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The Pediatric Spine Normal Spine Development Thoracic Kyphosis =

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Presentation on theme: "The Pediatric Spine Normal Spine Development Thoracic Kyphosis ="— Presentation transcript:

1 The Pediatric Spine Normal Spine Development Thoracic Kyphosis =
20-45 deg Lumbar lordosis = 20-55 deg By age 4-5 usually 10-20 Cervical Mild truncal asymmetries common 10% of population School screen controversial Threshold of 7 deg. scoliometer suggested

2 Spinal Flexion and Extension
Cerebral Palsy Tightness: Cervical and capital flexion Thoracic extension Lateral flexion Lumbar flexion

3 Kyphosis Postural Congenital Failure of formation and/or segmentation
Apex of curve between T10 and L1 most common Failure of formation lead to paraplegia Progressive under deg. – post. Fusion More than 60 deg – ant. and post.

4 Scheurermann Disease Familial disorder of thoracic spine
Hx of heavy physical loading from work or athletics Vertebral wedging and kyphosis >45 deg <60 – encourage activity >60 - brace Mild scoliosis Tx: NSAIDs, rest, stress reduction, TSLO for pain

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6 Lordosis Variations common in typical prepubescent child Functional
Compensation for fixed deformity above or below LS level Structural hyper/hypo Arrest growth of post lumbar vert with shunting or rhizotomy Muscular dsytrophy = hypo Fractures = hypo or hyper

7 Cervical Problems Basilar impression Congenital or acquired
Cervical spine extends into the formamen magnum May be due to osteopenia: rickets osteogenesis imperfecta

8 Occipital-atlantal instability
Rare but sometimes in Down Syndrome Atlantoaxial instability Due to abnormalities of odontoid or to ligamentous laxity Down Syndrome, Rheummatoid arthritis, Sx: gait disturbance, ex. Intolerance, neck pain Mild weakness and hyperreflexia Avoid cervical spine stress

9 Can have instability until the epiphysis closes
X-rays recommended at age 2 y.r. in the case of Down Syndrome Case example: 7 year old high functioning child with Down Syndrome, in a regular classroom who can ride a tricycle independently. He/she falls off and becomes a paraplegic due to an unstable AA joint

10 Neurological Abnormalities
Progression of scoliosis Chiari malformation Tethered cord Tumors Unresolved torticollis Clumsiness UE weakness MRI studies helpful

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12 Abnormal skin lesions in midline
Dimples Hemangiomas Hair patches Cavus feet Leg atrophy Café au lait spots

13 Back Pain Referral important if Evaluate for: Onset before age 4 years
Symptoms persist longer than 4 weeks or increases Pain interferes with function Recent onset of scoliosis Evaluate for: Mobility and symmetry Tenderness Neurological signs – especially asymmetry Xray, SPECT scan or MR imaging

14 Spondylolysis and Spondylolisthesis
(B) or unilat defects of pars interarticularis = Sphondylolysis 4% of 4 yo, 6% by maturity If this displaces = spondylolisthesis Most common form of back pain in children and adults and in children with abnormal bone or connective tissue Usually due to stress fracture Tenderness over L5-S1 Limited SLR and forward bending Pain aggravated by activity, especially competitive sports Usually pain decreases as child becomes an adult due to decreased activity

15 Benign Back Pain in Children and Adolescents
over ½ in this category Limit back pack weight (< 20% of body wt Not EBP Encourage healthy lifestyle, activity, weight reduction Disc Herniation Rare unless family hx, recent trauma, facet asymmetry, spinal stenosis, transitional vertebrae and spondylolisthesis Usually L4-5 or L5-S1 with radicular pain and secondary spinal deformity Tx: NSAID, rest, limited activity, TLSO MR and disc excision if persists

16 Idiopathic Scoliosis Unknown origin Most common
Infantile: birth to 3 yrs Juvenile: 3-10 yrs Adolescent: >10 yrs Prevalence: 2-3% for curves <10 deg .2 to .3% for curves >20 deg

17 Nonstructural and Structural
Correct on lateral trunk bending Causes: Pelvic Obliquity LLD Tumor Muscle spasm Fixed and do not correct Rotary component present and visible with forward bending Primary and compensatory curves

18 Neurofibromatosis Mutation of neurofibromatosis (Type 1) or schwannomin (Type 2) gene Type 2 has fewer peripheral but more intracranilal lesions Scoliosis is idiopathic or dystropic Dystropic involves short and sharp angulation and spinal instability Fusion is indicated because of risk of boney dysplasia

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20 School Screenings Examine posture anteriorly and posteriorly
Forward bend test MD and RN do this and often miss some cases Assess for asymmetries: Shoulders, nipple, scapula, pelvic heights Asymmetrical folds of trunk Curvature of spine

21 Treatment for Scoliosis
Young adolescents prepuberty will most likely progress Curves <25 deg: observe Curves deg: non-surgical methods Goal is to maintain with exercise and brace TLSO hours per day until skeletal maturity PT: donning orthosis, schedule, skin care, exercise for ROM (hip flexors) and strength (abdom, gluts, paraspinals) Curves >40 deg: surgical intervention

22 Surgery for Scoliosis Indications: Instrumentation: PT:
Curve >40 deg Curve is progressing Decompensation of spine or thoracic cavity Instrumentation: Distract and compress the curve Correct or minimize the rotory component Stabilize and maintain Harrington rods PT: Preop and postop for ROM, trunk strengthening, deep breathing and coughing Early transfers and gait

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