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GI Motility: Microbiota and Macrophages Join Forces

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Presentation on theme: "GI Motility: Microbiota and Macrophages Join Forces"— Presentation transcript:

1 GI Motility: Microbiota and Macrophages Join Forces
Michelle L. Robinette, Marco Colonna  Cell  Volume 158, Issue 2, Pages (July 2014) DOI: /j.cell Copyright © 2014 Elsevier Inc. Terms and Conditions

2 Figure 1 A Model for Neuroimmune Crosstalk Regulating GI Tract Peristalsis The presence of bacterial products, including LPS, causes enteric neurons to produce CSF1, which binds to the CSF-1R on CX3CR1+CD11cloMHCIIhi MMs and promotes their maintenance. MMs produce BMP2, which binds to the BMP receptor, composed of BMPRI and BMPRII, the latter of which is expressed on axons in the myenteric plexus, but not in ICC or enteric glia. Through a yet-undefined mechanism, BMP2 binding to enteric neurons regulates intestinal motility. Submucosal neural plexus and additional ICC layers are excluded for simplicity. Cell  , DOI: ( /j.cell ) Copyright © 2014 Elsevier Inc. Terms and Conditions

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