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Update on massive transfusion
H.P. Pham, B.H. Shaz British Journal of Anaesthesia Volume 111, Pages i71-i82 (December 2013) DOI: /bja/aet376 Copyright © 2013 The Author(s) Terms and Conditions
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Fig 1 Pathogenesis of haemostasis abnormality in MT. Dilutional coagulopathy, activation of inflammatory mediators, hyperfibrinolysis, thrombocytopathy, and metabolic abnormalities (hypothermia, hypocalcaemia, and acidosis) all contribute to the pathogenesis of the haemostasis abnormality in massive haemorrhage. British Journal of Anaesthesia , i71-i82DOI: ( /bja/aet376) Copyright © 2013 The Author(s) Terms and Conditions
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Fig 2 TEG tracing. Modified from Figure 1 in Cushing and Shaz,44 with permission. R, reaction time; K, kinetics time; MA, maximum amplitude. British Journal of Anaesthesia , i71-i82DOI: ( /bja/aet376) Copyright © 2013 The Author(s) Terms and Conditions
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