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13 Assessment and Treatment of the Patient with Cardiac Emergencies
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Assessment and Treatment of the Patient With Cardiac Emergencies
Objectives Explain the indicators used to differentiate chest pain of cardiac origin from chest pain of noncardiac origin, on the basis of clinical evaluation of the patient
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Assessment and Treatment of the Patient With Cardiac Emergencies
Objectives Discuss the pathophysiology of angina pectoris Describe the assessment and management of the patient with angina pectoris
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Assessment and Treatment of the Patient With Cardiac Emergencies
Objectives Explain the pathophysiology of myocardial infarction Discuss the assessment and management of the patient with acute myocardial infarction Identify the physiology associated with congestive heart failure
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Assessment and Treatment of the Patient With Cardiac Emergencies
Objectives Discuss the signs and symptoms of left ventricular failure and those of right ventricular failure List the interventions prescribed for the patient in congestive heart failure Define and describe the pathophysiology, assessment, and management of cardiac tamponade
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Assessment and Treatment of the Patient With Cardiac Emergencies
Objectives Define and describe the pathophysiology, assessment, and management of cardiogenic shock
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Chest Pain Chest pain is the most common presenting symptom of cardiac disease Patients may express a feeling of "impending doom" Often patients prefer to believe that they are merely experiencing "indigestion" and symptoms would be gone by morning
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Chest Pain Chest pain of cardiac origin is typically described as "crushing" or "squeezing" in nature Associated with nausea, vomiting, and diaphoresis May radiate to other areas (jaw, shoulder, arm, etc.)
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Chest Pain
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Chest Pain Neuropathy due to destruction of nerve endings can cause inability to perceive pain due to diseases of the nerves Diabetics May present as congestive heart failure as the first symptom of AMI
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Noncardiac Causes of Chest Pain
Pleurisy Costrochondritis Pericarditis Myocardial contusion Muscle strain Trauma Secondary to trauma Pneumothorax Hemopneumothorax Tension pneumothorax
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Angina Pectoris Pain that results from reduction in blood supply to myocardial tissue Pain is typically temporary Commonly caused by atherosclerosis Often predictably associated with exercise Requires more oxygen than the narrowed blood vessels can supply
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Angina Pectoris
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Angina Pectoris "Stable," or predictable, angina
A particular activity may elicit chest pain Symptoms will usually respond well to appropriate treatment Rest Administration of oxygen
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Angina Pectoris "Unstable" angina Is not elicited by activity
Most often occurs at rest Indicates a progression of atherosclerotic heart disease, and is also referred to as preinfarctional angina
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Angina Pectoris Prinzmetal's angina
Occurs when coronary arteries experience spasms and constrict Decreases myocardial oxygenation It may be significant to note that while the majority of patients have underlying atherosclerotic disease, some may have little or none
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Angina Pectoris Management Place patient at rest in calm, quiet area
Provide reassurance Obtain 12-lead EKG if possible Administer oxygen IV life line Administer nitroglycerin
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Nitroglycerin Causes dilation of blood vessels that reduces the workload of the heart Reducing the need for oxygen because the heart has to pump blood against a lesser pressure Blood remains in dilated vessels and less is returned to the heart
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Acute Myocardial Infarction
Results from a prolonged lack of blood flow to a portion of myocardial tissue and results in a lack of oxygen Myocardial cellular death will follow Electrical properties of cardiac muscle altered or lost Ability of cardiac muscle to function properly is lost
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Acute Myocardial Infarction
Most common cause is thrombus formation Blocks coronary artery Arteries narrowed by atherosclerotic disease are one of the conditions that increase likelihood of myocardial infarction (MI)
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Acute Myocardial Infarction
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Patient Assessment and Management
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Patient Assessment and Management
100% oxygen Establish an IV life line Measure oxygen saturation level Continuous cardiac monitoring, 12 lead EKG to detect ST elevation or ST Depression
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Patient Assessment and Management
Pain control and management Aspirin, Nitroglycerin, Morphine Sulfate, Demerol Efficient and timely transportation to facility with capability to perform percutaneous coronary intervention (PCI) and/or Fibrinolytic therapy to limit the progression of the infarct.
