1. Pathogenesis and Treatment of Hepatitis E Virus Infection
Heiner Wedemeyer, Sven Pischke, Michael P. Manns 
Gastroenterology 
Volume 142, Issue 6, Pages e1 (May 2012)
DOI: /j.gastro
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2. Figure 1
(A) Genomic organization of HEV including the 3 ORFs. The scale shows nucleotides in thousands. (B) Phylogenetic tree depicting different genotypes and subtypes of HEV. The sequences from patients with chronic HEV genotype 3 infection are shown as a separate phylogenetic tree (right). Red squares show chronic HEV patients from Hannover after either heart (HTx) or lung (LTx) transplantation.66
Gastroenterology  , e1DOI: ( /j.gastro )
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3. Figure 2
(A) Worldwide prevalence of HEV and (B) the geographic distribution of the different HEV genotypes.
Gastroenterology  , e1DOI: ( /j.gastro )
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4. Figure 3
Diagnosis of hepatitis E. Typical evolution of the serologic titer of HEV-specific antibodies and levels of alanine aminotransferase (ALT) levels during (A) acute self-limited or (B) chronic HEV infection.
Gastroenterology  , e1DOI: ( /j.gastro )
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5. Figure 4 Course of HEV infection.
Gastroenterology  , e1DOI: ( /j.gastro )
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6. Supplementary Figure 1
H&E staining of a biopsy sample from a patient with chronic hepatitis E shows portal inflammation with predominant lymphocyte infiltration and interface hepatitis (A, 100×; B, 400×).
Gastroenterology  , e1DOI: ( /j.gastro )
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7. Gastroenterology 2012 142, 1388-1397. e1DOI: (10. 1053/j. gastro. 2012
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8. Gastroenterology 2012 142, 1388-1397. e1DOI: (10. 1053/j. gastro. 2012
Copyright © 2012 AGA Institute Terms and Conditions

9. Gastroenterology 2012 142, 1388-1397. e1DOI: (10. 1053/j. gastro. 2012
Copyright © 2012 AGA Institute Terms and Conditions