Presentation is loading. Please wait.

Presentation is loading. Please wait.

Accumulation of 2,4‐dienoyl‐CoA ester (C22:7) during β‐oxidation of docosahexaenoic acid (C22:6). Accumulation of 2,4‐dienoyl‐CoA ester (C22:7) during.

Published byDoddy Jayadi Modified over 5 years ago

Similar presentations

Presentation on theme: "Accumulation of 2,4‐dienoyl‐CoA ester (C22:7) during β‐oxidation of docosahexaenoic acid (C22:6). Accumulation of 2,4‐dienoyl‐CoA ester (C22:7) during."— Presentation transcript:

1 Accumulation of 2,4‐dienoyl‐CoA ester (C22:7) during β‐oxidation of docosahexaenoic acid (C22:6).
Accumulation of 2,4‐dienoyl‐CoA ester (C22:7) during β‐oxidation of docosahexaenoic acid (C22:6). TLC analysis of 14C‐labelled product derived from docosahexaenoic acid (C22:6) β‐oxidation in wild‐type (wt) and Δidp3 cells. The products formed were analysed after incubation for 0, 30 and 60 min with docosahexaenoic acid (C22:6). Lane M corresponds to an incubation to which acyl‐CoA oxidase was added to allow conversion of C22:6‐CoA to C22:7‐CoA. Carlo W.T. van Roermund et al. EMBO J. 1998;17: © as stated in the article, figure or figure legend

Download ppt "Accumulation of 2,4‐dienoyl‐CoA ester (C22:7) during β‐oxidation of docosahexaenoic acid (C22:6). Accumulation of 2,4‐dienoyl‐CoA ester (C22:7) during."

Similar presentations

Validating Cdk‐dependent chromatin association changes of selected outliers A–EValidation of changes in chromatin affinity following Cdk inhibition of.

Validating Cdk‐dependent chromatin association changes of selected outliers A–EValidation of changes in chromatin affinity following Cdk inhibition of.
Hydration profiles of GLIC′s pore in WT and Ser6′ mutant simulations.

Hydration profiles of GLIC′s pore in WT and Ser6′ mutant simulations.
Protection of HIF‐1α against VHL‐mediated degradation requires nuclear translocation of HIF‐1α and a hypoxia‐dependent intranuclear signal. Protection.

Protection of HIF‐1α against VHL‐mediated degradation requires nuclear translocation of HIF‐1α and a hypoxia‐dependent intranuclear signal. Protection.
Induction of mPer1 mRNA expression by prostaglandin E2 (PGE2) in NIH3T3 cells. Induction of mPer1 mRNA expression by prostaglandin E2 (PGE2) in NIH3T3.

Induction of mPer1 mRNA expression by prostaglandin E2 (PGE2) in NIH3T3 cells. Induction of mPer1 mRNA expression by prostaglandin E2 (PGE2) in NIH3T3.
GFP‐Sed5p localization defect in sgt2Δ.

GFP‐Sed5p localization defect in sgt2Δ.
Mba1 cofractionates with mitochondrial ribosomes on sucrose gradients.

Mba1 cofractionates with mitochondrial ribosomes on sucrose gradients.
Purified Nop1p and Rnt1p are able to process pre‐U18 molecules.

Purified Nop1p and Rnt1p are able to process pre‐U18 molecules.
Mitochondrial ribosomes remain associated with the inner membrane in the absence of the ribosome‐binding domain of Oxa1 and of Mba1. Mitochondrial ribosomes.

Mitochondrial ribosomes remain associated with the inner membrane in the absence of the ribosome‐binding domain of Oxa1 and of Mba1. Mitochondrial ribosomes.
CG3066 encodes a prophenoloxidase‐activating enzyme.

CG3066 encodes a prophenoloxidase‐activating enzyme.
Surface distribution of immunogold‐labelled PS I (A–C) and ATP synthase (D–F) complexes. Surface distribution of immunogold‐labelled PS I (A–C) and ATP.

Surface distribution of immunogold‐labelled PS I (A–C) and ATP synthase (D–F) complexes. Surface distribution of immunogold‐labelled PS I (A–C) and ATP.
IRES‐dependent translation is specifically abolished by an IRES point mutation. IRES‐dependent translation is specifically abolished by an IRES point mutation.

IRES‐dependent translation is specifically abolished by an IRES point mutation. IRES‐dependent translation is specifically abolished by an IRES point mutation.
Lsh does not regulate global histone methylation levels.

Lsh does not regulate global histone methylation levels.
Expression of adenylyl cyclases and miR‐142‐3p in CD4+CD25− T cells and CD4+CD25+ regulatory T cells. Expression of adenylyl cyclases and miR‐142‐3p in.

Expression of adenylyl cyclases and miR‐142‐3p in CD4+CD25− T cells and CD4+CD25+ regulatory T cells. Expression of adenylyl cyclases and miR‐142‐3p in.
Increased proliferation and delayed differentiation of adult primary myoblasts derived from MyoDR99,102 mice. Increased proliferation and delayed differentiation.

Increased proliferation and delayed differentiation of adult primary myoblasts derived from MyoDR99,102 mice. Increased proliferation and delayed differentiation.
Mitochondrial uptake of different DNA substrates.

Mitochondrial uptake of different DNA substrates.
Conductin levels oscillate during the cell cycle.

Conductin levels oscillate during the cell cycle.
Leaky scanning past the CPA1 and arg‐2 uORFs.

Leaky scanning past the CPA1 and arg‐2 uORFs.
Yeast containing pICC10 (Tir) and pICC267 (CK18) and pTY474S and pICC267 demonstrated an approximately 12.5‐fold increase in β‐galactosidase activity compared.

Yeast containing pICC10 (Tir) and pICC267 (CK18) and pTY474S and pICC267 demonstrated an approximately 12.5‐fold increase in β‐galactosidase activity compared.
The polypyrimidine tract of βIVS‐II induces efficient 3′‐end formation

The polypyrimidine tract of βIVS‐II induces efficient 3′‐end formation
SERCA2 is a functional adaptor for the alternative toll‐like receptor 9 (TLR9) signalling in cardiomyocytes Co‐immunoprecipitated TLR9 with SERCA2 antibody.

SERCA2 is a functional adaptor for the alternative toll‐like receptor 9 (TLR9) signalling in cardiomyocytes Co‐immunoprecipitated TLR9 with SERCA2 antibody.

Similar presentations

Ads by Google