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Angina Pectoris Prepared by : Ansam Sharef Ahmad Aswad
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Angina Pectoris Definition :
Angina pectoris is a clinical syndrome usually characterized by episodes of pain or pressure in the anterior chest . The cause is usually insufficient coronary blood flow which results in a decreased oxygen supply to meet an increased myocardial demand for oxygen in response to physical exertion or emotional stress.
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Pathophysiology Myocardial ischemia can result from:
A reduction of coronary blood flow caused by fixed &\or dynamic epicardial artery stenosis. Abnormal constriction or deficient relaxation of coronary artery. Reduce O2-carrying capacity of the blood .
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Factors are associated with typical angina pain
Physical exertion (increase myocardial o2 demand). Exposure to cold ( cause elevation of BP increase oxygen demand ) Eating heavy meals Stress or any emotion-provoking situation
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Type of angina Stable angina: predictable & consistent pain that occurs on exertion and relieved by rest. Unstable angina or preinfarction or cresendo angina: symptoms occur more frequently and longer than stable angina. Intractable or refractory angina: sever incapacitating chest pain.
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Type of angina Variant angina or prinzmetals angina : pain at rest with reversable ST-segment elevation, caused by coronary artery vasospasm. Silent ischemia: objective evidence of ischemia (as ECG changes with a test), but pt report no symptom.
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Risk Factors for atherosclerosis
Family history of premature coronary artery disease. DM, systemic HTN. Cigarette smoking. Hypercholesterolemia. Others as obesity, increase levels of lipoprotein,fibrinogen, s.triglycerides.
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Clinical manifestations
May produce pain vary in severity from feeling of indigestion to chocking in retrosternal area , radiate to neck , jaw shoulders , inner aspects of upper arms Feeling of weakness or numbness in the arms , wrists and the hands Shortness of breath
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Clinical manifestations
Pallor, Diaphoresis Dizziness or lightheadedness Nausea and vomiting
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Diagnostic findings Often made by evaluating the clinical manifestations of ischemia and the pts history 12-Lead ECG and blood laboratory values help in making diagnosis C-reactive protein ( CRP) is a marker for inflammation of vascular endothelium which caused by CAD
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Diagnostic findings An elevated blood level of homocysteine ,an amino acid proposed as an independent risk factor for cardiovascular disease , but no studies supported the relationship between elevation of homocysteine and atherosclarosis
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Medical management for the pt with angina pectoris
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The objective is to decrease oxygen demand of myocardium and to increase oxygen supply
1.Nitroglycerin : administered to reduce myocardial oxygen consumption
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2.Beta-adrenergic blocking agents :
( propranolol ,metoprolol , atenolol) appear to reduce myocardial oxygen consumption, result in a reduction in heart rate, decreased BP , and reduced myocardial contractility .
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3-Calcium channel blocking agents : some decrease sinoatrial node automaticity and node conduction ,resulting in slower heart rate
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4- Antiplatelet and anticoagulent medications
Aspirin: prevents platelet activation and reduces the incidence of MI and death with CAD Heparin : prevents the formation of new blood clots
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Oxygen administration :
usually initiated at the onset of chest pain to increase the amount of oxygen delivered to the myocardium and to decrease the pain.
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Prevention Self care action plan changing habits. Stop smoking
Increase level of exercise Cut down on fatty foods Eat more oats, which decrease cholesterol
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Lose wt if u DR. thinks you are overweight.
Make sure your BP is not high by regular check Consider another method of contraceptive if you take pill
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Nsg process for pt with Angina pectoris
Assessment The nurse gather information about the pts symptoms and activities . The nurse may ask about the period that angina last , and if any medication relieve the angina.
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Diagnosis Ineffective myocardial tissue perfusion secondary to CAD as
evidence by chest pain or equivalent symptoms Anxiety related to fear of death
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Diagnosis Noncompliance , ineffective management of therapeutic regimen related to failure to accept necessary lifestyle changes
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Planning and goals The major goal include immediate treatment when angina occur , preventing of angina , reduction of anxiety and absence of complications
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NSG interventions Treating angina : when pt experiences angina the nurse should direct pt to stop activities and sit or rest in bed in semi-fowler position Reducing anxiety : The nurse should explore and implicate that the diagnosis has for the pt providing information about the illness , treatment and methods of preventing its progression
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Preventing pain : when the pt has pain with minimal activity , the nurse alternates the pts activities with rest periods
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Evaluation expected pts outcomes may include
Reports that pain is relieved promptly Reports decreased anxiety
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Understands ways to avoid complications and demonstrates freedom from complications
Adheres to self-care program
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THANK YOU
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