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SITE OF LESION TESTING:
Distinguishing: Sensory (cochlear) from neural (retro-cochlear) disorder. Different sources of conductive disorder
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MEASURES OF SUCCESS: SENSITIVITY
Percentage of persons with a disorder who show up on your test as having that disorder. In this application, % of persons with neural disorder that show a “neural result” on the site of lesion test.
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MEASURES OF SUCCESS: SPECIFICITY
percentage of persons without a disorder who show up on your test as not having that disorder. In this application, % of persons with a cochlear disorder (or no auditory disorder at all) who show up on your test as not having any neural disorder.
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Loudness Recruitment Tests
Based on the changes in loudness perception that accompany different auditory disorders.
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Loudness Growth Patterns
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Recruitment: "Abnormal growth of loudness" or, persistence of normal loudness above threshold. More common at higher frequencies.
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Complete: loudness curve meets normal line
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Partial: loudness curve approaches normal line
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Hyper- loudness curve crosses above normal line
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Recruitment is consistent with cochlear damage
from noise ototoxic substances aging and other causes
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Decruitment: Abnormal impairment of loudness growth
loudness curve actually moves away from normal line lack of functioning nerve cells to code intensity associated with retro-cochlear (VIIIth n.) lesions.
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Decruitment
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The Alternate Binaural Loudness Balance (ABLB)Test
requires: - normal hrg in one ear at freq to be used - difference in between ears > 25 dB
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ABLB tones pulse alternating between ears 2 or 3 times per judgement.
pt is asked which ear is louder or same - begin at 20 SL in poorer ear, - 0 SL in better ear. - adjust level in better ear 5 dB steps.
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ABLB - find level where loudness judged equal.
- increase poorer ear by 10 or 20 dB and repeat adjustments in better ear.
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PLOTTING ABLB RESULTS:
Use the “LADDERGRAM” Connect decibel values judged equally loud
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ABLB SUCCESS? Sensitivity = 51% Specificity = 88%
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The Alternate Monaural LB (AMLB) Test
tone alternates between 2 frequencies in the same ear. judgment and procedure is similar to ABLB, but comparing "the high pitch versus the low pitch.” generally this is harder for people to do.
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Differential Intensity Discrimination
The Short Increment Sensitivity Index (SISI) The High Level SISI
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The Short Increment Sensitivity Index
detection of brief (200 ms) 1 dB-increments in a 20 SL tone 20 trials > 70 % = cochlear damage < 30 % = other damage or normal
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B. High Level SISI at 75 dB HL Results: > 70 % = normal or cochlear
< 30 % = retrocochlear
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SISI SUCCESS? Sensitivity = 68% Specificity = 90%
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Tone Decay: Loss of audibility for a tone that is on continuously.
Greater decay is indicative of retrocochlear problem. There are different methods:
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Some Tone Decay Tests Carhart: begin at 0 SL, up in 5 dB steps until tone is heard for a full minute Olson-Noffsinger: begin at 20 SL, up until heard for full minute.
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Tone Decay Results: Type I: no decay: norm, conduct or cochlear
Type II: heard for longer times as level is increased: cochlear Type III: No growth with increasing level: retrocochlear
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TONE DECAY SUCCESS? Sensitivity = 75% Specificity = 91%
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Bekesy Audiometry: Pt. controls level of tone,
Continuous tone: tone on constantly (C) Interrupted tone: pulsed on and off (I) Adaptation should only occur for C, not I
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Bekesy Results: I: C and I overlap: norm or cond.
II: C below I at freqs of HL: Cochlear III: I follows loss, C drops to bottom: Retro IV: C below I by dB: Coch or Ret V: I below C: False hearing loss
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BEKESY AUDIOMETRY SUCCESS?
Sensitivity = 42% Specificity = 95%
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Acoustic Reflex/ARD Success?
Sensitivity = 85% Specificity = 86%
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Auditory Evoked Potentials:
ABR: within 10 ms of click: Brainstem disorders. EcochG: Meniere's disease MLR: Primary auditory cortex: difficult to pin down. Late Cognitive Potentials: processing of sense info
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Auditory Brainstem Response:
Response within 10 ms of stimulus waves labeled with Roman numerals Peaks I, III, and V most useful Latencies are the key measure Disorders will produce delays
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ABR SUCCESS? Sensitivity = 97% Specificity = 88%
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Middle Latency Response
10-80ms From primary auditory cortex Highly variable--poor clinical utility Some correlation to Central Auditory Processing Disorders
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Late Cognitive Potentials
ms Processing of sensory information From Primary Auditory and Aud. Association Cortex Varies with Attention/Subject wakefulness
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P-300 Obtained in “oddball” task Not just auditory
Reflects Change in Working Memory-- “Aha!” Changes in latency and amplitude with variety of disorders
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