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Deconstructing Export of Malaria Proteins

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Presentation on theme: "Deconstructing Export of Malaria Proteins"— Presentation transcript:

1 Deconstructing Export of Malaria Proteins
Serge Bonnefoy, Robert Ménard  Cell  Volume 134, Issue 1, Pages (July 2008) DOI: /j.cell Copyright © 2008 Elsevier Inc. Terms and Conditions

2 Figure 1 The Malaria Parasite Remodels the Host Erythrocyte
After invading the host red blood cell (RBC), the malaria parasite Plasmodium falciparum resides within a parasitophorous vacuole (PV) and induces extensive remodeling of the host erythrocyte. For example, P. falciparum induces the formation of a tubulo-vesicular network (TVN) linked to the PV, as well as flat lamellar membranes called Maurer's clefts that lie below the erythrocyte plasma membrane. The parasite also directs the formation of molecular protuberances (knobs) at the host cell surface that display parasite surface proteins such as PfEMP1, which allow infected erythrocytes to adhere to host endothelial cells through interaction with endothelial receptors. Cell  , 20-22DOI: ( /j.cell ) Copyright © 2008 Elsevier Inc. Terms and Conditions

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