1. DR.HINA ADNAN

2. 1. Abscesses of periodontium. 2. Necrotizing periodontal diseases. 3. Gingival disease of viral origin – herpes virus. 4. Recurrent aphthous stomatitis. 5. Allergic reaction.

3. Abscesses of periodontium 1. Gingival abscess. 2. Periodontal abscess. 3. Pericoronal abscess.

4. Periodontal Vs periapical abscess periodontal periapical Vital tooth. No Carious. Pocket. lateral radioluicency. Mobility. Percussion sensitivity variable. Sinus tract open via keratinized gingiva. Non vital. Carious. No pocket. Apical radioluicency. No or minimal mobility. Percussion sensitivity. Sinus tract via alveolar mucosa.

5. Necrotizing periodontal diseases

6. Gingival disease of viral origin – herpes virus Acute manifestation of viral infections of oral mucosa characterized by redness and multiple vesicles that easy rupture to form painful ulcers affecting the gingiva.

7. Primary herpetic gingivostomatitis Classic initial infection of herpes simples virus type 1. Mainly in young children. 90% of primary oral infection are asymptomatic.

8. Clinical features 1. Painful severe gingivitis with ulcerations, edema and stomatitis. 2. Vesicles rupture coalesce and form ulcers. 3. Fever and Lymphadenopathy are classic features. 4. lesion usually resolve in 7-14 days.

9. Treatment 1. Bed rest. 2. Fluid. 3. Nutrition. 4. Antipyretics. 5. Pain relieve. 6. Antiviral medications.

10. Recurrent oral herpes ‘Fever blisters’ or ‘cold sores’. Oral lesion usually herpes simples type 1. Recurrent infections in 20%-40% of those with primary infection. herpes labials are common. Less severe than primary infection.

12. Clinical features 1. Prodromal syndrome. 2. lesion start as vesicles and then rupture leave ulcers. 3. A cluster of small painful ulcers on attached gingiva or lip is characteristic. 4. Can cause post operative pain following dental treatment.

13. Virus reactivation 1. Fever. 2. systemic infection. 3. Ultraviolet reaction. 4. Stress. 5. Immune system changes. 6. Trauma. 7. Undefined cases.

14. Treatment 1. Palliative care. 2. Antiviral medication.

15. Recurrent aphthous stomatitis Canker sores. Etiology unknown. 10%-20% of population. Usually begins in childhood. Decreasing with age.

18. Clinical features 1. Affect mobile mucosa. 2. Three forms: I. Minor. II. Major. III. Herpitiform.

19. Minor aphthous Clinical features: 1. most common. 2. Small shallow ulceration with slightly raised erythematous borders. 3. Central are covers by white-yellow pseudomembrane. 4. Heal without scarring in 10-14 days.

21. Major aphthous Clinical features: 1. Herpetiform aphthae : small recurrent ulcerations Lesion usually small than herpetic stomatitis. 2. Similar to herpetic stomatitis but no vesicles. 3. Heal with 7-10 days without scaring.

23. Predisposing factors 1. Stress. 2. Trauma. 3. Food hypersensitivity. 4. Previous viral infection. 5. Nutritional deficiency.

24. Allergic reaction Dental restorative materials.( mercury, nickel, gold, zinc). Toothpaste and mouthwashes. Foods ( peanut, red peppers )