ADLs, encourage bed rest with diversional activities To reposition frequently & assess skin Measure urine hourly or as ordered Assess – Color, consistency of urine Lung sounds, edema, speech & mental functioning Weigh client daily Monitor laboratory reports NURSING PROCESS Assessment Subjective Data Client may complain of: Morning headache Pruritis Decreased ability to concentrate Fatigue Dyspnea Facial edema Objective Data Fluid retention becomes evident, leading to shortness of breath, especially at night. Vital signs, usually hypertension Lung sounds every shift – crackles – sign of fluid retention Weight daily, degree of edema, pitting or nonpitting Urine becomes pale & dilute Client Teaching See Nursing diagnosis for a client with chronic glomerulonephritis (Anxiety), p. 1013 Assist client to express concerns about possible dialysis

41 PHARMACOLOGY Thiazides: hydrochlorothiazide Loop diuretics
Types of diuretics: Thiazides: hydrochlorothiazide Loop diuretics Potassium sparing diuretics Osmotic diuretics Carbonic Anhydrase inhibitor diuretics Monitor output, maintain fluid restrictions and weigh daily. Monitor for fluid overload; changes in pulse rates, respirations, cardiac sounds, lung sounds. Accurate I&O V?S q 4 hrs until stabilized; no over use of salt ; addition of potassium (baked potatoes, apricots, raw bananas, navel oranges); sometimes potassium sibs needed. May be potentiating effect with other meds as diuretic begins to work. Loop diuretics: useful if greater diuretic effect is desired. Furosemide (Lasix)

42 PHARMACOLOGY Diuretics: -drug that increases the secretion of urine.
-kidney disease often causes excess fluid retention (edema). -many different types of diuretics used for different purposes.

43 PHARMACOLOGY Antihypertensives methydopa (Aldomet)
minoxidil ( Loniten) hydralazine HCL ( Apresoline) Monitor BP, pulse, postural hypotension, and K, Na,Cl, and CO2 and I&O

44 PHARMACOLOGY Phosphate binding antacids: aluminum hydroxide gel ( Amphogel) Potassium exchange: sodium polystrene Electrolyte Replacement: calcitrol (Rocaltrol)


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