1. Interaction between vasodilators and vasopressin in internal mammary artery and clinical significance 
Wei Wei, MD, H.Storm Floten, MD, Guo-Wei He, MD, PhD 
The Annals of Thoracic Surgery 
Volume 73, Issue 2, Pages (February 2002)
DOI: /S (01)

2. Fig 1
(A) Mean concentration (−log10 mol/L)-relaxation (% reversal of arginine vasopressin [10−8.5 mol/L]-induced contraction) curves for five relaxant agents in the human internal mammary artery (n = 11 for nifedipine [NIF], n = 10 for verapamil [VER], n = 9 for diltiazem [DIL], n = 10 for nitroglycerin [NTG], and n = 11 for selective V1 receptor antagonist [dPVDAVP]). The rings were taken from 9 to 11 patients in each group. (B) Mean concentration (−log10 mol/L)-relaxation (% reversal of 25 mmol/L K+-induced contraction) curve for dPVDAVP. Values are expressed as mean ± standard error of the mean. (*p = versus dPVDAVP; two-way analysis of variance.)
The Annals of Thoracic Surgery  , DOI: ( /S (01) )

3. Fig 2
Mean concentration (−log10 mol/L)-contraction (percentage of 100 mmol/L K+-induced contraction) curves for three calcium-channel antagonists: nifedipine (NIF; A), verapamil (VER; B), and diltiazem (DIL; C). Four rings from each of 7 patients were randomly allocated to different treatments for each calcium-channel antagonist. Calcium-channel antagonists were added into organ baths 20 minutes before the start of AVP contraction. Values are expressed as mean ± standard error of the mean (maximal contraction, p = for nifedipine [A], p = for verapamil [B], and p = for diltiazem [C]; one-way analysis of variance). No significant difference was found among the four curves of each drug by two-way analysis of variance.
The Annals of Thoracic Surgery  , DOI: ( /S (01) )

4. Fig 3
Mean concentration (−log10 mol/L)-contraction (percentage of 100 mmol/L K+-induced contraction) curves for nitroglycerin (NTG). Four rings from each of 7 patients were randomly allocated to four groups. Nitroglycerin (10−7, 10−6, 10−4.5 mol/L, or vehicle) was added 10 minutes before the start of arginine vasopressin (AVP) contraction. Values are expressed as mean ± standard error of the mean. (*p = 0.04 for nitroglycerin [10−6 mol/L] versus control; p = for nitroglycerin among the four curves by one-way analysis of variance.)
The Annals of Thoracic Surgery  , DOI: ( /S (01) )

5. Fig 4
Mean concentration (−log10 mol/L)-contraction (percentage of 100 mmol/L K+-induced contraction) curves for the selective V1 receptor antagonist [1-deaminopenicillamine, 4-valine, 8-d-arginine] vasopressin (dPVDAVP). Four rings from each of 6 patients were allocated to four groups. [1-Deaminopenicillamine, 4-valine, 8-d-arginine] vasopressin (10−8, 10−7, 10−6 mol/L, or vehicle) was added into the organ bath 20 minutes before the start of arginine vasopressin (AVP) contraction. Values are expressed as mean ± standard error of the mean. (*p < 0.05, **p < versus control curve; one-way analysis of variance; p = for dPVDAVP [10−8 mol/L], *p = for dPVDAVP [10−7 mol/L], **p = for dPVDAVP [10−6 mol/L] versus control; by two-way analysis of variance.)
The Annals of Thoracic Surgery  , DOI: ( /S (01) )