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Heart Failure Is the inability of the myocardium to meet the cardiac output demands of the body caused by Coronary disease Valvular disease
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Heart Failure Is the inability of the myocardium to meet the cardiac output demands of the body caused by Myocardial injury Dysrhythmias Hypertension Pulmonary emboli Systemic sepsis Electrolyte disturbances
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Left Ventricular Failure
When a patient's left ventricle ceases to function in an adequate capacity as to sustain sufficient systemic cardiac output Stroke volume decreased Increased heart rate, vasoconstriction to compensate
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Left Ventricular Failure
When a patient's left ventricle ceases to function in an adequate capacity as to sustain sufficient systemic cardiac output Increased pressure in right and left atrium Blood pushed back into pulmonary system
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Left Ventricular Failure
When a patient's left ventricle ceases to function in an adequate capacity as to sustain sufficient systemic cardiac output Develop pulmonary edema and hypoxia Pink, frothy sputum and significant dyspnea
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Left Ventricular Failure
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Left Ventricular Failure
Emergency management Have patient assume position of comfort 100% high-flow oxygen Utilize pulse oximetry at 90% saturation Consider the use of CPAP in the presence of Pulmonary Edema
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Left Ventricular Failure
Emergency management Monitor LOC for signs of deterioration Establish IV at KVO rate Maintain EKG monitoring Follow protocol for administration of medications
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Left Ventricular Failure
Emergency management Medications Morphine sulfate Furosemide (lasix) Nitroglycerin Beta Blockers Digitalis
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Right Heart Failure When the right ventricle ceases to function properly Causes increase in pressure in right atrium, forcing blood backward into systemic venous system Most common cause is left heart failure
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Right Heart Failure Common causes Valvular heart disease
COPD or cor pulmonale Pulmonary embolism Chronic hypertension Left heart failure
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Right Heart Failure
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Right Heart Failure Emergency management
Have patient assume position of comfort Oxygenation at level to maintain saturation of at least 95% Establish IV at KVO rate Maintain EKG monitoring
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Right Heart Failure Emergency management
Consult physician on administration of medications Observe for signs of developing left heart failure
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Cardiac Tamponade Excess accumulation of fluid in the pericardial sac
Causes vary between trauma and medical Signs and Symptoms: Muffled heart sounds Distention of jugular veins Narrowing pulse pressures
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Cardiac Tamponade Causes vary between trauma and medical
Signs and Symptoms: Hypotension Dyspnea Weak, rapid pulse
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Cardiac Tamponade Beck's triad Pulsus paradoxus Muffled heart sounds
JVD Narrowed pulse pressure Pulsus paradoxus Systolic blood pressure that drops more than 10–15 mmHg during inspiration
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Cardiac Tamponade Emergency management
Ensure and maintain patent airway Administer 100% high-flow oxygen Monitor pulse oximetry Establish and maintain IV support Administer pharmacological agents as indicated
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Cardiac Tamponade Emergency management Pericardiocentesis
Invasive aspiration of fluid from the pericardium with a needle
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Cardiogenic Shock When left ventricular function is so severely compromised that the heart can no longer meet metabolic requirements of the body Often results from extensive myocardial infarction
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Cardiogenic Shock Most critical form of CHF
Ineffective myocardial contractions result in Marked decreased stroke volume Decreased cardiac output Leading to inadequate tissue perfusion Profound hypotension Compensatory tachycardia
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Cardiogenic Shock Ineffective myocardial contractions result in
Tachypnea, often resulting from pulmonary edema Cool, clammy skin caused by massive vasoconstriction Major dysrhythmias
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Cardiogenic Shock Ineffective myocardial contractions result in
Respiratory difficulty Peripheral edema Pulmonary edema
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Cardiogenic Shock Aggressive treatment measures
Airway management with high flow-oxygen Circulatory support, including IV therapy Patient to assume position of comfort
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Cardiogenic Shock Aggressive treatment measures Cardiac monitoring
Pulse oximetry, maintain O2 saturation at least 95% Consider the use of CPAP in the presence of pulmonary edema
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Cardiogenic Shock Medication therapy Various vasopressors Dopamine
Dobutamine Levophed
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Cardiogenic Shock Medication therapy Other medications
Morphine Sulfate Nitroglycerin Lasix Digitalis
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