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Published byJean-Louis Thibault Lortie Modified over 6 years ago
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Current management of the complications of cirrhosis and portal hypertension: Variceal hemorrhage, ascites, and spontaneous bacterial peritonitis Guadalupe Garcia–Tsao Gastroenterology Volume 120, Issue 3, Pages (February 2001) DOI: /gast Copyright © 2001 American Gastroenterological Association Terms and Conditions
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Fig. 1 Pathogenesis of portal hypertension and its two main complications: varices and ascites. Gastroenterology , DOI: ( /gast ) Copyright © 2001 American Gastroenterological Association Terms and Conditions
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Fig. 2 Treatment options for variceal hemorrhage placed in the context of its pathogenesis. Although theoretically (and in vitro) the mechanism of action of venodilators is a reduction in intrahepatic resistance, in vivo the action seems to be through reflex splanchnic vasoconstriction. Gastroenterology , DOI: ( /gast ) Copyright © 2001 American Gastroenterological Association Terms and Conditions
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Fig. 3 Treatment options for ascites placed in the context of its pathogenesis. Gastroenterology , DOI: ( /gast ) Copyright © 2001 American Gastroenterological Association Terms and Conditions
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Fig. 4 Treatment options for HRS and SBP. HRS is the result of extreme vasodilatation and an extreme decrease in effective blood volume. SBP can lead to renal dysfunction through aggravation of vasodilatation. TIPS applies to therapy of HRS but not of SBP. Gastroenterology , DOI: ( /gast ) Copyright © 2001 American Gastroenterological Association Terms and Conditions
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