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Nat. Rev. Clin. Oncol. doi: /nrclinonc

Published byJúlio César Felgueiras Guimarães Modified over 5 years ago

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Presentation on theme: "Nat. Rev. Clin. Oncol. doi: /nrclinonc"— Presentation transcript:

1 Nat. Rev. Clin. Oncol. doi:10.1038/nrclinonc.2017.14
Figure 2 Body fluids as a source of tumour-derived molecular information Figure 2 | Body fluids as a source of tumour-derived molecular information. Schematic representation of different body fluids (other than blood) that can contain tumour-derived molecular information, specifically, urine, cerebrospinal fluid (CSF), saliva, and pleural effusions. The localization of the primary tumour and of any metastatic lesions influences the presence of circulating tumour-derived nucleic acids, cells, and microvesicles in individual body fluids. As indicated, point mutations, gene fusions, and methylation changes associated with the cancer can be detected through analysis of DNA or mRNA derived from these blood-borne tumour materials. CTCs, circulating tumour cells; ctDNA, circulating cell-free tumour DNA; miRNA, microRNA. Siravegna, G. et al. (2017) Integrating liquid biopsies into the management of cancer Nat. Rev. Clin. Oncol. doi: /nrclinonc

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CtDNA NGS testing identified a high-level MET amplification (copy number of 53.6 in circulation) (Figure 1A). The test was repeated on a second tube of.

CtDNA NGS testing identified a high-level MET amplification (copy number of 53.6 in circulation) (Figure 1A). The test was repeated on a second tube of.
Figure 1 Food, nutrition, obesity, physical activity, and the cellular processes linked to cancer Figure 1 | Food, nutrition, obesity, physical activity,

Figure 1 Food, nutrition, obesity, physical activity, and the cellular processes linked to cancer Figure 1 | Food, nutrition, obesity, physical activity,
Figure 1 The heterogenous landscape of triple-negative breast cancer

Figure 1 The heterogenous landscape of triple-negative breast cancer
Figure 2 Response after initial increase in total tumour burden

Figure 2 Response after initial increase in total tumour burden
Figure 3 Response after appearance of a new lesion

Figure 3 Response after appearance of a new lesion
Figure 5 Mutational heterogeneity in oesophageal and gastric cancer

Figure 5 Mutational heterogeneity in oesophageal and gastric cancer
Figure 1 Concept of the therapeutic index

Figure 1 Concept of the therapeutic index
Figure 1 Key elements of cancer-related inflammation

Figure 1 Key elements of cancer-related inflammation
Nat. Rev. Clin. Oncol. doi: /nrclinonc

Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 2 Multiscale modelling in oncology

Figure 2 Multiscale modelling in oncology
Figure 3 Risk-adapted and response-adapted

Figure 3 Risk-adapted and response-adapted
Figure 5 Schematic illustration of different clinical trial designs

Figure 5 Schematic illustration of different clinical trial designs
Nat. Rev. Clin. Oncol. doi: /nrclinonc

Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 1 Generations of cancer vaccine antigens

Figure 1 Generations of cancer vaccine antigens
Nat. Rev. Clin. Oncol. doi: /nrclinonc

Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 3 Monitoring clonal evolution using liquid biopsies

Figure 3 Monitoring clonal evolution using liquid biopsies
Figure 2 Three-step approach to the assessment and management

Figure 2 Three-step approach to the assessment and management
Nat. Rev. Clin. Oncol. doi: /nrclinonc

Nat. Rev. Clin. Oncol. doi: /nrclinonc
Nat. Rev. Clin. Oncol. doi: /nrclinonc

Nat. Rev. Clin. Oncol. doi: /nrclinonc
Figure 5 Identification of mucinous carcinoma

Figure 5 Identification of mucinous carcinoma

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